Cholecystokinin receptors do not mediate the suppression of food-motivated behavior by lipopolysaccharide and interleukin-1 beta in mice
暂无分享,去创建一个
[1] Akil,et al. Evidence that Cholecystokinin Receptors are not Involved in the Hypothalamic‐Pituitary‐Adrenal Response to Intraperitoneal Administration of Interleukin‐1β , 1999, Journal of neuroendocrinology.
[2] S. Maier,et al. Interleukin-1β in Immune Cells of the Abdominal Vagus Nerve: a Link between the Immune and Nervous Systems? , 1999, The Journal of Neuroscience.
[3] S. Maier,et al. Bacterial Endotoxin Induces Fos Immunoreactivity in Primary Afferent Neurons of the Vagus Nerve , 1998, Neuroimmunomodulation.
[4] Στυλιανή Τζιαφέρη,et al. Interleukin-1 induces c-Fos immunoreactivity in primary afferent neurons of the vagus nerve , 1998, Brain Research.
[5] R. Dantzer,et al. Systemic Capsaicin Pretreatment Fails to Block the Decrease in Food-Motivated Behavior Induced by Lipopolysaccharide and Interleukin-1β , 1997, Brain Research Bulletin.
[6] S. Maier,et al. Vagal Paraganglia Bind Biotinylated Interleukin-1 Receptor Antagonist: A Possible Mechanism for Immune-to-Brain Communication , 1997, Brain Research Bulletin.
[7] R. Dantzer,et al. Cholecystokinin Receptors do not Mediate the Behavioral Effects of Lipopolysaccharide in Mice , 1997, Physiology & Behavior.
[8] T. Lundeberg,et al. Interleukin-1β sensitizes the response of the gastric vagal afferent to cholecystokinin in rat , 1997, Neuroscience Letters.
[9] T. Lundeberg,et al. Interleukin-1 increases activity of the gastric vagal afferent nerve partly via stimulation of type A CCK receptor in anesthetized rats. , 1997, Journal of the autonomic nervous system.
[10] R. Dantzer,et al. Mechanisms of sickness-induced decreases in food-motivated behavior , 1996, Neuroscience & Biobehavioral Reviews.
[11] A. Niijima,et al. The afferent discharges from sensors for interleukin 1 beta in the hepatoportal system in the anesthetized rat. , 1996, Journal of the autonomic nervous system.
[12] R. Dantzer,et al. Vagotomy blocks behavioural effects of interleukin-1 injected via the intraperitoneal route but not via other systemic routes. , 1996, Neuroreport.
[13] R. Dantzer,et al. Lipopolysaccharide and Interleukin-1 Depress Food-Motivated Behavior in Mice by a Vagal-Mediated Mechanism , 1995, Brain, Behavior, and Immunity.
[14] S. Maier,et al. Illness-induced hyperalgesia is mediated by spinal neuropeptides and excitatory amino acids , 1994, Brain Research.
[15] J. Walsh,et al. Cholecystokinin suppresses food intake by a nonendocrine mechanism in rats. , 1994, The American journal of physiology.
[16] R. Dantzer,et al. A behaviorally active dose of lipopolysaccharide increases sensory neuropeptides levels in mouse spinal cord , 1994, Neuroscience Letters.
[17] W. Langhans,et al. Does a learned taste aversion contribute to the anorectic effect of bacterial lipopolysaccharide? , 1993, Physiology & Behavior.
[18] D. Farningham,et al. Satiety signals in sheep: Involvement of CCK, propionate, and vagal CCK binding sites , 1993, Physiology & Behavior.
[19] D. McCarthy,et al. The role of cholecystokinin in interleukin-1-induced anorexia , 1993, Physiology & Behavior.
[20] P. Ghezzi,et al. Cytokine down-regulation in endotoxin tolerance. , 1993, European cytokine network.
[21] R. Reidelberger. Abdominal vagal mediation of the satiety effects of exogenous and endogenous cholecystokinin in rats. , 1992, The American journal of physiology.
[22] S. Matsukura,et al. Stimulation of cholecystokinin (CCK) release from superfused rat hypothalamo-neurohypophyseal complexes by interleukin-1 (IL-1) , 1992, Brain Research.
[23] J. Mercer,et al. Selectivity of cholecystokinin (CCK) receptor antagonists, MK-329 and L-365,260, for axonally-transported CCK binding sites on the rat vagus nerve , 1992, Neuroscience Letters.
[24] C. Plata-salamán. Immunoregulators in the nervous system , 1991, Neuroscience & Biobehavioral Reviews.
[25] S. Cooper,et al. Multiple cholecystokinin (CCK) receptors and CCK-monoamine interactions are instrumental in the control of feeding , 1990, Physiology & Behavior.
[26] T. Shiraishi. CCK as a central satiety factor: Behavioral and electrophysiological evidence , 1990, Physiology & Behavior.
[27] R. J. Crosby,et al. Central and peripheral vagal transport of cholecystokinin binding sites occurs in afferent fibers , 1990, Brain Research.
[28] Toshinori Mori,et al. Actions of interferonα and interleukin-1β on the glucose-responsive neurons in the ventromedial hypothalamus , 1990, Brain Research Bulletin.
[29] J. Lin,et al. Effects of protein binding and experimental disease states on brain uptake of benzodiazepines in rats. , 1990, The Journal of pharmacology and experimental therapeutics.
[30] T. Yaksh,et al. Sites in the brain at which cholecystokinin octapeptide (CCK-8) acts to suppress feeding in rats: A mapping study , 1990, Neuropharmacology.
[31] S. Iversen,et al. Evidence that decreased feeding induced by systemic injection of cholecystokinin is mediated by CCK-A receptors. , 1989, European journal of pharmacology.
[32] N. Mrosovsky,et al. Anorexic effects of interleukin 1 in the rat. , 1989, The American journal of physiology.
[33] N. Rothwell,et al. Comparison of the effects of several endotoxin preparations on body temperature and metabolic rate in the rat. , 1989, Canadian journal of physiology and pharmacology.
[34] R. Chang,et al. A new potent and selective non-peptide gastrin antagonist and brain cholecystokinin receptor (CCK-B) ligand: L-365,260. , 1989, European journal of pharmacology.
[35] S. Zuckerman,et al. Differential regulation of lipopolysaccharide‐induced interleukin 1 and tumor necrosis factor synthesis: effects of endogenous and exogenous glucocorticoids and the role of the pituitary‐adrenal axis , 1989, European journal of immunology.
[36] C. Plata-salamán,et al. Tumor necrosis factor and interleukin-1β: suppression of food intake by direct action in the central nervous system , 1988, Brain Research.
[37] H. Besedovsky,et al. Interleukin 1 affects glucose homeostasis. , 1987, The American journal of physiology.
[38] R. Pullen,et al. Penetration of diazepam and the non‐peptide CCK antagonist, L‐364, 718, into rat brain , 1987, The Journal of pharmacy and pharmacology.
[39] J. Morley,et al. Modulation of memory processing by cholecystokinin: dependence on the vagus nerve. , 1987, Science.
[40] R. Chang,et al. Biochemical and pharmacological characterization of an extremely potent and selective nonpeptide cholecystokinin antagonist. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[41] J. Crawley,et al. Paraventricular nucleus lesions abolish the inhibition of feeding induced by systemic cholecystokinin , 1985, Peptides.
[42] J. A. Deutsch,et al. An antiemetic is antidotal to the satiety effects of cholecystokinin , 1985, Nature.
[43] A. Vander,et al. The role of fever in appetite suppression after endotoxin administration. , 1984, The American journal of clinical nutrition.
[44] M. Palkovits,et al. Cholecystokinin in the nucleus of the solitary tract of the rat: evidence for its vagal origin , 1982, Brain Research.
[45] G. P. Smith,et al. Abdominal vagotomy blocks the satiety effect of cholecystokinin in the rat. , 1981, Science.