Pituitary Cells Excitable.
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[1] S. Rawlings. Pituitary adenylate cyclase-activating polypeptide regulates [Ca2+]i and electrical activity in pituitary cells through cell type-specific mechanisms , 1996, Trends in Endocrinology & Metabolism.
[2] W. Schlegel,et al. c-fos mRNA and FOS protein expression is induced by Ca2+ influx in GH3B6 pituitary cells. , 1996, Journal of molecular endocrinology.
[3] H. Horstmann,et al. Docked granules, the exocytic burst, and the need for ATP hydrolysis in endocrine cells , 1995, Neuron.
[4] Y. Kuryshev,et al. Three high threshold calcium channel subtypes in rat corticotropes. , 1995, Endocrinology.
[5] N. Mons,et al. Adenylyl cyclases and the interaction between calcium and cAMP signalling , 1995, Nature.
[6] J. Hescheler,et al. Stimulation of single L‐type calcium channels in rat pituitary GH3 cells by thyrotropin‐releasing hormone. , 1995, The EMBO journal.
[7] M. L Vitale,et al. Chromaffin cell cortical actin network dynamics control the size of the release-ready vesicle pool and the initial rate of exocytosis , 1995, Neuron.
[8] W. Schlegel,et al. Cytosolic Ca2+ of excitable pituitary cells at resting potentials is controlled by steady state Ca2+ currents sensitive to dihydropyridines. , 1994, The Journal of biological chemistry.
[9] J. Meldolesi,et al. Molecular and cellular physiology of intracellular calcium stores. , 1994, Physiological reviews.
[10] L. Orci,et al. Differential expression of gap junction connexins in endocrine and exocrine glands. , 1993, Endocrinology.
[11] J. Corcuff,et al. Multiple cytosolic calcium signals and membrane electrical events evoked in single arginine vasopressin-stimulated corticotrophs. , 1993, The Journal of biological chemistry.
[12] D. Golan,et al. Mechanism of spontaneous intracellular calcium fluctuations in single GH4C1 rat pituitary cells. , 1993, The Biochemical journal.
[13] L. Stryer,et al. Range of messenger action of calcium ion and inositol 1,4,5-trisphosphate. , 1992, Science.
[14] P. Mcnaughton. Fundamental Properties of the Na‐Ca Exchange , 1991 .
[15] A. Shcherbatko,et al. Enzymatic Gating of Voltage‐Activated Calcium Channels , 1991, Annals of the New York Academy of Sciences.
[16] H. Scherübl,et al. Steady-state currents through voltage-dependent, dihydropyridine-sensitive Ca2+ channels in GH3 pituitary cells , 1991, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[17] A. Tabarin,et al. Spontaneous and corticotropin-releasing factor-induced cytosolic calcium transients in corticotrophs. , 1991, Endocrinology.
[18] Richard E. White,et al. Somatostatin stimulates Ca2+-activated K+ channels through protein dephosphorylation , 1991, Nature.
[19] J. Vincent,et al. Dopamine inhibits two characterized voltage-dependent calcium currents in identified rat lactotroph cells. , 1990, Endocrinology.
[20] J. Barker,et al. Somatostatin blocks Ca2+ action potential activity in prolactin-secreting pituitary tumor cells through coordinate actions on K+ and Ca2+ conductances. , 1988, Endocrinology.
[21] E. Ogata,et al. Requirement of GTP on somatostatin-induced K+ current in human pituitary tumor cells. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[22] C. Wollheim,et al. Oscillations of cytosolic Ca2+ in pituitary cells due to action potentials , 1987, Nature.
[23] J. Barker,et al. Intracellular Ca2+-dependent protein kinase C activation mimics delayed effects of thyrotropin-releasing hormone on clonal pituitary cell excitability. , 1987, Endocrinology.
[24] R. Eckert,et al. Voltage-activated calcium channels that must be phosphorylated to respond to membrane depolarization. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[25] A. Brown,et al. Membrane currents of identified isolated rat corticotropes and gonadotropes. , 1987, The American journal of physiology.
[26] S. Ozawa,et al. Electrophysiology of excitable endocrine cells. , 1986, Physiological reviews.
[27] W. Mason,et al. [15] Techniques for studying the role of electrical activity in control of secretion by normal anterior pituitary cells , 1986 .
[28] A. Cm,et al. Two distinct populations of calcium channels in a clonal line of pituitary cells. , 1985 .
[29] M B Jackson,et al. Action potentials and membrane ion channels in clonal anterior pituitary cells. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[30] J. Vincent,et al. Dopamine inhibition of action potentials in a prolactin secreting cell line is modulated by oestrogen , 1979, Nature.
[31] M. Endo,et al. Calcium release from the sarcoplasmic reticulum. , 1977, Physiological reviews.
[32] W. Douglas,et al. Stimulus‐secretion coupling: the concept and clues from chromaffin and other cells , 1968, British journal of pharmacology.
[33] Alexander Sandow,et al. Excitation-Contraction Coupling in Muscular Response * , 1952, The Yale journal of biology and medicine.