Endoplasmic reticulum calcium pool depletion-induced apoptosis is coupled with activation of the death receptor 5 pathway

[1]  Qin He,et al.  Sulindac sulfide-induced apoptosis involves death receptor 5 and the caspase 8-dependent pathway in human colon and prostate cancer cells. , 2001, Cancer research.

[2]  A. Fornace,et al.  Death and decoy receptors and p53-mediated apoptosis , 2000, Leukemia.

[3]  V. Dixit,et al.  Characterization of Calcium Release-activated Apoptosis of LNCaP Prostate Cancer Cells* , 2000, The Journal of Biological Chemistry.

[4]  W. El-Deiry,et al.  Wild-type p53 transactivates the KILLER/DR5 gene through an intronic sequence-specific DNA-binding site , 2000, Oncogene.

[5]  S. Esnault,et al.  Calcium ionophore upregulation of AUUUA-specific binding protein activity is contemporaneous with granulocyte macrophage colony-stimulating factor messenger RNA stabilization in AML14.3D10 cells. , 1999, American journal of respiratory cell and molecular biology.

[6]  S. Korsmeyer,et al.  BCL-2 family members and the mitochondria in apoptosis. , 1999, Genes & development.

[7]  R. Kaufman,et al.  Stress signaling from the lumen of the endoplasmic reticulum: coordination of gene transcriptional and translational controls. , 1999, Genes & development.

[8]  V. Dixit,et al.  Apoptosis control by death and decoy receptors. , 1999, Current opinion in cell biology.

[9]  J. Krebs.  The role of calcium in apoptosis , 1998, Biometals.

[10]  A. Fornace,et al.  Ultraviolet-irradiation-induced apoptosis is mediated via ligand independent activation of tumor necrosis factor receptor 1 , 1998, Oncogene.

[11]  Y. Lazebnik,et al.  Caspases: enemies within. , 1998, Science.

[12]  Junying Yuan,et al.  Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.

[13]  Xiaodong Wang,et al.  Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors , 1998, Cell.

[14]  D. Ferrari,et al.  Apoptosis signaling by death receptors. , 1998, European journal of biochemistry.

[15]  M. Michalak,et al.  Regulation of Calreticulin Gene Expression by Calcium , 1997, The Journal of cell biology.

[16]  P. O'Connor,et al.  Abrogation of p53 function affects gadd gene responses to DNA base-damaging agents and starvation. , 1996, DNA and cell biology.

[17]  G. Salvesen,et al.  Molecular Ordering of Apoptotic Mammalian CED-3/ICE-like Proteases* , 1996, The Journal of Biological Chemistry.

[18]  C. Y. Chen,et al.  AU-rich elements: characterization and importance in mRNA degradation. , 1995, Trends in biochemical sciences.

[19]  Y. Furuya,et al.  The role of calcium, pH, and cell proliferation in the programmed (apoptotic) death of androgen-independent prostatic cancer cells induced by thapsigargin. , 1994, Cancer research.

[20]  A. Fornace,et al.  Genotoxic stress confers preferential and coordinate messenger RNA stability on the five gadd genes. , 1994, Cancer research.

[21]  D. Ron,et al.  CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription. , 1992, Genes & development.

[22]  I. Krantz,et al.  KILLER/DR5 is a DNA damage–inducible p53–regulated death receptor gene , 1997, Nature Genetics.