Global Analysis of Smad2/3-Dependent TGF-β Signaling in Living Mice Reveals Prominent Tissue-Specific Responses to Injury1
暂无分享,去创建一个
Christopher H Contag | Denis Vivien | Tony Wyss-Coray | T. Wyss-Coray | C. Contag | D. Vivien | P. ten Dijke | A. Lin | Peter ten Dijke | Jian Luo | Amy H Lin | Jian Luo | Lauren H Mondshein | L. H. Mondshein
[1] Anita B. Roberts,et al. Expression of transforming growth factor-β2 and β3 mRNAs and proteins in the developing chicken embryo , 1994 .
[2] Denis Vivien,et al. Direct binding of Smad3 and Smad4 to critical TGFβ‐inducible elements in the promoter of human plasminogen activator inhibitor‐type 1 gene , 1998, The EMBO journal.
[3] G. Krystal,et al. LPS-induced upregulation of SHIP is essential for endotoxin tolerance. , 2004, Immunity.
[4] P. Carvey,et al. Combined toxicity of prenatal bacterial endotoxin exposure and postnatal 6-hydroxydopamine in the adult rat midbrain , 2004, Neuroscience.
[5] E. Ruoslahti,et al. Transforming growth factor-beta in disease: the dark side of tissue repair. , 1992, The Journal of clinical investigation.
[6] M. Recce,et al. Gene expression profiling of acute spinal cord injury reveals spreading inflammatory signals and neuron loss. , 2001, Physiological genomics.
[7] D. Morgan,et al. Time-dependent reduction in Aβ levels after intracranial LPS administration in APP transgenic mice , 2004, Experimental Neurology.
[8] Hans Clevers,et al. Signaling pathways in intestinal development and cancer. , 2004, Annual review of cell and developmental biology.
[9] C. Contag,et al. Advances in in vivo bioluminescence imaging of gene expression. , 2002, Annual review of biomedical engineering.
[10] F. Moore. The role of the gastrointestinal tract in postinjury multiple organ failure. , 1999, American journal of surgery.
[11] J. Wrana,et al. Myostatin Signals through a Transforming Growth Factor β-Like Signaling Pathway To Block Adipogenesis , 2003, Molecular and Cellular Biology.
[12] 八木 健,et al. Alternatively spliced variant of smad2 lacking exon 3 : comparison with wild-type smad2 and smad3 , 2001 .
[13] M. Goumans,et al. Distribution of phosphorylated Smad2 identifies target tissues of TGF beta ligands in mouse development. , 2003, Gene expression patterns : GEP.
[14] B. O’Malley,et al. Development of gene-switch transgenic mice that inducibly express transforming growth factor beta1 in the epidermis. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[15] Subhabrata Sanyal,et al. Retrograde Regulation in the CNS Neuron-Specific Interpretations of TGF-β Signaling , 2004, Neuron.
[16] K. Flanders,et al. TRANSFORMING GROWTH FACTOR-βS IN NEURODEGENERATIVE DISEASE , 1998, Progress in Neurobiology.
[17] R. Kucherlapati,et al. Hierarchical model of gene regulation by transforming growth factor β , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[18] L. Mucke,et al. Wild-type but not Alzheimer-mutant amyloid precursor protein confers resistance against p53-mediated apoptosis. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[19] S. Wahl,et al. TGF‐β: the perpetrator of immune suppression by regulatory T cells and suicidal T cells , 2004, Journal of leukocyte biology.
[20] H. Carlsen,et al. In Vivo Imaging of NF-κB Activity1 , 2002, The Journal of Immunology.
[21] A. Brivanlou,et al. The orphan receptor ALK7 and the Activin receptor ALK4 mediate signaling by Nodal proteins during vertebrate development. , 2001, Genes & development.
[22] C. Klein,et al. Growth differentiation factor-9 signaling is mediated by the type I receptor, activin receptor-like kinase 5. , 2004, Molecular endocrinology.
[23] K. Flanders,et al. Characterization of the mouse Smad1 gene and its expression pattern in adult mouse tissues. , 2000, Gene.
[24] J. Massagué,et al. TGFβ Signaling in Growth Control, Cancer, and Heritable Disorders , 2000, Cell.
[25] M. Goumans,et al. Transforming growth factor β signal transduction , 2002, Journal of leukocyte biology.
[26] S. P. Oh,et al. Activin type IIA and IIB receptors mediate Gdf11 signaling in axial vertebral patterning. , 2002, Genes & development.
[27] E. Masliah,et al. Loss of TGF-β1 Leads to Increased Neuronal Cell Death and Microgliosis in Mouse Brain , 2003, Neuron.
[28] Chad H. Koonce,et al. Mice exclusively expressing the short isoform of Smad2 develop normally and are viable and fertile. , 2005, Genes & development.
[29] T. Wyss-Coray,et al. Modelling neuroinflammatory phenotypes in vivo , 2004 .
[30] Edward S. Kim,et al. Immunohistochemical expression of smads 1–6 in the 15‐day gestation mouse embryo: signaling by BMPs and TGF‐βs † , 2001, Developmental dynamics : an official publication of the American Association of Anatomists.
[31] L. Mucke,et al. Increased central nervous system production of extracellular matrix components and development of hydrocephalus in transgenic mice overexpressing transforming growth factor-beta 1. , 1995, The American journal of pathology.
[32] C. Mummery,et al. Spatio-temporal activation of Smad1 and Smad5 in vivo: monitoring transcriptional activity of Smad proteins , 2004, Journal of Cell Science.
[33] T. Macdonald,et al. Immunity, Inflammation, and Allergy in the Gut , 2005, Science.
[34] L. Mucke,et al. TGF-β1 promotes microglial amyloid-β clearance and reduces plaque burden in transgenic mice , 2001, Nature Medicine.
[35] E. Jansen,et al. Selective IκB Kinase Expression in Airway Epithelium Generates Neutrophilic Lung Inflammation1 , 2003, The Journal of Immunology.
[36] M. Goumans,et al. Balancing the activation state of the endothelium via two distinct TGF‐β type I receptors , 2002, The EMBO journal.
[37] J. Massagué,et al. Mechanisms of TGF-β Signaling from Cell Membrane to the Nucleus , 2003, Cell.
[38] K. Miyazono,et al. Characterization of a bone morphogenetic protein-responsive Smad-binding element. , 2000, Molecular biology of the cell.
[39] V. Perry,et al. Transforming growth factor-beta 1-mediated neuroprotection against excitotoxic injury in vivo. , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.