Cigarette-smoke-induced atherogenic lipid profiles in plasma and vascular tissue of apolipoprotein E-deficient mice are attenuated by smoking cessation.

[1]  M. Peitsch,et al.  Modulation of atherogenic lipidome by cigarette smoke in apolipoprotein E-deficient mice. , 2012, Atherosclerosis.

[2]  Christophe Ladroue,et al.  Comparative Lipidomics Profiling of Human Atherosclerotic Plaques , 2011, Circulation. Cardiovascular genetics.

[3]  Karolina Błajecka,et al.  Phosphatidylinositol 3-kinase isoforms as novel drug targets. , 2011, Current drug targets.

[4]  Megan E. Piper,et al.  Effects of smoking and smoking cessation on lipids and lipoproteins: outcomes from a randomized clinical trial. , 2011, American heart journal.

[5]  D. Peake,et al.  Selective Reduction in the Sphingomyelin Content of Atherogenic Lipoproteins Inhibits Their Retention in Murine Aortas and the Subsequent Development of Atherosclerosis , 2010, Arteriosclerosis, thrombosis, and vascular biology.

[6]  Pierre Amarenco,et al.  Meta-analysis of the effect of nicotinic acid alone or in combination on cardiovascular events and atherosclerosis. , 2010, Atherosclerosis.

[7]  Kim Ekroos,et al.  Lipidomics: A Tool for Studies of Atherosclerosis , 2010, Current atherosclerosis reports.

[8]  P. Juvan,et al.  Acrolein consumption induces systemic dyslipidemia and lipoprotein modification. , 2010, Toxicology and applied pharmacology.

[9]  Office on Smoking.  How Tobacco Smoke Causes Disease: The Biology and Behavioral Basis for Smoking-Attributable Disease: A Report of the Surgeon General , 2010 .

[10]  S. Lebrun,et al.  Progression of atherosclerosis in the Apo E-/- model: 12-month exposure to cigarette mainstream smoke combined with high-cholesterol/fat diet. , 2009, Atherosclerosis.

[11]  B. Stamford,et al.  Smoking and smoking cessation -- the relationship between cardiovascular disease and lipoprotein metabolism: a review. , 2008, Atherosclerosis.

[12]  F. Paccaud,et al.  Consequences of smoking for body weight, body fat distribution, and insulin resistance. , 2008, The American journal of clinical nutrition.

[13]  H. Katus,et al.  Sphingosine-1-Phosphate Analogue FTY720 Causes Lymphocyte Redistribution and Hypercholesterolemia in ApoE-Deficient Mice , 2007, Arteriosclerosis, thrombosis, and vascular biology.

[14]  B. Levkau,et al.  The Sphingosine-1-Phosphate Analogue FTY720 Reduces Atherosclerosis in Apolipoprotein E–Deficient Mice , 2007, Arteriosclerosis, thrombosis, and vascular biology.

[15]  W. Toyofuku,et al.  Atherosclerosis and plasma and liver lipids in nine inbred strains of mice , 1993, Lipids.

[16]  Thomas P. Erlinger,et al.  Smoking Cessation and Cardiovascular Disease Risk Factors: Results from the Third National Health and Nutrition Examination Survey , 2005, PLoS medicine.

[17]  M. Hojjati,et al.  Effect of Myriocin on Plasma Sphingolipid Metabolism and Atherosclerosis in apoE-deficient Mice* , 2005, Journal of Biological Chemistry.

[18]  J. Ambrose,et al.  The pathophysiology of cigarette smoking and cardiovascular disease: an update. , 2004, Journal of the American College of Cardiology.

[19]  P. Vanscheeuwijck,et al.  Toxicological evaluation of an electrically heated cigarette. Part 4: Subchronic inhalation toxicology , 2003, Journal of applied toxicology : JAT.

[20]  G. Lowe,et al.  Relationship between smoking and cardiovascular risk factors in the development of peripheral arterial disease and coronary artery disease: Edinburgh Artery Study. , 1999, European heart journal.

[21]  A. Bhunia,et al.  Oxidized low density lipoproteins stimulate galactosyltransferase activity, ras activation, p44 mitogen activated protein kinase and c-fos expression in aortic smooth muscle cells. , 1997, Glycobiology.

[22]  A. Bhunia,et al.  Redox-regulated Signaling by Lactosylceramide in the Proliferation of Human Aortic Smooth Muscle Cells* , 1997, The Journal of Biological Chemistry.

[23]  J. Breslow,et al.  Apolipoprotein E and the apolipoprotein E-deficient mouse. , 1995, Annual review of nutrition.

[24]  R. Ross,et al.  ApoE-deficient mice develop lesions of all phases of atherosclerosis throughout the arterial tree. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.

[25]  D. Steinberg,et al.  Lysophosphatidylcholine: a chemotactic factor for human monocytes and its potential role in atherogenesis. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[26]  W. Willett,et al.  Relative and absolute excess risks of coronary heart disease among women who smoke cigarettes. , 1987, The New England journal of medicine.

[27]  P. Barter,et al.  Differences in plasma cholesteryl ester transfer activity in sixteen vertebrate species. , 1982, Comparative biochemistry and physiology. B, Comparative biochemistry.

[28]  O. Portman,et al.  Lysophosphatidylcholine concentrations and metabolism in aortic intima plus inner media: effect of nutritionally induced atherosclerosis. , 1969, Journal of lipid research.