Hormonal regulation of ICAM-1 gene expression in thyroid cells, FRTL-5

[1]  D. Singer,et al.  Iodide suppression of major histocompatibility class I gene expression in thyroid cells involves enhancer A and the transcription factor NF-kappa B. , 1998, Molecular endocrinology.

[2]  Keunchil Park,et al.  Production of IL-12 from gene modified human dermal fibroblasts: A preclinical study for IL-12 cancer gene therapy , 1997 .

[3]  In‐San Kim,et al.  Effect of high glucose on synthesis and gene expression of collagen and fibronectin in cultured vascular smooth muscle cells , 1997, Experimental & Molecular Medicine.

[4]  Y. Kohno,et al.  Induction of Graves-like disease in mice by immunization with fibroblasts transfected with the thyrotropin receptor and a class II molecule. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[5]  M. David,et al.  Activation of Protein Kinase A Inhibits Interferon Induction of the Jak/Stat Pathway in U266 Cells (*) , 1996, The Journal of Biological Chemistry.

[6]  J. Darnell,et al.  Maximal activation of transcription by statl and stat3 requires both tyrosine and serine phosphorylation , 1995, Cell.

[7]  L. Ivashkiv.  Cytokines and STATs: how can signals achieve specificity? , 1995, Immunity.

[8]  C. Thompson,et al.  Distinct roles for the costimulatory ligands B7-1 and B7-2 in T helper cell differentiation? , 1995, Cell.

[9]  M. Saji,et al.  Hormonal modulation of major histocompatibility complex class I gene expression involves an enhancer A-binding complex consisting of Fra-2 and the p50 subunit of NF-kappa B , 1995, The Journal of Biological Chemistry.

[10]  D. Singer,et al.  Periocular inflammation in mice with experimental systemic lupus erythematosus. A new experimental blepharitis and its modulation. , 1995, Journal of immunology.

[11]  D. Harlan,et al.  Potential roles of the B7 and CD28 receptor families in autoimmunity and immune evasion. , 1995, Clinical immunology and immunopathology.

[12]  J. Gustafsson,et al.  Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids. , 1995, Molecular endocrinology.

[13]  E. Puré,et al.  Activation of cAMP-dependent pathways in human airway smooth muscle cells inhibits TNF-alpha-induced ICAM-1 and VCAM-1 expression and T lymphocyte adhesion. , 1995, Journal of immunology.

[14]  A. Sharpe.  Analysis of lymphocyte costimulation in vivo using transgenic and 'knockout' mice. , 1995, Current opinion in immunology.

[15]  M. Krönke,et al.  Functional dichotomy of neutral and acidic sphingomyelinases in tumor necrosis factor signaling , 1994, Cell.

[16]  Young Kun Kim,et al.  The Cytokines, Interleukin-1β, Interleukin-6 and Interferon-γ Upregulate the Expression of Intercellular Adhesion Molecule-1 (ICAM-1) in Rat Thyroid Cell Line, FRTL-5 , 1994, The Korean journal of internal medicine.

[17]  Young Kun Kim,et al.  Interleukin-1β and Interleukin-6 Induce Intercellular Adhesion Molecule-1 (ICAM-1) Gene Expression in Rat Thyroid Cell Line, FRTL-5 , 1994 .

[18]  A. Weetman,et al.  Expression of the costimulatory molecule B7/BB1 in autoimmune thyroid disease. , 1994, The Quarterly journal of medicine.

[19]  A. Weetman.  The potential immunological role of the thyroid cell in autoimmune thyroid disease. , 1994, Thyroid : official journal of the American Thyroid Association.

[20]  J. Darnell,et al.  A single phosphotyrosine residue of Stat91 required for gene activation by interferon-gamma. , 1993, Science.

[21]  D. Singer,et al.  Resistance of MHC class I-deficient mice to experimental systemic lupus erythematosus. , 1993, Science.

[22]  M. Krönke,et al.  TNF activates NF-κB by phosphatidylcholine-specific phospholipase C-induced “Acidic” sphingomyelin breakdown , 1992, Cell.

[23]  M. Krönke,et al.  Human 55-kDa receptor for tumor necrosis factor coupled to signal transduction cascades. , 1992, The Journal of biological chemistry.

[24]  D. Singer,et al.  Major histocompatibility complex class I gene expression in rat thyroid cells is regulated by hormones, methimazole, and iodide as well as interferon. , 1992, The Journal of clinical endocrinology and metabolism.

[25]  T. Akamizu,et al.  Characterization of the 5'-flanking region of the rat thyrotropin receptor gene. , 1992, Molecular endocrinology.

[26]  E. Avvedimento,et al.  Regulation of thyrotropin receptor gene expression in rat FRTL-5 thyroid cells. , 1992, Endocrinology.

[27]  H. Loetscher,et al.  Tumor necrosis factor receptors-structure and function , 1992, Immunologic research.

[28]  L. Tartaglia,et al.  Two TNF receptors. , 1992, Immunology today.

[29]  C. Unger,et al.  Tumor necrosis factor induces rapid production of 1'2'diacylglycerol by a phosphatidylcholine-specific phospholipase C , 1991, The Journal of experimental medicine.

[30]  C. Stratowa,et al.  Cloning of the human gene for intercellular adhesion molecule 1 and analysis of its 5'-regulatory region. Induction by cytokines and phorbol ester. , 1991, Journal of immunology.

[31]  M. Miyasaka,et al.  Adhesion molecule expression by the FRTL-5 rat thyroid cell line. , 1991, The Journal of endocrinology.

[32]  W. Fiers.  Tumor necrosis factor Characterization at the molecular, cellular and in vivo level , 1991, FEBS letters.

[33]  Timothy A. Springer,et al.  Adhesion receptors of the immune system , 1990, Nature.

[34]  T. Davies,et al.  Induction of human thyroid cell ICAM-1 (CD54) antigen expression and ICAM-1-mediated lymphocyte binding. , 1990, Endocrinology.

[35]  M. Freeman,et al.  Functional analysis of intercellular adhesion molecule‐1‐expressing human thyroid cells , 1990, European journal of immunology.

[36]  Michael Loran Dustin,et al.  A human intercellular adhesion molecule (ICAM-1) distinct from LFA-1. , 1986, Journal of immunology.

[37]  J. Bonner,et al.  Differentiation , 1968, Nature.