Associations between amyloid β and white matter hyperintensities: A systematic review
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Sandra E. Black | Joel Ramirez | J. Ramirez | J. Edwards | A. Roseborough | Austyn Roseborough | Jodi D. Edwards | S. Black | Austyn Roseborough
[1] S. Pocock,et al. The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) statement: guidelines for reporting observational studies. , 2007, Preventive medicine.
[2] J. S. Kim,et al. Identification of pure subcortical vascular dementia using 11C-Pittsburgh compound B , 2011, Neurology.
[3] E. Rostrup,et al. Decline in intelligence is associated with progression in white matter hyperintensity volume , 2005, Journal of Neurology, Neurosurgery & Psychiatry.
[4] Olivier Salvado,et al. Incidence of cerebral microbleeds in preclinical Alzheimer disease , 2014, Neurology.
[5] P. Scheltens,et al. White matter lesions on magnetic resonance imaging in dementia with Lewy bodies, Alzheimer’s disease, vascular dementia, and normal aging , 1999, Journal of neurology, neurosurgery, and psychiatry.
[6] Miranka Wirth,et al. Neuroprotective pathways: lifestyle activity, brain pathology, and cognition in cognitively normal older adults , 2014, Neurobiology of Aging.
[7] S le Cessie,et al. Induction versus expectant monitoring for intrauterine growth restriction at term: randomised equivalence trial (DIGITAT) , 2010, BMJ : British Medical Journal.
[8] Jordan Muraskin,et al. White matter hyperintensities and cerebral amyloidosis: necessary and sufficient for clinical expression of Alzheimer disease? , 2013, JAMA neurology.
[9] K. McGuire,et al. Digging the New York City Skyline: Soil Fungal Communities in Green Roofs and City Parks , 2013, PloS one.
[10] B C Bowen,et al. MR signal abnormalities in memory disorder and dementia. , 1990, AJNR. American journal of neuroradiology.
[11] Sue J. Kang,et al. The effects of small vessel disease and amyloid burden on neuropsychiatric symptoms: a study among patients with subcortical vascular cognitive impairments , 2013, Neurobiology of Aging.
[12] M. Wortmann,et al. World Alzheimer report 2014: Dementia and risk reduction , 2015, Alzheimer's & Dementia.
[13] D. Werring,et al. White matter hyperintensities are associated with amyloid burden in APOE4 non-carriers. , 2014, Journal of Alzheimer's disease : JAD.
[14] M. Weiner,et al. Effects of cerebrovascular disease and amyloid beta burden on cognition in subjects with subcortical vascular cognitive impairment , 2014, Neurobiology of Aging.
[15] T. Duong,et al. White matter lesion load is associated with resting state functional MRI activity and amyloid pet but not FDG in mild cognitive impairment and early alzheimer's disease patients , 2015, Journal of magnetic resonance imaging : JMRI.
[16] C. Sorg,et al. Small Vessel Disease, but Neither Amyloid Load nor Metabolic Deficit, Is Dependent on Age at Onset in Alzheimer’s Disease , 2015, Biological Psychiatry.
[17] L. Kuller,et al. Pulse wave velocity is associated with β-amyloid deposition in the brains of very elderly adults , 2013, Neurology.
[18] Brian A. Gordon,et al. The effects of white matter hyperintensities and amyloid deposition on Alzheimer dementia , 2015, NeuroImage: Clinical.
[19] E. Ross,et al. Philosophy of Science Association , 2022 .
[20] A. Alavi,et al. MR signal abnormalities at 1.5 T in Alzheimer's dementia and normal aging. , 1987, AJR. American journal of roentgenology.
[21] A. Brun,et al. A Clinico-Pathological Study of Heart and Brain Lesions in Vascular Dementia , 2005, Dementia and Geriatric Cognitive Disorders.
[22] H. Feldman,et al. Periventricular hyperintensities are associated with elevated cerebral amyloid , 2016, Neurology.
[23] D. Werring,et al. Blood viscosity in subcortical vascular mild cognitive impairment with versus without cerebral amyloid burden. , 2014, Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.
[24] W. Jagust,et al. Coronary risk correlates with cerebral amyloid deposition , 2012, Neurobiology of Aging.
[25] A. Brickman,et al. APOE ε4 and risk for Alzheimer's disease: Do regionally distributed white matter hyperintensities play a role? , 2014, Alzheimer's & Dementia.
[26] Paul Maruff,et al. Beta-amyloid imaging and memory in non-demented individuals: evidence for preclinical Alzheimer's disease. , 2007, Brain : a journal of neurology.
[27] Christos Davatzikos,et al. White matter hyperintensities and imaging patterns of brain ageing in the general population. , 2016, Brain : a journal of neurology.
[28] H. Braak,et al. Staging of alzheimer's disease-related neurofibrillary changes , 1995, Neurobiology of Aging.
[29] Nick C Fox,et al. White matter hyperintensities are a core feature of Alzheimer's disease: Evidence from the dominantly inherited Alzheimer network , 2016, Annals of neurology.
[30] W. Markesbery,et al. Alzheimer's neurofibrillary pathology and the spectrum of cognitive function: Findings from the Nun Study , 2002, Annals of neurology.
[31] Jae Seung Kim,et al. Seoul criteria for PiB(−) subcortical vascular dementia based on clinical and MRI variables , 2014, Neurology.
[32] C. Jack,et al. Brain injury biomarkers are not dependent on β‐amyloid in normal elderly , 2013, Annals of neurology.
[33] William J Powers,et al. Autoregulation of Cerebral Blood Flow to Changes in Arterial Pressure in Mild Alzheimer's Disease , 2010, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[34] Y. J. Kim,et al. White matter microstructural changes in pure Alzheimer's disease and subcortical vascular dementia , 2015, European journal of neurology.
[35] Martin Prince,et al. World Alzheimer Report 2014: Dementia and risk reduction: An analysis of protective and modifiable risk factors , 2014 .
[36] D. Moher,et al. Preferred reporting items for systematic reviews and meta-analyses: the PRISMA statement. , 2010, International journal of surgery.
[37] C. Rowe,et al. Amyloid imaging results from the Australian Imaging, Biomarkers and Lifestyle (AIBL) study of aging , 2010, Neurobiology of Aging.
[38] Alan C. Evans,et al. Epidemic Spreading Model to Characterize Misfolded Proteins Propagation in Aging and Associated Neurodegenerative Disorders , 2014, PLoS Comput. Biol..
[39] Florian Auer,et al. White matter hyperintensities predict amyloid increase in Alzheimer's disease , 2012, Neurobiology of Aging.
[40] Nick C Fox,et al. Neuroimaging standards for research into small vessel disease and its contribution to ageing and neurodegeneration , 2013, The Lancet Neurology.
[41] Nick C Fox,et al. Regional variability of imaging biomarkers in autosomal dominant Alzheimer’s disease , 2013, Proceedings of the National Academy of Sciences.
[42] Keith A. Johnson,et al. Cerebral amyloid angiopathy burden associated with leukoaraiosis: A positron emission tomography/magnetic resonance imaging study , 2013, Annals of neurology.
[43] L. Pantoni. Cerebral small vessel disease: from pathogenesis and clinical characteristics to therapeutic challenges , 2010, The Lancet Neurology.
[44] Thomas Wisniewski,et al. Clearance systems in the brain—implications for Alzheimer disease , 2015, Nature Reviews Neurology.
[45] Kenneth Rockwood,et al. Diagnostic Criteria for Vascular Cognitive Disorders: A VASCOG Statement , 2014, Alzheimer disease and associated disorders.
[46] M. Weiner,et al. Individual Subject Classification of Mixed Dementia from Pure Subcortical Vascular Dementia Based on Subcortical Shape Analysis , 2013, PloS one.
[47] H. Markus,et al. The clinical importance of white matter hyperintensities on brain magnetic resonance imaging: systematic review and meta-analysis , 2010, BMJ : British Medical Journal.
[48] J. Becker,et al. Amyloid, neurodegeneration, and small vessel disease as predictors of dementia in the oldest-old , 2014, Neurology.
[49] Jae Seung Kim,et al. Cognitive deficits of pure subcortical vascular dementia vs Alzheimer disease , 2013, Neurology.
[50] Mette E. Haahr,et al. Lack of association between prior depressive episodes and cerebral [11C]PiB binding , 2012, Neurobiology of Aging.
[51] T. Yen,et al. Amyloid deposition after cerebral hypoperfusion: Evidenced on [18F]AV-45 positron emission tomography , 2012, Journal of the Neurological Sciences.
[52] D. Werring,et al. Pathogenesis of cerebral microbleeds: In vivo imaging of amyloid and subcortical ischemic small vessel disease in 226 individuals with cognitive impairment , 2013, Annals of neurology.
[53] I. Deary,et al. APOE/TOMM 40 genetic loci, white matter hyperintensities, and cerebral microbleeds , 2015, International journal of stroke : official journal of the International Stroke Society.
[54] M. Munafo,et al. A Validation Study of Vascular Cognitive Impairment Genetics Meta-Analysis Findings in an Independent Collaborative Cohort. , 2016, Journal of Alzheimer's disease : JAD.
[55] C. Jack,et al. Vascular and amyloid pathologies are independent predictors of cognitive decline in normal elderly , 2015, Brain : a journal of neurology.
[56] J. H. Lee,et al. Cortical thickness and hippocampal shape in pure vascular mild cognitive impairment and dementia of subcortical type , 2014, European journal of neurology.
[57] Cindee M. Madison,et al. Cerebrovascular disease, beta-amyloid, and cognition in aging , 2012, Neurobiology of Aging.
[58] M. Weiner,et al. Effects of APOE ɛ4 on brain amyloid, lacunar infarcts, and white matter lesions: a study among patients with subcortical vascular cognitive impairment , 2013, Neurobiology of Aging.
[59] Susan M Resnick,et al. Longitudinal Cerebral Blood Flow and Amyloid Deposition: An Emerging Pattern? , 2008, Journal of Nuclear Medicine.
[60] Rebecca F. Halperin,et al. A high-density whole-genome association study reveals that APOE is the major susceptibility gene for sporadic late-onset Alzheimer's disease. , 2007, The Journal of clinical psychiatry.
[61] P. Vemuri,et al. Brain β-amyloid load approaches a plateau , 2013, Neurology.
[62] P. Matthews,et al. White matter lesion progression, brain atrophy, and cognitive decline: The Austrian stroke prevention study , 2005, Annals of neurology.
[63] S. Black,et al. Vascular Contributions to Cognitive Impairment and Dementia: A Statement for Healthcare Professionals From the American Heart Association/American Stroke Association , 2011, Stroke.
[64] Miranka Wirth,et al. Vascular risk and Aβ interact to reduce cortical thickness in AD vulnerable brain regions , 2014, Neurology.
[65] W. Klunk,et al. Uncharged thioflavin-T derivatives bind to amyloid-beta protein with high affinity and readily enter the brain. , 2001, Life sciences.
[66] Masanori Ichise,et al. Cerebral autoregulation, beta amyloid, and white matter hyperintensities are interrelated , 2015, Neuroscience Letters.
[67] Cindee M. Madison,et al. Associations between Alzheimer disease biomarkers, neurodegeneration, and cognition in cognitively normal older people. , 2013, JAMA neurology.
[68] Jae Seung Kim,et al. Amyloid burden, cerebrovascular disease, brain atrophy, and cognition in cognitively impaired patients , 2015, Alzheimer's & Dementia.
[69] Vijaya L. Melnick,et al. Alzheimer’s Dementia , 1985, Contemporary Issues in Biomedicine, Ethics, and Society.
[70] D. Werring,et al. White Matter Changes in Dementia: Role of Impaired Drainage of Interstitial Fluid , 2015, Brain pathology.
[71] L. Kuller,et al. Arterial stiffness is associated with amyloid deposition in the brain independent of blood pressure , 2013, Alzheimer's & Dementia.
[72] D. Na,et al. Differential effects of white matter hyperintensity on geriatric depressive symptoms according to APOE-ε4 status. , 2015, Journal of affective disorders.
[73] Cindee M. Madison,et al. Cortical thickness mediates the effect of β-amyloid on episodic memory , 2014, Neurology.
[74] C. Jack,et al. Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade , 2010, The Lancet Neurology.
[75] K. Jellinger. The enigma of mixed dementia , 2007, Alzheimer's & Dementia.
[76] J. Haxby,et al. The effect of white matter hyperintensity volume on brain structure, cognitive performance, and cerebral metabolism of glucose in 51 healthy adults , 1995, Neurology.
[77] Tyler E. Benedum,et al. Preclinical Properties of 18F-AV-45: A PET Agent for Aβ Plaques in the Brain , 2009, Journal of Nuclear Medicine.
[78] L. Wilkins. Identification of pure subcortical vascular dementia using 11C-Pittsburgh compound B , 2011, Neurology.
[79] Cindee M. Madison,et al. The aging brain and cognition: contribution of vascular injury and aβ to mild cognitive dysfunction. , 2013, JAMA neurology.
[80] D. Moher,et al. Reprint--preferred reporting items for systematic reviews and meta-analyses: the PRISMA statement. , 2009, Physical therapy.
[81] W. Jagust,et al. Imaging Vascular Disease and Amyloid in the Aging Brain: Implications for Treatment. , 2015, The journal of prevention of Alzheimer's disease.
[82] W. Klunk,et al. Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound‐B , 2004, Annals of neurology.
[83] Nick C. Fox,et al. Genetic determinants of white matter hyperintensities and amyloid angiopathy in familial Alzheimer's disease , 2015, Neurobiology of Aging.
[84] N. De Stefano,et al. APOE ɛ2 is associated with white matter hyperintensity volume in CADASIL , 2016, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.