Preview : Published ahead of advance online publication Therapeutic Potential of Inhibitors of Endocannabinoid Degradation for the Treatment of Stress-Related Hyperalgesia in an Animal Model of Chronic Pain
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B. Lutz | L. Bindila | F. Remmers | Ermelinda Lomazzo | U. Hoheisel | Claudia Schwitter | Raissa Lerner
[1] Livio Luongo,et al. Endocannabinoids and neuropathic pain: focus on neuron–glia and endocannabinoid–neurotrophin interactions , 2014, The European journal of neuroscience.
[2] Irene Tracey,et al. Pain vulnerability: a neurobiological perspective , 2014, Nature Neuroscience.
[3] D. Piomelli,et al. Peripheral gating of pain signals by endogenous lipid mediators , 2014, Nature Neuroscience.
[4] V. Marzo,et al. Endocannabinoids: A unique opportunity to develop multitarget analgesics , 2013, PAIN®.
[5] Sachin Patel,et al. Translational evidence for the involvement of the endocannabinoid system in stress-related psychiatric illnesses , 2013, Biology of Mood & Anxiety Disorders.
[6] R. Treede,et al. Injection of nerve growth factor into a low back muscle induces long-lasting latent hypersensitivity in rat dorsal horn neurons , 2013, PAIN®.
[7] S. Chattarji,et al. Disruption of fatty acid amide hydrolase activity prevents the effects of chronic stress on anxiety and amygdalar microstructure , 2013, Molecular Psychiatry.
[8] D. Selley,et al. Repeated Low-Dose Administration of the Monoacylglycerol Lipase Inhibitor JZL184 Retains Cannabinoid Receptor Type 1–Mediated Antinociceptive and Gastroprotective Effects , 2013, The Journal of Pharmacology and Experimental Therapeutics.
[9] B. Cravatt,et al. Dual Inhibition of Endocannabinoid Catabolic Enzymes Produces Enhanced Antiwithdrawal Effects in Morphine-Dependent Mice , 2013, Neuropsychopharmacology.
[10] A. Alpár,et al. Nerve growth factor scales endocannabinoid signaling by regulating monoacylglycerol lipase turnover in developing cholinergic neurons , 2013, Proceedings of the National Academy of Sciences.
[11] B. Lutz,et al. Biphasic Effects of Cannabinoids in Anxiety Responses: CB1 and GABAB Receptors in the Balance of GABAergic and Glutamatergic Neurotransmission , 2012, Neuropsychopharmacology.
[12] Carl Y. Saab,et al. Pain-related changes in the brain: diagnostic and therapeutic potentials , 2012, Trends in Neurosciences.
[13] M. Schmelz,et al. NGF-evoked sensitization of muscle fascia nociceptors in humans , 2012, PAIN®.
[14] M. Häring,et al. Genetic Dissection of the Role of Cannabinoid Type-1 Receptors in the Emotional Consequences of Repeated Social Stress in Mice , 2012, Neuropsychopharmacology.
[15] J. Long,et al. Dual fatty acid amide hydrolase and monoacylglycerol lipase blockade produces THC-like Morris water maze deficits in mice. , 2012, ACS chemical neuroscience.
[16] D. Nomura,et al. Endocannabinoid Hydrolysis Generates Brain Prostaglandins That Promote Neuroinflammation , 2011, Science.
[17] A. Hohmann,et al. Enhancement of endocannabinoid signaling with JZL184, an inhibitor of the 2-arachidonoylglycerol hydrolyzing enzyme monoacylglycerol lipase, produces anxiolytic effects under conditions of high environmental aversiveness in rats. , 2011, Pharmacological research.
[18] F. Basile,et al. Endocannabinoids inhibit release of nerve growth factor by inflammation-activated mast cells. , 2011, Biochemical pharmacology.
[19] L. Arendt-Nielsen,et al. Local Pain and Spreading Hyperalgesia Induced by Intramuscular Injection of Nerve Growth Factor Are Not Reduced by Local Anesthesia of the Muscle , 2011, The Clinical journal of pain.
[20] F. Luo,et al. Increased thermal and mechanical nociceptive thresholds in rats with depressive-like behaviors , 2010, Brain Research.
[21] Peter T. Nguyen,et al. Chronic monoacylglycerol lipase blockade causes functional antagonism of the endocannabinoid system , 2010, Nature Neuroscience.
[22] D. Centonze,et al. Preservation of Striatal Cannabinoid CB1 Receptor Function Correlates with the Antianxiety Effects of Fatty Acid Amide Hydrolase Inhibition , 2010, Molecular Pharmacology.
[23] J. Vlaeyen,et al. Treatments Addressing Pain-Related Fear and Anxiety in Patients with Chronic Musculoskeletal Pain: A Preliminary Review , 2010, Cognitive behaviour therapy.
[24] A. Cattaneo. Tanezumab, a recombinant humanized mAb against nerve growth factor for the treatment of acute and chronic pain. , 2010, Current opinion in molecular therapeutics.
[25] S. Waxman,et al. Unilateral focal burn injury is followed by long-lasting bilateral allodynia and neuronal hyperexcitability in spinal cord dorsal horn. , 2010, The journal of pain : official journal of the American Pain Society.
[26] D. Nomura,et al. Dual blockade of FAAH and MAGL identifies behavioral processes regulated by endocannabinoid crosstalk in vivo , 2009, Proceedings of the National Academy of Sciences.
[27] M. McCarthy,et al. Differential effects of chronic unpredictable stress on hippocampal CB1 receptors in male and female rats , 2009, Behavioural Brain Research.
[28] J. Long,et al. Blockade of 2-Arachidonoylglycerol Hydrolysis by Selective Monoacylglycerol Lipase Inhibitor 4-Nitrophenyl 4-(Dibenzo[d][1,3]dioxol-5-yl(hydroxy)methyl)piperidine-1-carboxylate (JZL184) Enhances Retrograde Endocannabinoid Signaling , 2009, Journal of Pharmacology and Experimental Therapeutics.
[29] D. Boger,et al. Blockade of Endocannabinoid-Degrading Enzymes Attenuates Neuropathic Pain , 2009, Journal of Pharmacology and Experimental Therapeutics.
[30] B. Lutz. Endocannabinoid signals in the control of emotion. , 2009, Current opinion in pharmacology.
[31] Benjamin F. Cravatt,et al. Selective blockade of 2-arachidonoylglycerol hydrolysis produces cannabinoid behavioral effects , 2008, Nature chemical biology.
[32] B. Lutz,et al. Reduced anxiety-like behaviour induced by genetic and pharmacological inhibition of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (FAAH) is mediated by CB1 receptors , 2008, Neuropharmacology.
[33] R. Mangieri,et al. Antidepressant-like Activity of the Fatty Acid Amide Hydrolase Inhibitor URB597 in a Rat Model of Chronic Mild Stress , 2007, Biological Psychiatry.
[34] M. Jann,et al. Antidepressant Agents for the Treatment of Chronic Pain and Depression , 2007, Pharmacotherapy.
[35] D. Barrett,et al. Analgesic Effects of Fatty Acid Amide Hydrolase Inhibition in a Rat Model of Neuropathic Pain , 2006, The Journal of Neuroscience.
[36] Wim E Crusio,et al. Effects of unpredictable chronic mild stress on anxiety and depression-like behavior in mice , 2006, Behavioural Brain Research.
[37] Daniele Piomelli,et al. Pharmacological profile of the selective FAAH inhibitor KDS-4103 (URB597). , 2006, CNS drug reviews.
[38] T. Unger,et al. Excitatory and modulatory effects of inflammatory cytokines and neurotrophins on mechanosensitive group IV muscle afferents in the rat , 2005, Pain.
[39] W. Farquhar-Smith,et al. A Novel Neuroimmune Mechanism in Cannabinoid-mediated Attenuation of Nerve Growth Factor–induced Hyperalgesia , 2003, Anesthesiology.
[40] K. Sluka,et al. Unilateral intramuscular injections of acidic saline produce a bilateral, long‐lasting hyperalgesia , 2001, Muscle & nerve.
[41] T. Yaksh,et al. Quantitative assessment of tactile allodynia in the rat paw , 1994, Journal of Neuroscience Methods.
[42] Shafiqur Rahman,et al. Analgesic activity of the different fractions of the aerial parts of Commelina benghalensis Linn. , 2010 .
[43] S. Gaetani,et al. Modulation of anxiety through blockade of anandamide hydrolysis , 2003, Nature Medicine.