Biophysical and behavioral correlates of memory storage, degradation, and reactivation.
暂无分享,去创建一个
[1] D L Alkon,et al. Sequential modification of membrane currents with classical conditioning. , 1988, Biophysical journal.
[2] D. Alkon,et al. Cellular mechanisms of learning, memory, and information storage. , 1985, Annual review of psychology.
[3] Norman E. Spear,et al. The processing of memories : forgetting and retention , 1980 .
[4] J. H. Schwartz,et al. Molecular mechanisms for memory: second-messenger induced modifications of protein kinases in nerve cells. , 1987, Annual review of neuroscience.
[5] D. L. Alkon,et al. Membrane changes in a single photoreceptor cause associative learning in Hermissenda. , 1983, Science.
[6] D. Alkon,et al. Classical conditioning of Hermissenda: origin of a new response , 1986, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[7] G. Keppel,et al. Design and Analysis: A Researcher's Handbook , 1976 .
[8] D L Alkon,et al. Primary changes of voltage responses during retention of associative learning. , 1982, Journal of neurophysiology.
[9] Daniel L. Alkon,et al. Extinction of associative learning in Hermissenda: Behavior and neural correlates , 1984, Behavioural Brain Research.
[10] T. Alexinsky,et al. Differential effects of several retrieval cues over time: Evidence for time-dependent reorganization of memory , 1989 .
[11] T. Crow,et al. Light paired with serotonin in vivo produces both short- and long-term enhancement of generator potentials of identified B-photoreceptors in Hermissenda , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[12] R. Nicoll,et al. The Role of Calcium in Long‐Term Potentiation , 1989, Annals of the New York Academy of Sciences.
[13] R. R. Miller,et al. Retrieval variability: sources and consequences. , 1986, The American journal of psychology.
[14] Y Nishizuka,et al. Activation of calcium and phospholipid-dependent protein kinase by diacylglycerol, its possible relation to phosphatidylinositol turnover. , 1980, The Journal of biological chemistry.
[15] D L Alkon,et al. Reduction of two voltage-dependent K+ currents mediates retention of a learned association. , 1985, Behavioral and neural biology.
[16] D L Alkon,et al. Pavlovian conditioning of distinct components of Hermissenda's responses to rotation. , 1990, Behavioral and neural biology.
[17] E. Kandel,et al. Classical conditioning and sensitization share aspects of the same molecular cascade in Aplysia. , 1983, Cold Spring Harbor symposia on quantitative biology.
[18] D L Alkon,et al. Classical conditioning induces long-term translocation of protein kinase C in rabbit hippocampal CA1 cells. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[19] D. L. Alkon,et al. Membrane depolarization accumulates during acquisition of an associative behavioral change. , 1980, Science.
[20] D L Alkon,et al. Regulation of Hermissenda K+ Channels by Cytoplasmic and Membrane‐Associated C‐Kinase , 1988, Journal of neurochemistry.
[21] J. Farley,et al. Protein kinase C activation induces conductance changes in Hermissenda photoreceptors like those seen in associative learning , 1986, Nature.
[22] D L Alkon,et al. Light- and voltage-dependent increases of calcium ion concentration in molluscan photoreceptors. , 1984, Journal of neurophysiology.
[23] Ralph R. Miller. Classical conditioning: The new hyperbole , 1989, Behavioral and Brain Sciences.
[24] Joseph Farley,et al. Associative training results in persistent reductions in a calcium-activated potassium current in Hermissenda type B photoreceptors , 1988 .
[25] J. Farley,et al. Contingency learning and causal detection in Hermissenda: I. Behavior. , 1987, Behavioral neuroscience.
[26] G. Lynch,et al. Long-term potentiation: Persisting problems and recent results , 1988, Brain Research Bulletin.
[27] M. Bouton. Slow reacquisition following the extinction of conditioned suppression , 1986 .
[28] J. Byrne,et al. Associative conditioning of single sensory neurons suggests a cellular mechanism for learning. , 1983, Science.
[29] D. Alkon. Calcium-mediated reduction of ionic currents: a biophysical memory trace. , 1984, Science.
[30] D. Alkon,et al. Change in a specific phosphoprotein band following associative learning in Hermissenda , 1981, Nature.
[31] Developmental Changes in the Time-Dependent Nature of Memory Retrieval , 1990 .
[32] D. Alkon,et al. GABA-induced potentiation of neuronal excitability occurs during contiguous pairings with intracellular calcium elevation , 1991, Brain Research.
[33] D. Alkon,et al. Associative Behavioral Modification in Hermissenda: Cellular Correlates , 1980, Science.
[34] D L Alkon,et al. Acquisition of conditioned associations in Hermissenda: additive effects of contiguity and the forward interstimulus interval. , 1990, Behavioral neuroscience.
[35] T. Crow,et al. Retention of an associative behavioral change in Hermissenda. , 1978, Science.
[36] D. Alkon,et al. Regulation of short-term associative memory by calcium-dependent protein kinase , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[37] J. Wickens. Electrically coupled but chemically isolated synapses: Dendritic spines and calcium in a rule for synaptic modification , 1988, Progress in Neurobiology.
[38] R. Mowrer,et al. An extinction trial as a reminder treatment following electroconvulsive shock , 1980 .
[39] Joseph Farley,et al. Associative learning changes intrinsic to Hermissenda type A photoreceptors. , 1990, Behavioral neuroscience.
[40] D. Lovinger,et al. Translocation of protein kinase C activity may mediate hippocampal long-term potentiation. , 1986, Science.
[41] J. Byrne. Cellular analysis of associative learning. , 1987, Physiological reviews.
[42] T. Crow. Cellular and molecular analysis of associative learning and memory in Hermissenda , 1988, Trends in Neurosciences.