Septal rebound stretch reflects the functional substrate to cardiac resynchronization therapy and predicts volumetric and neurohormonal response
暂无分享,去创建一个
[1] Piet Claus,et al. Toward understanding response to cardiac resynchronization therapy: left ventricular dyssynchrony is only one of multiple mechanisms. , 2009, European heart journal.
[2] J. McMurray,et al. How should we optimize cardiac resynchronization therapy? , 2008, European heart journal.
[3] David O. Martin,et al. The magnitude of reverse remodelling irrespective of aetiology predicts outcome of heart failure patients treated with cardiac resynchronization therapy. , 2008, European heart journal.
[4] Adisai Buakhamsri,et al. Longitudinal Strain Delay Index by Speckle Tracking Imaging: A New Marker of Response to Cardiac Resynchronization Therapy , 2008, Circulation.
[5] N. Freemantle,et al. Predicting the long-term effects of cardiac resynchronization therapy on mortality from baseline variables and the early response a report from the CARE-HF (Cardiac Resynchronization in Heart Failure) Trial. , 2008, Journal of the American College of Cardiology.
[6] Theo Arts,et al. Mechanical discoordination rather than dyssynchrony predicts reverse remodeling upon cardiac resynchronization. , 2008, American journal of physiology. Heart and circulatory physiology.
[7] J. Oh,et al. Strain Dyssynchrony Index Correlates With Improvement in Left Ventricular Volume After Cardiac Resynchronization Therapy Better Than Tissue Velocity Dyssynchrony Indexes , 2008, Circulation. Cardiovascular imaging.
[8] F. Prinzen,et al. Three-dimensional mapping of mechanical activation patterns, contractile dyssynchrony and dyscoordination by two-dimensional strain echocardiography: Rationale and design of a novel software toolbox , 2008 .
[9] Jeroen J. Bax,et al. Results of the Predictors of Response to CRT (PROSPECT) Trial , 2008, Circulation.
[10] Mathias Meine,et al. Practical and conceptual limitations of tissue Doppler imaging to predict reverse remodelling in cardiac resynchronisation therapy , 2008, European journal of heart failure.
[11] D. Kass. An epidemic of dyssynchrony: but what does it mean? , 2008, Journal of the American College of Cardiology.
[12] P. Tchou,et al. Cardiac-resynchronization therapy in heart failure with narrow QRS complexes. , 2007, The New England journal of medicine.
[13] C. Vahlhaus,et al. Diastolic filling pattern and left ventricular diameter predict response and prognosis after cardiac resynchronisation therapy , 2007, Heart.
[14] Nick Freemantle,et al. Predictors and treatment response with cardiac resynchronization therapy in patients with heart failure characterized by dyssynchrony: a pre-defined analysis from the CARE-HF trial. , 2007, European heart journal.
[15] J. Daubert,et al. Early and sustained effects of cardiac resynchronization therapy on N-terminal pro-B-type natriuretic peptide in patients with moderate to severe heart failure and cardiac dyssynchrony. , 2006, European heart journal.
[16] Nael F Osman,et al. Three-Dimensional Mapping of Optimal Left Ventricular Pacing Site for Cardiac Resynchronization , 2007, Circulation.
[17] Jeroen J. Bax,et al. Impact of viability and scar tissue on response to cardiac resynchronization therapy in ischaemic heart failure patients. , 2006, European heart journal.
[18] J. Daubert,et al. Predicting the long-term effects of cardiac resynchronisation therapy from baseline variables and the early response. A report from CARE-HF (Cardiac Resynchronisation in Heart Failure) , 2006 .
[19] A. Capucci,et al. Left intraventricular myocardial deformation dyssynchrony identifies responders to cardiac resynchronization therapy in patients with heart failure. , 2006, European heart journal.
[20] Maxime Cannesson,et al. Novel Speckle-Tracking Radial Strain From Routine Black-and-White Echocardiographic Images to Quantify Dyssynchrony and Predict Response to Cardiac Resynchronization Therapy , 2006, Circulation.
[21] David L Hayes,et al. Sustained Reverse Left Ventricular Structural Remodeling With Cardiac Resynchronization at One Year Is a Function of Etiology: Quantitative Doppler Echocardiographic Evidence From the Multicenter InSync Randomized Clinical Evaluation (MIRACLE) , 2006, Circulation.
[22] Jeroen J. Bax,et al. Left Ventricular Reverse Remodeling but Not Clinical Improvement Predicts Long-Term Survival After Cardiac Resynchronization Therapy , 2005, Circulation.
[23] J. Daubert,et al. The effect of cardiac resynchronization on morbidity and mortality in heart failure. , 2005, The New England journal of medicine.
[24] AngeloAuricchio,et al. Characterization of Left Ventricular Activation in Patients With Heart Failure and Left Bundle-Branch Block , 2004 .
[25] Angelo Auricchio,et al. Characterization of Left Ventricular Activation in Patients With Heart Failure and Left Bundle-Branch Block , 2004, Circulation.
[26] H. Halperin,et al. Systolic Improvement and Mechanical Resynchronization Does Not Require Electrical Synchrony in the Dilated Failing Heart With Left Bundle-Branch Block , 2002, Circulation.
[27] E. Hartter,et al. Prognostic evaluation of neurohumoral plasma levels before and during beta-blocker therapy in advanced left ventricular dysfunction. , 2001, Journal of the American College of Cardiology.
[28] Gregory S. Nelson,et al. Left Ventricular or Biventricular Pacing Improves Cardiac Function at Diminished Energy Cost in Patients With Dilated Cardiomyopathy and Left Bundle-Branch Block , 2000, Circulation.
[29] F W Prinzen,et al. Mapping of regional myocardial strain and work during ventricular pacing: experimental study using magnetic resonance imaging tagging. , 1999, Journal of the American College of Cardiology.
[30] F. Prinzen,et al. Regional electrical activation and mechanical function in the partially ischemic left ventricle of dogs. , 1996, The American journal of physiology.
[31] A. Pezzano,et al. Value of transthoracic two-dimensional echocardiography in predicting viability in patients with healed Q-wave anterior wall myocardial infarction. , 1995, The American journal of cardiology.
[32] J. Hanley,et al. A method of comparing the areas under receiver operating characteristic curves derived from the same cases. , 1983, Radiology.
[33] R. C. Reeves,et al. Mechanism of abnormal interventricular septal motion during delayed left ventricular activation. , 1982, Circulation.