Catalpol protects vascular structure and promotes angiogenesis in cerebral ischemic rats by targeting HIF-1α/VEGF.
暂无分享,去创建一个
Xiaoyu Xu | Qiang Xue | Hongjin Wang | Yue Yin | Xiaogang Xu | Shiqi Yu | Huijing Ren | Xiao-gang Xu
[1] Huifeng Zhu,et al. Catalpol induces cell activity to promote axonal regeneration via the PI3K/AKT/mTOR pathway in vivo and in vitro stroke model. , 2019, Annals of translational medicine.
[2] S. Gerecht,et al. Role of iPSC-derived pericytes on barrier function of iPSC-derived brain microvascular endothelial cells in 2D and 3D , 2019, Fluids and Barriers of the CNS.
[3] Xiaoyu Xu,et al. Knockdown of HIF‐1α impairs post‐ischemic vascular reconstruction in the brain via deficient homing and sprouting bmEPCs , 2018, Brain pathology.
[4] N. Hooper,et al. Tissue Engineering 3D Neurovascular Units: A Biomaterials and Bioprinting Perspective. , 2018, Trends in biotechnology.
[5] S. Gerecht,et al. Engineering the human blood-brain barrier in vitro , 2017, Journal of Biological Engineering.
[6] Szu-Fu Chen,et al. Low-intensity pulsed ultrasound improves behavioral and histological outcomes after experimental traumatic brain injury , 2017, Scientific Reports.
[7] C. Iadecola. The Neurovascular Unit Coming of Age: A Journey through Neurovascular Coupling in Health and Disease , 2017, Neuron.
[8] R. Lipton,et al. Association Between Vascular Pathology and Rate of Cognitive Decline Independent of Alzheimer's Disease Pathology , 2017, Journal of the American Geriatrics Society.
[9] Luca Cucullo,et al. New experimental models of the blood-brain barrier for CNS drug discovery , 2017, Expert opinion on drug discovery.
[10] L. Zhiqiang,et al. Catalpol stimulates VEGF production via the JAK2/STAT3 pathway to improve angiogenesis in rats' stroke model. , 2016, Journal of ethnopharmacology.
[11] U. Meyding-Lamadé,et al. Immortalized endothelial cell lines for in vitro blood–brain barrier models: A systematic review , 2016, Brain Research.
[12] B. Zlokovic,et al. Neurovascular dysfunction and neurodegeneration in dementia and Alzheimer's disease. , 2016, Biochimica et biophysica acta.
[13] A. Lazou,et al. Oxygen–Glucose Deprivation (OGD) Modulates the Unfolded Protein Response (UPR) and Inflicts Autophagy in a PC12 Hypoxia Cell Line Model , 2015, Cellular and Molecular Neurobiology.
[14] T. Hinchliffe,et al. Catalpol: a potential therapeutic for neurodegenerative diseases. , 2015, Current medicinal chemistry.
[15] Mandy B. Esch,et al. TEER Measurement Techniques for In Vitro Barrier Model Systems , 2015, Journal of laboratory automation.
[16] Stefan Roth,et al. Modeling Stroke in Mice: Permanent Coagulation of the Distal Middle Cerebral Artery , 2014, Journal of visualized experiments : JoVE.
[17] R. Anne Stetler,et al. Vascular remodeling after ischemic stroke: Mechanisms and therapeutic potentials , 2014, Progress in Neurobiology.
[18] Chao-Ching Huang,et al. Cerebral Microvascular Damage Occurs Early after Hypoxia–Ischemia via nNOS Activation in the Neonatal Brain , 2014, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] Xiaoyu Xu,et al. A simple method for isolating and culturing the rat brain microvascular endothelial cells. , 2013, Microvascular research.
[20] R. Daneman. The blood–brain barrier in health and disease , 2012, Annals of neurology.
[21] Rui Zhang,et al. Memory defect induced by beta-amyloid plus glutamate receptor agonist is alleviated by catalpol and donepezil through different mechanisms , 2012, Brain Research.
[22] Xing-shu Chen,et al. Protective effects of catalpol on oligodendrocyte death and myelin breakdown in a rat model of chronic cerebral hypoperfusion , 2011, Neuroscience Letters.
[23] V. I. Skvortsova,et al. The Significance of Toll-Like Receptors in the Development of Ischemic Damage , 2011, Neuroscience and Behavioral Physiology.
[24] Huifeng Zhu,et al. Catalpol Increases Brain Angiogenesis and Up-Regulates VEGF and EPO in the Rat after Permanent Middle Cerebral Artery Occlusion , 2010, International journal of biological sciences.
[25] K. Hirschi,et al. Diverse roles of the vasculature within the neural stem cell niche. , 2009, Regenerative medicine.
[26] S. Liu,et al. Catalpol ameliorates beta amyloid–induced degeneration of cholinergic neurons by elevating brain-derived neurotrophic factors , 2009, Neuroscience.
[27] R. Jahan,et al. Treatment of acute ischemic stroke: intravenous and endovascular therapies , 2009, Expert review of cardiovascular therapy.
[28] Z. Jia,et al. Rehmannia glutinosa: review of botany, chemistry and pharmacology. , 2008, Journal of ethnopharmacology.
[29] T. Terasaki,et al. Expression of nuclear receptor mRNA and liver X receptor-mediated regulation of ABC transporter A1 at rat blood–brain barrier , 2008, Neurochemistry International.
[30] B. Zlokovic. The Blood-Brain Barrier in Health and Chronic Neurodegenerative Disorders , 2008, Neuron.
[31] M. Chopp,et al. Intracarotid transplantation of bone marrow stromal cells increases axon-myelin remodeling after stroke , 2006, Neuroscience.
[32] J. Castillo,et al. Vascular Protection in Brain Ischemia , 2006, Cerebrovascular Diseases.
[33] J. Barker,et al. Development of an Ischemic Tolerance Model in a PC12 Cell Line , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[34] L. An,et al. Neuroprotection of catalpol in transient global ischemia in gerbils , 2004, Neuroscience Research.
[35] P. Carmeliet,et al. VEGF: once regarded as a specific angiogenic factor, now implicated in neuroprotection , 2004, BioEssays : news and reviews in molecular, cellular and developmental biology.
[36] N. Ferrara,et al. The biology of VEGF and its receptors , 2003, Nature Medicine.
[37] W. Pardridge,et al. Drug and Gene Delivery to the Brain The Vascular Route , 2002, Neuron.
[38] J. Broderick,et al. Predicting prognosis after stroke , 2000, Neurology.
[39] S. Ylä-Herttuala,et al. Vascular protection: A novel nonangiogenic cardiovascular role for vascular endothelial growth factor. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[40] A. Shuaib,et al. Quantification of infarct size on focal cerebral ischemia model of rats using a simple and economical method , 1998, Journal of Neuroscience Methods.
[41] U. Dirnagl,et al. Induction of hypoxia inducible factor 1 by oxygen glucose deprivation is attenuated by hypoxic preconditioning in rat cultured neurons , 1998, Neuroscience Letters.
[42] Y. Itoyama,et al. Rapid induction of vascular endothelial growth factor gene expression after transient middle cerebral artery occlusion in rats. , 1997, Stroke.
[43] S. Constantini,et al. An experimental model of closed head injury in mice: pathophysiology, histopathology, and cognitive deficits. , 1996, Journal of neurotrauma.
[44] M. Goldberg,et al. Combined oxygen and glucose deprivation in cortical cell culture: calcium-dependent and calcium-independent mechanisms of neuronal injury , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[45] D. Duverger,et al. The Quantification of Cerebral Infarction following Focal Ischemia in the Rat: Influence of Strain, Arterial Pressure, Blood Glucose Concentration, and Age , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[46] E. Hansson,et al. Astrocyte–endothelial interactions at the blood–brain barrier , 2006, Nature Reviews Neuroscience.