Tea catechins' affinity for human cannabinoid receptors.
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P. Sand | P. Schreier | J. Heilmann | S. Geiger | G. Korte | A. Dreiseitel | A. Oehme | S. Locher
[1] Chwan-Li Shen,et al. Open Access Bmc Musculoskeletal Disorders Green Tea Polyphenols and Tai Chi for Bone Health: Designing a Placebo-controlled Randomized Trial , 2022 .
[2] S. Troufflard,et al. Absorption, metabolism and excretion of Choladi green tea flavan-3-ols by humans. , 2009, Molecular nutrition & food research.
[3] M. Westerterp-Plantenga,et al. Green tea catechin plus caffeine supplementation to a high protein diet has no additional effect on body weight maintenance after weight loss , 2009, Appetite.
[4] R. Quirion,et al. Comparative Neuroprotective Properties of Stilbene and Catechin Analogs: Action Via a Plasma Membrane Receptor Site? , 2009, CNS neuroscience & therapeutics.
[5] E. Timofeeva,et al. The brain endocannabinoid system in the regulation of energy balance. , 2009, Best practice & research. Clinical endocrinology & metabolism.
[6] T. Hase,et al. A Catechin‐rich Beverage Improves Obesity and Blood Glucose Control in Patients With Type 2 Diabetes , 2009, Obesity.
[7] M. Wink,et al. Epigallocatechin Gallate from Green Tea (Camellia sinensis) Increases Lifespan and Stress Resistance in Caenorhabditis elegans , 2008, Planta medica.
[8] M. Rondanelli,et al. Administration of a dietary supplement ( N-oleyl-phosphatidylethanolamine and epigallocatechin-3-gallate formula) enhances compliance with diet in healthy overweight subjects: a randomized controlled trial. , 2008, The British journal of nutrition.
[9] F. Rizzi,et al. Molecular targets of (-)-epigallocatechin-3-gallate (EGCG): specificity and interaction with membrane lipid rafts. , 2008, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.
[10] Y. Aizawa,et al. Consumption of green and roasted teas and the risk of stroke incidence: results from the Tokamachi-Nakasato cohort study in Japan. , 2008, International journal of epidemiology.
[11] Chung S. Yang,et al. The major green tea polyphenol, (-)-epigallocatechin-3-gallate, inhibits obesity, metabolic syndrome, and fatty liver disease in high-fat-fed mice. , 2008, The Journal of nutrition.
[12] R. Liu,et al. Structure-activity relationships of flavonoids in the cellular antioxidant activity assay. , 2008, Journal of agricultural and food chemistry.
[13] D. Heber,et al. Nongallated compared with gallated flavan-3-ols in green and black tea are more bioavailable. , 2008, The Journal of nutrition.
[14] I. Galve-Roperh,et al. Mechanisms of control of neuron survival by the endocannabinoid system. , 2008, Current pharmaceutical design.
[15] Stephen P. H. Alexander,et al. Inhibition of fatty acid amide hydrolase and cyclooxygenase-2 increases levels of endocannabinoid related molecules and produces analgesia via peroxisome proliferator-activated receptor-alpha in a model of inflammatory pain , 2008, Neuropharmacology.
[16] S. Tiamkao,et al. Biphasic effects of Morus alba leaves green tea extract on mice in chronic forced swimming model , 2008, Phytotherapy research : PTR.
[17] B. Thinkhamrop,et al. Effectiveness of green tea on weight reduction in obese Thais: A randomized, controlled trial , 2008, Physiology & Behavior.
[18] R. Bauer,et al. CB receptor ligands from plants. , 2008, Current topics in medicinal chemistry.
[19] S. Cox,et al. Validation of green tea polyphenol biomarkers in a phase II human intervention trial. , 2008, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[20] A. Sant'ana,et al. Antinociceptive activity of Maytenus rigida stem bark. , 2007, Fitoterapia.
[21] M. Maeda-Yamamoto,et al. In vitro and in vivo anti-allergic effects of ‘benifuuki’ green tea containing O-methylated catechin and ginger extract enhancement , 2007, Cytotechnology.
[22] Wei-Jan Huang,et al. (−)‐Epigallocatechin gallate, the most active polyphenolic catechin in green tea, presynaptically facilitates Ca2+‐dependent glutamate release via activation of protein kinase C in rat cerebral cortex , 2007, Synapse.
[23] H. Navarro,et al. Flavonoids as opioid receptor ligands: identification and preliminary structure-activity relationships. , 2007, Journal of natural products.
[24] R. Ross,et al. Can EGCG Reduce Abdominal Fat in Obese Subjects? , 2007, Journal of the American College of Nutrition.
[25] F. F. Rocha,et al. Antidepressant-like effect of Cecropia glazioui Sneth and its constituents - in vivo and in vitro characterization of the underlying mechanism. , 2007, Phytomedicine : international journal of phytotherapy and phytopharmacology.
[26] M. Quon,et al. Epigallocatechin Gallate, a Green Tea Polyphenol, Mediates NO-dependent Vasodilation Using Signaling Pathways in Vascular Endothelium Requiring Reactive Oxygen Species and Fyn* , 2007, Journal of Biological Chemistry.
[27] Wei Wei,et al. Effects and mechanisms of catechin for adjuvant arthritis in rats , 2007, Advances in therapy.
[28] D. Hou,et al. Green tea proanthocyanidins inhibit cyclooxygenase-2 expression in LPS-activated mouse macrophages: molecular mechanisms and structure-activity relationship. , 2007, Archives of biochemistry and biophysics.
[29] M. Furuse,et al. Galloyl Group is not Necessary for a Sedative Effect of Catechin Through GABAergic System , 2007 .
[30] T. Murase,et al. Reduction of diet-induced obesity by a combination of tea-catechin intake and regular swimming , 2006, International Journal of Obesity.
[31] M. Furuse,et al. (-)-Epigallocatechin gallate attenuates acute stress responses through GABAergic system in the brain. , 2006, European journal of pharmacology.
[32] R. Nagatomi,et al. Green tea consumption and cognitive function: a cross-sectional study from the Tsurugaya Project 1. , 2006, The American journal of clinical nutrition.
[33] M. Westerterp-Plantenga,et al. Effect of green tea on resting energy expenditure and substrate oxidation during weight loss in overweight females , 2005, British Journal of Nutrition.
[34] K. Chopra,et al. Modulatory role of green tea extract on antinociceptive effect of morphine in diabetic mice. , 2005, Journal of medicinal food.
[35] D. M. Morré,et al. Medicinal benefits of green tea: Part I. Review of noncancer health benefits. , 2005, Journal of alternative and complementary medicine.
[36] A. Santos,et al. Antinociceptive effect of proanthocyanidins from Croton celtidifolius bark , 2005, The Journal of pharmacy and pharmacology.
[37] A. Finazzi-Agro’,et al. Lipid Rafts Control Signaling of Type-1 Cannabinoid Receptors in Neuronal Cells , 2005, Journal of Biological Chemistry.
[38] T. Kakuda,et al. Tea catechins with a galloyl moiety suppress postprandial hypertriacylglycerolemia by delaying lymphatic transport of dietary fat in rats. , 2005, The Journal of nutrition.
[39] K. Chopra,et al. Reversal of LPS‐induced central and peripheral hyperalgesia by green tea extract , 2005, Phytotherapy research : PTR.
[40] A. Hohmann,et al. Cannabinoid mechanisms of pain suppression. , 2005, Handbook of experimental pharmacology.
[41] K. Bae,et al. Green tea catechins as a BACE1 (β-secretase) inhibitor , 2003 .
[42] Q. Guo,et al. The galloyl moiety of green tea catechins is the critical structural feature to inhibit fatty-acid synthase. , 2003, Biochemical pharmacology.
[43] Tianhong Pan,et al. Potential Therapeutic Properties of Green Tea Polyphenols in Parkinson’s Disease , 2003, Drugs & aging.
[44] K. Leung,et al. Inhibitory effects of green tea catechins on protein tyrosine phosphatase in Prevotella intermedia. , 2003, Oral microbiology and immunology.
[45] B. Frei,et al. Tea Catechins and Polyphenols: Health Effects, Metabolism, and Antioxidant Functions , 2003, Critical reviews in food science and nutrition.
[46] T. Kakuda. Neuroprotective effects of the green tea components theanine and catechins. , 2002, Biological & pharmaceutical bulletin.
[47] M. Menegazzi,et al. Antiinflammatory Action of EGCG, the Main Component of Green Tea, through STAT‐1 Inhibition , 2002, Annals of the New York Academy of Sciences.
[48] T. Nohara,et al. Activity-guided fractionation of green tea extract with antiproliferative activity against human stomach cancer cells. , 2002, Biological & pharmaceutical bulletin.
[49] N. Sugihara,et al. The contribution of the pyrogallol moiety to the superoxide radical scavenging activity of flavonoids. , 2002, Biological & pharmaceutical bulletin.
[50] G. Beecher,et al. Catechins are bioavailable in men and women drinking black tea throughout the day. , 2001, The Journal of nutrition.
[51] D. Kromhout,et al. Catechin intake and associated dietary and lifestyle factors in a representative sample of Dutch men and women , 2001, European Journal of Clinical Nutrition.
[52] R. Hiipakka,et al. Modulation of obesity by a green tea catechin. , 2000, The American journal of clinical nutrition.
[53] J. Ruidavets,et al. Catechin in the Mediterranean diet: vegetable, fruit or wine? , 2000, Atherosclerosis.
[54] I. Arts,et al. Catechin contents of foods commonly consumed in The Netherlands. 1. Fruits, vegetables, staple foods, and processed foods. , 2000, Journal of agricultural and food chemistry.
[55] T. Miyase,et al. Importance of a pyrogallol-type structure in catechin compounds for apoptosis-inducing activity. , 2000, Phytochemistry.
[56] A. Capasso,et al. Flavonoids Reduce Morphine Withdrawal In‐vitro , 1998, The Journal of pharmacy and pharmacology.
[57] E. Feskens,et al. Dietary Flavonoids, Antioxidant Vitamins, and Incidence of Stroke: The Zutphen Study , 1996 .
[58] M. Herkenham,et al. Localization of cannabinoid receptors and nonsaturable high-density cannabinoid binding sites in peripheral tissues of the rat: implications for receptor-mediated immune modulation by cannabinoids. , 1994, The Journal of pharmacology and experimental therapeutics.
[59] S. Munro,et al. Molecular characterization of a peripheral receptor for cannabinoids , 1993, Nature.
[60] T. Bonner,et al. Structure of a cannabinoid receptor and functional expression of the cloned cDNA , 1990, Nature.
[61] W. Rewerski,et al. The analgesic action of some flavonoids in the hot plate test. , 1979, Acta physiologica Polonica.
[62] Y. Cheng,et al. Relationship between the inhibition constant (K1) and the concentration of inhibitor which causes 50 per cent inhibition (I50) of an enzymatic reaction. , 1973, Biochemical pharmacology.