Pituitary Cells Excitable.

Since 1975, endocrine pituitary cells have been known to be excitable neuronlike cells. Using powerful single-cell approaches, in particular the patch clamp electrophysiological recording technique and the mon- itoring of Ca 2+ with fluorescent probes, solid evidence has been pro- vided in the last 10 years that intracellular

[1]  S. Rawlings Pituitary adenylate cyclase-activating polypeptide regulates [Ca2+]i and electrical activity in pituitary cells through cell type-specific mechanisms , 1996, Trends in Endocrinology & Metabolism.

[2]  W. Schlegel,et al.  c-fos mRNA and FOS protein expression is induced by Ca2+ influx in GH3B6 pituitary cells. , 1996, Journal of molecular endocrinology.

[3]  H. Horstmann,et al.  Docked granules, the exocytic burst, and the need for ATP hydrolysis in endocrine cells , 1995, Neuron.

[4]  Y. Kuryshev,et al.  Three high threshold calcium channel subtypes in rat corticotropes. , 1995, Endocrinology.

[5]  N. Mons,et al.  Adenylyl cyclases and the interaction between calcium and cAMP signalling , 1995, Nature.

[6]  J. Hescheler,et al.  Stimulation of single L‐type calcium channels in rat pituitary GH3 cells by thyrotropin‐releasing hormone. , 1995, The EMBO journal.

[7]  M. L Vitale,et al.  Chromaffin cell cortical actin network dynamics control the size of the release-ready vesicle pool and the initial rate of exocytosis , 1995, Neuron.

[8]  W. Schlegel,et al.  Cytosolic Ca2+ of excitable pituitary cells at resting potentials is controlled by steady state Ca2+ currents sensitive to dihydropyridines. , 1994, The Journal of biological chemistry.

[9]  J. Meldolesi,et al.  Molecular and cellular physiology of intracellular calcium stores. , 1994, Physiological reviews.

[10]  L. Orci,et al.  Differential expression of gap junction connexins in endocrine and exocrine glands. , 1993, Endocrinology.

[11]  J. Corcuff,et al.  Multiple cytosolic calcium signals and membrane electrical events evoked in single arginine vasopressin-stimulated corticotrophs. , 1993, The Journal of biological chemistry.

[12]  D. Golan,et al.  Mechanism of spontaneous intracellular calcium fluctuations in single GH4C1 rat pituitary cells. , 1993, The Biochemical journal.

[13]  L. Stryer,et al.  Range of messenger action of calcium ion and inositol 1,4,5-trisphosphate. , 1992, Science.

[14]  P. Mcnaughton Fundamental Properties of the Na‐Ca Exchange , 1991 .

[15]  A. Shcherbatko,et al.  Enzymatic Gating of Voltage‐Activated Calcium Channels , 1991, Annals of the New York Academy of Sciences.

[16]  H. Scherübl,et al.  Steady-state currents through voltage-dependent, dihydropyridine-sensitive Ca2+ channels in GH3 pituitary cells , 1991, Proceedings of the Royal Society of London. Series B: Biological Sciences.

[17]  A. Tabarin,et al.  Spontaneous and corticotropin-releasing factor-induced cytosolic calcium transients in corticotrophs. , 1991, Endocrinology.

[18]  Richard E. White,et al.  Somatostatin stimulates Ca2+-activated K+ channels through protein dephosphorylation , 1991, Nature.

[19]  J. Vincent,et al.  Dopamine inhibits two characterized voltage-dependent calcium currents in identified rat lactotroph cells. , 1990, Endocrinology.

[20]  J. Barker,et al.  Somatostatin blocks Ca2+ action potential activity in prolactin-secreting pituitary tumor cells through coordinate actions on K+ and Ca2+ conductances. , 1988, Endocrinology.

[21]  E. Ogata,et al.  Requirement of GTP on somatostatin-induced K+ current in human pituitary tumor cells. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[22]  C. Wollheim,et al.  Oscillations of cytosolic Ca2+ in pituitary cells due to action potentials , 1987, Nature.

[23]  J. Barker,et al.  Intracellular Ca2+-dependent protein kinase C activation mimics delayed effects of thyrotropin-releasing hormone on clonal pituitary cell excitability. , 1987, Endocrinology.

[24]  R. Eckert,et al.  Voltage-activated calcium channels that must be phosphorylated to respond to membrane depolarization. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[25]  A. Brown,et al.  Membrane currents of identified isolated rat corticotropes and gonadotropes. , 1987, The American journal of physiology.

[26]  S. Ozawa,et al.  Electrophysiology of excitable endocrine cells. , 1986, Physiological reviews.

[27]  W. Mason,et al.  [15] Techniques for studying the role of electrical activity in control of secretion by normal anterior pituitary cells , 1986 .

[28]  A. Cm,et al.  Two distinct populations of calcium channels in a clonal line of pituitary cells. , 1985 .

[29]  M B Jackson,et al.  Action potentials and membrane ion channels in clonal anterior pituitary cells. , 1983, Proceedings of the National Academy of Sciences of the United States of America.

[30]  J. Vincent,et al.  Dopamine inhibition of action potentials in a prolactin secreting cell line is modulated by oestrogen , 1979, Nature.

[31]  M. Endo,et al.  Calcium release from the sarcoplasmic reticulum. , 1977, Physiological reviews.

[32]  W. Douglas,et al.  Stimulus‐secretion coupling: the concept and clues from chromaffin and other cells , 1968, British journal of pharmacology.

[33]  Alexander Sandow,et al.  Excitation-Contraction Coupling in Muscular Response * , 1952, The Yale journal of biology and medicine.