Cell death in Alzheimer's disease evaluated by DNA fragmentation in situ

[1]  J. Wegiel,et al.  Ultrastructural studies of the cells forming amyloid in the cortical vessel wall in Alzheimer's disease , 2004, Acta Neuropathologica.

[2]  S. Ludwin,et al.  Oligodendrocyte survival in Wallerian degeneration , 2004, Acta Neuropathologica.

[3]  Khadija Iqbal,et al.  Ferritin is a component of the neuritic (senile) plaque in Alzheimer dementia , 2004, Acta Neuropathologica.

[4]  H. Wiśniewski,et al.  Alzheimer neurofibrillary tangles: Monoclonal antibodies to inherent antigen(s) , 2004, Acta Neuropathologica.

[5]  C. Cotman,et al.  Mechanisms of Neuronal Death in Alzheimer's Disease , 1996, Brain pathology.

[6]  K. Jellinger,et al.  Patterns of oligodendroglia pathology in multiple sclerosis. , 1994, Brain : a journal of neurology.

[7]  H. Hartung,et al.  Differentiation between cellular apoptosis and necrosis by the combined use of in situ tailing and nick translation techniques. , 1994, Laboratory investigation; a journal of technical methods and pathology.

[8]  C. Behl,et al.  Amyloid β peptide induces necrosis rather than apoptosis , 1994, Brain Research.

[9]  C. Behl,et al.  Amyloid beta peptide induces necrosis rather than apoptosis. , 1994, Brain research.

[10]  L. Schwartz,et al.  Programmed cell death, apoptosis and killer genes. , 1993, Immunology today.

[11]  H. Lassmann,et al.  Apoptosis of T lymphocytes in experimental autoimmune encephalomyelitis. Evidence for programmed cell death as a mechanism to control inflammation in the brain. , 1993, The American journal of pathology.

[12]  H. Lassmann,et al.  Detection of DNA fragmentation in apoptosis: application of in situ nick translation to cell culture systems and tissue sections. , 1993, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[13]  G. Forloni,et al.  Apoptosis mediated neurotoxicity induced by chronic application of beta amyloid fragment 25-35. , 1993, Neuroreport.

[14]  S. Mirra,et al.  Making the diagnosis of Alzheimer's disease. A primer for practicing pathologists. , 1993, Archives of pathology & laboratory medicine.

[15]  D. Price,et al.  Synapse loss in the temporal lobe in Alzheimer's disease , 1993, Annals of neurology.

[16]  D. Walker,et al.  Microglia in degenerative neurological disease , 1993, Glia.

[17]  J. H. Dierendonck,et al.  A new method to detect apoptosis in paraffin sections: in situ end-labeling of fragmented DNA. , 1993, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[18]  S. Ben‐Sasson,et al.  Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation , 1992, The Journal of cell biology.

[19]  G. Cole,et al.  Vitamin E protects nerve cells from amyloid βprotein toxicity , 1992 .

[20]  S. Hoyer Oxidative energy metabolism in Alzheimer brain. Studies in early-onset and late-onset cases. , 1992, Molecular and chemical neuropathology.

[21]  K. Jellinger,et al.  Synaptic pathology in alzheimer's disease: Immunological data for markers of synaptic and large dense-core vesicles , 1992, Neuroscience.

[22]  H. Wiśniewski,et al.  Detection of point mutations in codon 331 of mitochondrial NADH dehydrogenase subunit 2 in Alzheimer's brains. , 1992, Biochemical and biophysical research communications.

[23]  M. Mesulam,et al.  β-Amyloid and the pathogenesis of alzheimer's disease , 1991 .

[24]  C. Cotman,et al.  Beta-amyloid increases neuronal susceptibility to injury by glucose deprivation. , 1991, Neuroreport.

[25]  D. Salmon,et al.  Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment , 1991, Annals of neurology.

[26]  J. Cohen,et al.  Programmed cell death in the immune system. , 1991, Advances in immunology.

[27]  C. Cotman,et al.  β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage , 1990, Brain Research.

[28]  D. Kirschner,et al.  Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides. , 1990, Science.

[29]  S. DeKosky,et al.  Synapse loss in frontal cortex biopsies in Alzheimer's disease: Correlation with cognitive severity , 1990, Annals of neurology.

[30]  E. Mullaart,et al.  Increased levels of DNA breaks in cerebral cortex of Alzheimer's disease patients , 1990, Neurobiology of Aging.

[31]  H. Lassmann,et al.  Differential Ultrastructural Localization of Myelin Basic Protein, Myelin/Oligodendroglial Glycoprotein, and 2′,3′‐Cyclic Nucleotide 3′‐Phosphodiesterase in the CNS of Adult Rats , 1989, Journal of neurochemistry.

[32]  S. Iseki DNA strand breaks in rat tissues as detected by in situ nick translation. , 1986, Experimental cell research.

[33]  Z. Khachaturian Diagnosis of Alzheimer's disease. , 1985, Archives of neurology.

[34]  D. Mann,et al.  Some morphometric observations on the cerebral cortex and hippocampus in presenile Alzheimer's disease, senile dementia of Alzheimer type and Down's syndrome in middle age , 1985, Journal of the Neurological Sciences.

[35]  M. Raff,et al.  Effects of neonatal transection on glial cell development in the rat optic nerve: evidence that the oligodendrocyte-type 2 astrocyte cell lineage depends on axons for its survival , 1984, Journal of neurocytology.

[36]  R. Tarone,et al.  RADIOSENSITIVITY IN ALZHEIMER DISEASE AND PARKINSON DISEASE , 1983, The Lancet.

[37]  A. Wyllie,et al.  Cell death: the significance of apoptosis. , 1980, International review of cytology.

[38]  A. Alzheimer über eigenartige Krankheitsfälle des späteren Alters , 1911 .