Ultraviolet-sensitive syndrome cells are defective in transcription-coupled repair of cyclobutane pyrimidine dimers.

[1]  A. Avrutskaya,et al.  Differential involvement of the human mismatch repair proteins, hMLH1 and hMSH2, in transcription-coupled repair. , 1997, Cancer research.

[2]  M. Smerdon,et al.  Transcription-coupled repair in RNA polymerase I-transcribed genes of yeast , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[3]  P. Hanawalt,et al.  Effect of Thymine Glycol on Transcription Elongation by T7 RNA Polymerase and Mammalian RNA Polymerase II* , 2001, The Journal of Biological Chemistry.

[4]  C. Ingles,et al.  A compromised yeast RNA Polymerase II enhances UV sensitivity in the absence of global genome nucleotide excision repair , 2001, Zeitschrift für Induktive Abstammungs- und Vererbungslehre.

[5]  J. Hoeijmakers,et al.  XAB2, a Novel Tetratricopeptide Repeat Protein Involved in Transcription-coupled DNA Repair and Transcription* , 2000, The Journal of Biological Chemistry.

[6]  M. Perricaudet,et al.  BRCA1 and BRCA2 are necessary for the transcription-coupled repair of the oxidative 8-oxoguanine lesion in human cells. , 2000, Cancer research.

[7]  M. Barton,et al.  UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[8]  Philip C. Hanawalt,et al.  DNA repair: The bases for Cockayne syndrome , 2000, Nature.

[9]  M. Yamaizumi,et al.  Reinvestigation of the classification of five cell strains of xeroderma pigmentosum group E with reclassification of three of them. , 2000, The Journal of investigative dermatology.

[10]  A. Sarasin,et al.  RETRACTED: Transcription-Coupled Repair of 8-oxoGuanine Requirement for XPG, TFIIH, and CSB and Implications for Cockayne Syndrome , 2000, Cell.

[11]  J. Hoeijmakers,et al.  Nucleotide excision repair and human syndromes. , 2000, Carcinogenesis.

[12]  P. Hanawalt,et al.  Structural Characterization of RNA Polymerase II Complexes Arrested by a Cyclobutane Pyrimidine Dimer in the Transcribed Strand of Template DNA* , 1999, The Journal of Biological Chemistry.

[13]  M. Nitta,et al.  High sensitivity of the ultraviolet-induced p53 response in ultraviolet-sensitive syndrome, a photosensitive disorder with a putative defect in deoxyribonucleic acid repair of actively transcribed genes. , 1999, Mutation research.

[14]  P. Hanawalt,et al.  Transcription-Coupled DNA Repair: Which Lesions? Which Diseases? , 1999 .

[15]  T. Lindahl,et al.  Base excision repair of oxidative DNA damage activated by XPG protein. , 1999, Molecular cell.

[16]  B. Koller,et al.  BRCA1 required for transcription-coupled repair of oxidative DNA damage. , 1998, Science.

[17]  A. Avrutskaya,et al.  Requirement for DNA mismatch repair proteins in the transcription-coupled repair of thymine glycols in Saccharomyces cerevisiae. , 1998, Mutation research.

[18]  P. Hanawalt,et al.  Expression of Wild-type p53 Is Required for Efficient Global Genomic Nucleotide Excision Repair in UV-irradiated Human Fibroblasts* , 1997, The Journal of Biological Chemistry.

[19]  S. A. Leadon,et al.  A common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: implications for a second XPG function. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[20]  S. Clarkson,et al.  Defective Transcription-Coupled Repair of Oxidative Base Damage in Cockayne Syndrome Patients from XP Group G , 1997, Science.

[21]  R. Wood,et al.  Open complex formation around a lesion during nucleotide excision repair provides a structure for cleavage by human XPG protein , 1997, The EMBO journal.

[22]  J. Chiu,et al.  Regulation and activities of alpha-fetoprotein. , 1997, Critical reviews in eukaryotic gene expression.

[23]  M. Ljungman,et al.  Blockage of RNA polymerase as a possible trigger for u.v. light-induced apoptosis. , 1996, Oncogene.

[24]  P. Hanawalt,et al.  Mismatch repair mutants in yeast are not defective in transcription-coupled DNA repair of UV-induced DNA damage. , 1996, Genetics.

[25]  M. Yamaizumi,et al.  Clinical characteristics of three patients with UVS syndrome, a photosensitive disorder with defective DNA repair , 1996, The British journal of dermatology.

[26]  C. Boland,et al.  Transcription-Coupled Repair Deficiency and Mutations in Human Mismatch Repair Genes , 1996, Science.

[27]  E. Friedberg,et al.  Rodent complementation group 8 (ERCC8) corresponds to Cockayne syndrome complementation group A. , 1996, Mutation research.

[28]  I. Mellon,et al.  Products of DNA mismatch repair genes mutS and mutL are required for transcription-coupled nucleotide-excision repair of the lactose operon in Escherichia coli. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[29]  M. Yamaizumi,et al.  UVs syndrome, a new general category of photosensitive disorder with defective DNA repair, is distinct from xeroderma pigmentosum variant and rodent complementation group I. , 1995, American journal of human genetics.

[30]  P. Hanawalt,et al.  Determination of Damage and Repair in Specific DNA Sequences , 1995 .

[31]  T. Sugano,et al.  U.v.-induced nuclear accumulation of p53 is evoked through DNA damage of actively transcribed genes independent of the cell cycle. , 1994, Oncogene.

[32]  P. Hanawalt,et al.  Transcript cleavage by RNA polymerase II arrested by a cyclobutane pyrimidine dimer in the DNA template. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[33]  P. Cooper,et al.  Defective Repair of Ionizing Radiation Damage in Cockayne's Syndrome and Xeroderma Pigmentosum Group G a , 1994, Annals of the New York Academy of Sciences.

[34]  R. Wood,et al.  Isolation of active recombinant XPG protein, a human DNA repair endonuclease. , 1994, The Journal of biological chemistry.

[35]  P. Hanawalt,et al.  Preferential repair of ultraviolet light‐induced dna damage in the transcribed strand of the human p53 gene , 1994, Molecular carcinogenesis.

[36]  P. Hanawalt,et al.  Repair and Transcription: Collision or collusion? , 1994, Current Biology.

[37]  M. Yamaizumi,et al.  A new UV-sensitive syndrome not belonging to any complementation groups of xeroderma pigmentosum or Cockayne syndrome: siblings showing biochemical characteristics of Cockayne syndrome without typical clinical manifestations , 1994 .

[38]  L. Mullenders,et al.  Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells. , 1993, Nucleic acids research.

[39]  T. Lindahl,et al.  DNA excision-repair defect of xeroderma pigmentosum prevents removal of a class of oxygen free radical-induced base lesions. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[40]  A. Sancar,et al.  Molecular mechanism of transcription-repair coupling. , 1993, Science.

[41]  P. Hanawalt,et al.  Increased UV resistance of a xeroderma pigmentosum revertant cell line is correlated with selective repair of the transcribed strand of an expressed gene , 1993, Molecular and cellular biology.

[42]  P. Hanawalt,et al.  Stranded in an active gene , 1993, Current Biology.

[43]  J. Hoeijmakers,et al.  ERCC6, a member of a subfamily of putative helicases, is involved in Cockayne's syndrome and preferential repair of active genes , 1992, Cell.

[44]  D. Lawrence,et al.  Strand-selective repair of DNA damage in the yeast GAL7 gene requires RNA polymerase II. , 1992, The Journal of biological chemistry.

[45]  P. Hanawalt,et al.  Preferential repair of cyclobutane pyrimidine dimers in the transcribed strand of a gene in yeast chromosomes and plasmids is dependent on transcription. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[46]  L. Mullenders,et al.  (6-4) photoproducts and not cyclobutane pyrimidine dimers are the main UV-induced mutagenic lesions in Chinese hamster cells. , 1992, Mutation research.

[47]  P. Hanawalt,et al.  The genetic defect in the Chinese hamster ovary cell mutant UV61 permits moderate selective repair of cyclobutane pyrimidine dimers in an expressed gene. , 1991, Mutation research.

[48]  O. Nikaido,et al.  SIMULTANEOUS ESTABLISHMENT OF MONOCLONAL ANTIBODIES SPECIFIC FOR EITHER CYCLOBUTANE PYRIMIDINE DIMER OR (6‐4)PHOTOPRODUCT FROM THE SAME MOUSE IMMUNIZED WITH ULTRAVIOLET‐IRRADIATED DNA , 1991, Photochemistry and photobiology.

[49]  L. Mullenders,et al.  Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes , 1991, Molecular and cellular biology.

[50]  B. Vogelstein,et al.  Suppression of human colorectal carcinoma cell growth by wild-type p53. , 1990, Science.

[51]  L. Mullenders,et al.  The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[52]  M. Smerdon,et al.  Site-specific DNA repair at the nucleosome level in a yeast minichromosome , 1990, Cell.

[53]  O. Nikaido,et al.  Base sequence specificity of a monoclonal antibody binding to (6-4)photoproducts. , 1990, Mutation research.

[54]  P. Hanawalt,et al.  Induction of the Escherichia coli lactose operon selectively increases repair of its transcribed DNA strand , 1989, Nature.

[55]  P C Hanawalt,et al.  Heterogeneous DNA damage and repair in the mammalian genome. , 1987, Cancer research.

[56]  P. Hanawalt,et al.  Selective removal of transcription-blocking DNA damage from the transcribed strand of the mammalian DHFR gene , 1987, Cell.

[57]  D. Nelson,et al.  The human mid‐size neurofilament subunit: a repeated protein sequence and the relationship of its gene to the intermediate filament gene family. , 1987, The EMBO journal.

[58]  C. Will,et al.  5-Fluorouracil augmentation of dihydrofolate reductase gene transcripts containing intervening sequences in methotrexate-resistant KB cells. , 1986, Molecular pharmacology.

[59]  A. Steven,et al.  The molecular biology of intermediate filaments , 1985, Cell.

[60]  P. Hanawalt,et al.  DNA repair in an active gene: Removal of pyrimidine dimers from the DHFR gene of CHO cells is much more efficient than in the genome overall , 1985, Cell.

[61]  P. Hanawalt,et al.  Sensitive determination of pyrimidine dimers in DNA of UV-irradiated mammalian cells. Introduction of T4 endonuclease V into frozen and thawed cells. , 1981, Mutation research.

[62]  E. Lazarides.  Intermediate filaments as mechanical integrators of cellular space , 1980, Nature.