Lesions of the area postrema and underlying solitary nucleus fail to attenuate the inhibition of feeding produced by systemic injections of cholecystokinin in Syrian hamsters

[1]  C. Baile,et al.  Role of cholecystokinin and opioid peptides in control of food intake. , 1986, Physiological reviews.

[2]  M. Miceli,et al.  Effects of putative satiety peptides on feeding and drinking behavior in golden hamsters (Mesocricetus auratus). , 1985, Behavioral neuroscience.

[3]  M. Miceli.  Role of gastric distention in cholecystokinin's satiety effect in golden hamsters , 1985, Physiology & Behavior.

[4]  G. P. Smith,et al.  Afferent axons in abdominal vagus mediate satiety effect of cholecystokinin in rats. , 1985, The American journal of physiology.

[5]  N. Rowland.  Ingestive behaviour of Syrian hamsters: Advantages of the comparative approach , 1985, Brain Research Bulletin.

[6]  R. Miselis,et al.  The central organization of the vagus nerve innervating the stomach of the rat , 1985, The Journal of comparative neurology.

[7]  M. Miceli,et al.  Brainstem origins and projections of the cervical and abdominal vagus in the golden hamster: A horseradish peroxidase study , 1985, The Journal of comparative neurology.

[8]  M. Miceli.  Abdominal vagus and regulation of ingestive behavior and body weight in golden hamsters. , 1985, The American journal of physiology.

[9]  T. Hyde,et al.  Area postrema and adjacent solitary nucleus in water and energy balance. , 1984, Federation proceedings.

[10]  F. Kraly.  Vagotomy does not alter cholecystokinin's inhibition of sham feeding. , 1984, The American journal of physiology.

[11]  D. Kooy.  Area postrema: site where cholecystokinin acts to decrease food intake , 1984, Brain Research.

[12]  G. P. Smith,et al.  The neuroendocrinology of postprandial satiety , 1984 .

[13]  J. Schwaber,et al.  Nucleus tractus solitarius lesions block the behavioral actions of cholecystokinin , 1983, Peptides.

[14]  T. Hyde,et al.  Effects of area postrema/caudal medial nucleus of solitary tract lesions on food intake and body weight. , 1983, The American journal of physiology.

[15]  S. Snyder,et al.  Autoradiographic localization of cholecystokinin receptors in rodent brain , 1983, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[16]  M. L. Drugovich,et al.  Area postrema lesions produce feeding deficits in the rat: Effects of preoperative dieting and 2-deoxy-D-glucose , 1982, Physiology & Behavior.

[17]  R. Ritter,et al.  Area postrema lesions increase drinking to angiotensin and extracellular dehydration , 1982, Physiology & Behavior.

[18]  Steven A. Goldman,et al.  Vagal mediation of the cholecystokinin satiety effect in rats , 1982, Physiology & Behavior.

[19]  W. Oldendorf,et al.  Rapid appearance of intraventricularly administered neuropeptides in the peripheral circulation , 1982, Brain Research.

[20]  G. P. Smith,et al.  Abdominal vagotomy blocks the satiety effect of cholecystokinin in the rat. , 1981, Science.

[21]  R. Innis,et al.  Cholecystokinin receptors: presence and axonal flow in the rat vagus nerve. , 1981, Life sciences.

[22]  R. Ritter,et al.  Ablation of the area postrema causes exaggerated consumption of preferred foods in the rat , 1981, Brain Research.