Systolic Left Ventricular Function After Reperfusion Therapy for Acute Myocardial Infarction An Analysis of Determinants of Improvement
暂无分享,去创建一个
R. Califf | E. Topol | S. Ellis | K. Lee | D. Kereiakes | B. George | R. Stack | J. K. Harrison | Kerry L Lee | L. Woodlief | W. O'neill | E. George | Harrison Jk
[1] R. Califf,et al. Coronary bypass surgery improves global and regional left ventricular function following thrombolytic therapy for acute myocardial infarction , 1991 .
[2] W. Rogers,et al. Long-term effect of thrombolytic therapy on left ventricular ejection fraction after acute myocardial infarction. , 1991, The American journal of cardiology.
[3] P. Armstrong,et al. Coronary patency, infarct size and left ventricular function after thrombolytic therapy for acute myocardial infarction: results from the tissue plasminogen activator: Toronto (TPAT) placebo-controlled trial. TPAT Study Group. , 1991, Journal of the American College of Cardiology.
[4] F. Sheehan,et al. Early beneficial effect of streptokinase on left ventricular function in acute myocardial infarction. , 1991, The American journal of cardiology.
[5] J. K. Dunn,et al. Factors determining improvement in left ventricular function after reperfusion therapy for acute myocardial infarction: primacy of baseline ejection fraction. , 1991, Journal of the American College of Cardiology.
[6] I. Belenkie,et al. Relation between flow grade after thrombolytic therapy and the effect of angioplasty on left ventricular function: a prospective randomized trial. , 1991, American heart journal.
[7] R. Gibbons,et al. Mismatch of left ventricular function and infarct size demonstrated by technetium-99m isonitrile imaging after reperfusion therapy for acute myocardial infarction: identification of myocardial stunning and hyperkinesia. , 1990, Journal of the American College of Cardiology.
[8] M. Reznikoff,et al. Bootstrapping: a tool for clinical research. , 1990, Journal of clinical psychology.
[9] E J Topol,et al. Left ventricular ejection fraction may not be useful as an end point of thrombolytic therapy comparative trials. , 1990, Circulation.
[10] F. Harrell,et al. Cardiac rupture, mortality and the timing of thrombolytic therapy: a meta-analysis. , 1990, Journal of the American College of Cardiology.
[11] R. Stack,et al. Results of high dose intravenous urokinase for acute myocardial infarction. , 1990, The American journal of cardiology.
[12] N. Knight. Quantitative radionuclide assessment of regional ventricular function after thrombolytic therapy for acute myocardial infarction; results of phase I thrombolysis in myocardial infarction (TIMI) trial , 1990 .
[13] M Gent,et al. Tissue plasminogen activator: Toronto (TPAT) placebo-controlled randomized trial in acute myocardial infarction. , 1989, Journal of the American College of Cardiology.
[14] E. Braunwald,et al. Quantitative radionuclide assessment of regional ventricular function after thrombolytic therapy for acute myocardial infarction: results of phase I Thrombolysis in Myocardial Infarction (TIMI) trial. , 1989, Journal of the American College of Cardiology.
[15] E. Braunwald,et al. Myocardial reperfusion, limitation of infarct size, reduction of left ventricular dysfunction, and improved survival. Should the paradigm be expanded? , 1989, Circulation.
[16] K. Lee,et al. A randomized controlled trial of intravenous tissue plasminogen activator and early intravenous heparin in acute myocardial infarction. , 1989, Circulation.
[17] R. Kloner,et al. Altered myocardial states. The stunned and hibernating myocardium. , 1989, The American journal of medicine.
[18] F. Van de Werf. Discrepancies between the effects of coronary reperfusion on survival and left ventricular function. , 1989, Lancet.
[19] F. Van de Werf,et al. Intravenous tissue plasminogen activator and size of infarct, left ventricular function, and survival in acute myocardial infarction. , 1988, BMJ.
[20] T. Meinertz,et al. The German multicenter trial of anisoylated plasminogen streptokinase activator complex versus heparin for acute myocardial infarction. , 1988, The American journal of cardiology.
[21] F. Sheehan,et al. Intravenous streptokinase for acute myocardial infarction. Effects on global and regional systolic function. , 1988, Circulation.
[22] Dwight E. Peake,et al. Thrombolysis in myocardial infarction (TIMI) trial: Phase I. A comparison between intravenous tissue plasminogen activator and intravenous streptokinase , 1988 .
[23] R. Califf,et al. Coronary arterial thrombolysis with combined infusion of recombinant tissue-type plasminogen activator and urokinase in patients with acute myocardial infarction. , 1988, Circulation.
[24] Aims Trial Study Group. EFFECT OF INTRAVENOUS APSAC ON MORTALITY AFTER ACUTE MYOCARDIAL INFARCTION: PRELIMINARY REPORT OF A PLACEBO-CONTROLLED CLINICAL TRIAL , 1988, The Lancet.
[25] E. Topol,et al. A randomized trial of intravenous tissue plasminogen activator for acute myocardial infarction with subsequent randomization to elective coronary angioplasty. , 1987, The New England journal of medicine.
[26] H. White,et al. Effect of intravenous streptokinase on left ventricular function and early survival after acute myocardial infarction. , 1987, The New England journal of medicine.
[27] K. Lee,et al. A randomized trial of immediate versus delayed elective angioplasty after intravenous tissue plasminogen activator in acute myocardial infarction. , 1987, The New England journal of medicine.
[28] R Roberts,et al. Thrombolysis in Myocardial Infarction (TIMI) Trial, Phase I: A comparison between intravenous tissue plasminogen activator and intravenous streptokinase. Clinical findings through hospital discharge. , 1987, Circulation.
[29] F H Sheehan,et al. The effect of intravenous thrombolytic therapy on left ventricular function: a report on tissue-type plasminogen activator and streptokinase from the Thrombolysis in Myocardial Infarction (TIMI Phase I) trial. , 1987, Circulation.
[30] A. Nyhuis,et al. Effect of thrombolysis (streptokinase) on left ventricular function during acute myocardial infarction. , 1986, The American journal of cardiology.
[31] H W Woo,et al. Advantages and applications of the centerline method for characterizing regional ventricular function. , 1986, Circulation.
[32] M. Krucoff,et al. Noninvasive detection of coronary artery patency using continuous ST-segment monitoring. , 1986, The American journal of cardiology.
[33] P. Serruys,et al. Preservation of global and regional left ventricular function after early thrombolysis in acute myocardial infarction. , 1986, Journal of the American College of Cardiology.
[34] G. Timmis,et al. The influence of infarction site and size on the ventricular response to coronary thrombolysis. , 1985, Archives of internal medicine.
[35] D. Ferguson,et al. Influence of baseline ejection fraction and success of thrombolysis on mortality and ventricular function after acute myocardial infarction. , 1984, The American journal of cardiology.
[36] W. Rogers,et al. Return of left ventricular function after reperfusion in patients with myocardial infarction: importance of subtotal stenoses or intact collaterals. , 1984, Circulation.
[37] S. Vatner,et al. Salvage of Myocardial Function by Coronary Artery Reperfusion 1, 2, and 3 Hours after Occlusion in Conscious Dogs , 1983, Circulation research.
[38] J. Greenfield,et al. Functional improvement of jeopardized myocardium following intracoronary streptokinase infusion in acute myocardial infarction. , 1983, The Journal of clinical investigation.
[39] E. Braunwald,et al. Time course of functional and biochemical recovery of myocardium salvaged by reperfusion. , 1983, Journal of the American College of Cardiology.
[40] J. Lowe,et al. The Wavefront Phenomenon of Ischemic Cell Death: 1. Myocardial Infarct Size vs Duration of Coronary Occlusion in Dogs , 1977, Circulation.
[41] H Sandler,et al. The use of single plane angiocardiograms for the calculation of left ventricular volume in man. , 1968, American heart journal.