Deviations in Peripheral Blood Cell Populations are Associated with the Stage of Primary Biliary Cholangitis and Presence of Itching

[1]  J. Roliński,et al.  NK and NKT-Like Cells in Patients with Recurrent Furunculosis , 2017, Archivum Immunologiae et Therapiae Experimentalis.

[2]  J. Roliński,et al.  Imbalance in circulatory iNKT, Th17 and T regulatory cell frequencies in patients with B-cell non-Hodgkin's lymphoma. , 2017, Oncology letters.

[3]  J. Roliński,et al.  Subpopulations of natural killer-T-like cells before and after surgical treatment of laryngeal cancer , 2017, Central-European journal of immunology.

[4]  Xiaofeng Yin,et al.  Interleukin-17A-Induced Epithelial-Mesenchymal Transition of Human Intrahepatic Biliary Epithelial Cells: Implications for Primary Biliary Cirrhosis. , 2016, The Tohoku journal of experimental medicine.

[5]  J. Roliński,et al.  Assessment of peripheral blood and bone marrow T, NK, NKT and dendritic cells in patients with multiple myeloma. , 2015, Postepy higieny i medycyny doswiadczalnej.

[6]  Lina Zhang,et al.  The Distribution and the Fibrotic Role of Elevated Inflammatory Th17 Cells in Patients With Primary Biliary Cirrhosis , 2015, Medicine.

[7]  Wei Yang,et al.  Systems biologic analysis of T regulatory cells genetic pathways in murine primary biliary cirrhosis. , 2015, Journal of autoimmunity.

[8]  R. Flavell,et al.  Murine autoimmune cholangitis requires two hits: cytotoxic KLRG1(+) CD8 effector cells and defective T regulatory cells. , 2014, Journal of autoimmunity.

[9]  J. Roliński,et al.  Th17 and Treg cells in adolescents with Graves’ disease. Impact of treatment with methimazole on these cell subsets , 2014, Autoimmunity.

[10]  J. Roliński,et al.  Th17/IL-17A Might Play a Protective Role in Chronic Lymphocytic Leukemia Immunity , 2013, PloS one.

[11]  Patrick Leung,et al.  Identification of Potential Cytokine Pathways for Therapeutic Intervention in Murine Primary Biliary Cirrhosis , 2013, PloS one.

[12]  X. Xiang,et al.  IL4 and IL-17A provide a Th2/Th17-polarized inflammatory milieu in favor of TGF-β1 to induce bronchial epithelial-mesenchymal transition (EMT). , 2013, International journal of clinical and experimental pathology.

[13]  Z. Wang,et al.  BAFF promotes regulatory T-cell apoptosis and blocks cytokine production by activating B cells in primary biliary cirrhosis , 2013, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

[14]  O. Cummings,et al.  IL-17 induces type V collagen overexpression and EMT via TGF-β-dependent pathways in obliterative bronchiolitis. , 2013, American journal of physiology. Lung cellular and molecular physiology.

[15]  P. Kivisäkk,et al.  Increased Th17 response to myelin peptides in pediatric MS. , 2013, Clinical immunology.

[16]  M. Molina,et al.  Effect of interleukin‐6 receptor blockade on the balance between regulatory T cells and T helper type 17 cells in rheumatoid arthritis patients , 2013, Clinical and experimental immunology.

[17]  F. Kandeel,et al.  Foxp3+ Treg Expanded from Patients with Established Diabetes Reduce Helios Expression while Retaining Normal Function Compared to Healthy Individuals , 2013, PloS one.

[18]  S. Yamazaki,et al.  Photo(chemo)therapy Reduces Circulating Th17 Cells and Restores Circulating Regulatory T Cells in Psoriasis , 2013, PloS one.

[19]  P. Wolf,et al.  Involvement of IL-9 in Th17-Associated Inflammation and Angiogenesis of Psoriasis , 2013, PloS one.

[20]  S. Negrini,et al.  Th17 and regulatory T lymphocytes in primary biliary cirrhosis and systemic sclerosis as models of autoimmune fibrotic diseases. , 2012, Autoimmunity reviews.

[21]  Mahavir Singh,et al.  Regulatory T cells that recognize a ubiquitous stress-inducible self-antigen are long-lived suppressors of autoimmune arthritis , 2012, Proceedings of the National Academy of Sciences.

[22]  K. Akashi,et al.  The role of natural killer (NK) and NK T cells in the loss of tolerance in murine primary biliary cirrhosis , 2012, Clinical and experimental immunology.

[23]  D. Darmochwal-Kolarz,et al.  The predominance of Th17 lymphocytes and decreased number and function of Treg cells in preeclampsia. , 2012, Journal of reproductive immunology.

[24]  Guangjie Chen,et al.  IFN-β induces the proliferation of CD4+CD25+Foxp3+ regulatory T cells through upregulation of GITRL on dendritic cells in the treatment of multiple sclerosis , 2012, Journal of Neuroimmunology.

[25]  C. Teuscher,et al.  Activation of p38 MAPK in CD4 T cells controls IL-17 production and autoimmune encephalomyelitis. , 2011, Blood.

[26]  Hong Liu,et al.  Blocking IL-17A Promotes the Resolution of Pulmonary Inflammation and Fibrosis Via TGF-β1–Dependent and –Independent Mechanisms , 2011, The Journal of Immunology.

[27]  B. Ryffel,et al.  IL-1 and IL-23 Mediate Early IL-17A Production in Pulmonary Inflammation Leading to Late Fibrosis , 2011, PloS one.

[28]  P. Berg The role of the innate immune recognition system in the pathogenesis of primary biliary cirrhosis: a conceptual view , 2011, Liver international : official journal of the International Association for the Study of the Liver.

[29]  F. Jadidi-Niaragh,et al.  Th17 Cell, the New Player of Neuroinflammatory Process in Multiple Sclerosis , 2011, Scandinavian journal of immunology.

[30]  R. Coppel,et al.  Primary biliary cirrhosis , 2011, The Lancet.

[31]  J. Roliński,et al.  CD3+/CD16+CD56+ cell numbers in peripheral blood are correlated with higher tumor burden in patients with diffuse large B-cell lymphoma. , 2011, Folia histochemica et cytobiologica.

[32]  K. Tsuneyama,et al.  Innate immunity and primary biliary cirrhosis: Activated invariant natural killer T cells exacerbate murine autoimmune cholangitis and fibrosis , 2011, Hepatology.

[33]  P. Miossec,et al.  Effects of interleukin (IL)-17A and IL-17F in human rheumatoid arthritis synoviocytes , 2011, Annals of the rheumatic diseases.

[34]  G. Murdaca,et al.  The role of Th17 lymphocytes in the autoimmune and chronic inflammatory diseases , 2011, Internal and emergency medicine.

[35]  M. Gershwin,et al.  Phenotypical and functional alterations of CD8 regulatory T cells in primary biliary cirrhosis. , 2010, Journal of autoimmunity.

[36]  T. Wynn,et al.  Bleomycin and IL-1β–mediated pulmonary fibrosis is IL-17A dependent , 2010, The Journal of experimental medicine.

[37]  Wei Liu,et al.  IL-17 induces myocardial fibrosis and enhances RANKL/OPG and MMP/TIMP signaling in isoproterenol-induced heart failure. , 2009, Experimental and molecular pathology.

[38]  L. Cosmi,et al.  Properties and origin of human Th17 cells. , 2009, Molecular immunology.

[39]  C. Benoist,et al.  How punctual ablation of regulatory T cells unleashes an autoimmune lesion within the pancreatic islets. , 2009, Immunity.

[40]  K. Isse,et al.  Periductal interleukin‐17 production in association with biliary innate immunity contributes to the pathogenesis of cholangiopathy in primary biliary cirrhosis , 2009, Clinical and experimental immunology.

[41]  S. Brand Crohn’s disease: Th1, Th17 or both? The change of a paradigm: new immunological and genetic insights implicate Th17 cells in the pathogenesis of Crohn’s disease , 2009, Gut.

[42]  T. Jiang,et al.  Imbalance between T helper type 17 and T regulatory cells in patients with primary biliary cirrhosis: the serum cytokine profile and peripheral cell population , 2009, Clinical and experimental immunology.

[43]  K. Tsuneyama,et al.  Hepatic IL-17 responses in human and murine primary biliary cirrhosis. , 2009, Journal of autoimmunity.

[44]  C. Selmi,et al.  Innate immunity and primary biliary cirrhosis. , 2009, Current molecular medicine.

[45]  I. Mackay,et al.  Adoptive transfer of CD8+ T cells from transforming growth factor beta receptor type II (dominant negative form) induces autoimmune cholangitis in mice , 2008, Hepatology.

[46]  Lewis L Lanier,et al.  Up on the tightrope: natural killer cell activation and inhibition , 2008, Nature Immunology.

[47]  R. Flavell,et al.  Natural killer T cells exacerbate liver injury in a transforming growth factor β receptor II dominant‐negative mouse model of primary biliary cirrhosis , 2007 .

[48]  I. Mackay Autoimmune diseases of the liver, autoimmune hepatitis and primary biliary cirrhosis: Unfinished business , 2007, Hepatology research : the official journal of the Japan Society of Hepatology.

[49]  M. Kaplan,et al.  Quantitation and phenotypic analysis of natural killer T cells in primary biliary cirrhosis using a human CD1d tetramer. , 2002, Gastroenterology.

[50]  M. Kaplan,et al.  Primary biliary cirrhosis , 1998, Hepatology.

[51]  F. BordaCelaya,et al.  [Primary biliary cirrhosis]. , 1989, Anales de medicina interna.

[52]  Xiaofeng Yin,et al.  Interleukin-17 A-Induced Epithelial-Mesenchymal Transition of Human Intrahepatic Biliary Epithelial Cells : Implications for Primary Biliary Cirrhosis , 2016 .

[53]  E. Lubberts,et al.  Th17 cells, but not Th1 cells, from patients with early rheumatoid arthritis are potent inducers of matrix metalloproteinases and proinflammatory cytokines upon synovial fibroblast interaction, including autocrine interleukin-17A production. , 2011, Arthritis and rheumatism.

[54]  G. Ma,et al.  Autoimmune diseases of the liver. Autoimmune hepatitis , 1994 .