Septal rebound stretch reflects the functional substrate to cardiac resynchronization therapy and predicts volumetric and neurohormonal response

To develop a novel myocardial deformation index that is highly sensitive to the effect of cardiac resynchronization therapy (CRT) and that can be used to predict response to CRT.

[1]  Piet Claus,et al.  Toward understanding response to cardiac resynchronization therapy: left ventricular dyssynchrony is only one of multiple mechanisms. , 2009, European heart journal.

[2]  J. McMurray,et al.  How should we optimize cardiac resynchronization therapy? , 2008, European heart journal.

[3]  David O. Martin,et al.  The magnitude of reverse remodelling irrespective of aetiology predicts outcome of heart failure patients treated with cardiac resynchronization therapy. , 2008, European heart journal.

[4]  Adisai Buakhamsri,et al.  Longitudinal Strain Delay Index by Speckle Tracking Imaging: A New Marker of Response to Cardiac Resynchronization Therapy , 2008, Circulation.

[5]  N. Freemantle,et al.  Predicting the long-term effects of cardiac resynchronization therapy on mortality from baseline variables and the early response a report from the CARE-HF (Cardiac Resynchronization in Heart Failure) Trial. , 2008, Journal of the American College of Cardiology.

[6]  Theo Arts,et al.  Mechanical discoordination rather than dyssynchrony predicts reverse remodeling upon cardiac resynchronization. , 2008, American journal of physiology. Heart and circulatory physiology.

[7]  J. Oh,et al.  Strain Dyssynchrony Index Correlates With Improvement in Left Ventricular Volume After Cardiac Resynchronization Therapy Better Than Tissue Velocity Dyssynchrony Indexes , 2008, Circulation. Cardiovascular imaging.

[8]  F. Prinzen,et al.  Three-dimensional mapping of mechanical activation patterns, contractile dyssynchrony and dyscoordination by two-dimensional strain echocardiography: Rationale and design of a novel software toolbox , 2008 .

[9]  Jeroen J. Bax,et al.  Results of the Predictors of Response to CRT (PROSPECT) Trial , 2008, Circulation.

[10]  Mathias Meine,et al.  Practical and conceptual limitations of tissue Doppler imaging to predict reverse remodelling in cardiac resynchronisation therapy , 2008, European journal of heart failure.

[11]  D. Kass An epidemic of dyssynchrony: but what does it mean? , 2008, Journal of the American College of Cardiology.

[12]  P. Tchou,et al.  Cardiac-resynchronization therapy in heart failure with narrow QRS complexes. , 2007, The New England journal of medicine.

[13]  C. Vahlhaus,et al.  Diastolic filling pattern and left ventricular diameter predict response and prognosis after cardiac resynchronisation therapy , 2007, Heart.

[14]  Nick Freemantle,et al.  Predictors and treatment response with cardiac resynchronization therapy in patients with heart failure characterized by dyssynchrony: a pre-defined analysis from the CARE-HF trial. , 2007, European heart journal.

[15]  J. Daubert,et al.  Early and sustained effects of cardiac resynchronization therapy on N-terminal pro-B-type natriuretic peptide in patients with moderate to severe heart failure and cardiac dyssynchrony. , 2006, European heart journal.

[16]  Nael F Osman,et al.  Three-Dimensional Mapping of Optimal Left Ventricular Pacing Site for Cardiac Resynchronization , 2007, Circulation.

[17]  Jeroen J. Bax,et al.  Impact of viability and scar tissue on response to cardiac resynchronization therapy in ischaemic heart failure patients. , 2006, European heart journal.

[18]  J. Daubert,et al.  Predicting the long-term effects of cardiac resynchronisation therapy from baseline variables and the early response. A report from CARE-HF (Cardiac Resynchronisation in Heart Failure) , 2006 .

[19]  A. Capucci,et al.  Left intraventricular myocardial deformation dyssynchrony identifies responders to cardiac resynchronization therapy in patients with heart failure. , 2006, European heart journal.

[20]  Maxime Cannesson,et al.  Novel Speckle-Tracking Radial Strain From Routine Black-and-White Echocardiographic Images to Quantify Dyssynchrony and Predict Response to Cardiac Resynchronization Therapy , 2006, Circulation.

[21]  David L Hayes,et al.  Sustained Reverse Left Ventricular Structural Remodeling With Cardiac Resynchronization at One Year Is a Function of Etiology: Quantitative Doppler Echocardiographic Evidence From the Multicenter InSync Randomized Clinical Evaluation (MIRACLE) , 2006, Circulation.

[22]  Jeroen J. Bax,et al.  Left Ventricular Reverse Remodeling but Not Clinical Improvement Predicts Long-Term Survival After Cardiac Resynchronization Therapy , 2005, Circulation.

[23]  J. Daubert,et al.  The effect of cardiac resynchronization on morbidity and mortality in heart failure. , 2005, The New England journal of medicine.

[24]  AngeloAuricchio,et al.  Characterization of Left Ventricular Activation in Patients With Heart Failure and Left Bundle-Branch Block , 2004 .

[25]  Angelo Auricchio,et al.  Characterization of Left Ventricular Activation in Patients With Heart Failure and Left Bundle-Branch Block , 2004, Circulation.

[26]  H. Halperin,et al.  Systolic Improvement and Mechanical Resynchronization Does Not Require Electrical Synchrony in the Dilated Failing Heart With Left Bundle-Branch Block , 2002, Circulation.

[27]  E. Hartter,et al.  Prognostic evaluation of neurohumoral plasma levels before and during beta-blocker therapy in advanced left ventricular dysfunction. , 2001, Journal of the American College of Cardiology.

[28]  Gregory S. Nelson,et al.  Left Ventricular or Biventricular Pacing Improves Cardiac Function at Diminished Energy Cost in Patients With Dilated Cardiomyopathy and Left Bundle-Branch Block , 2000, Circulation.

[29]  F W Prinzen,et al.  Mapping of regional myocardial strain and work during ventricular pacing: experimental study using magnetic resonance imaging tagging. , 1999, Journal of the American College of Cardiology.

[30]  F. Prinzen,et al.  Regional electrical activation and mechanical function in the partially ischemic left ventricle of dogs. , 1996, The American journal of physiology.

[31]  A. Pezzano,et al.  Value of transthoracic two-dimensional echocardiography in predicting viability in patients with healed Q-wave anterior wall myocardial infarction. , 1995, The American journal of cardiology.

[32]  J. Hanley,et al.  A method of comparing the areas under receiver operating characteristic curves derived from the same cases. , 1983, Radiology.

[33]  R. C. Reeves,et al.  Mechanism of abnormal interventricular septal motion during delayed left ventricular activation. , 1982, Circulation.