Cyclophilin D gene ablation protects mice from ischemic renal injury.
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B. Padanilam | Kishor Devalaraja-Narashimha | Alicia M Diener | Babu J Padanilam | Kishor Devalaraja-Narashimha | A. M. Diener | Kishor B. Devalaraja-Narashimha
[1] Pierre Croisille,et al. Effect of cyclosporine on reperfusion injury in acute myocardial infarction. , 2008, The New England journal of medicine.
[2] B. Padanilam,et al. PARP-1 inhibits glycolysis in ischemic kidneys. , 2009, Journal of the American Society of Nephrology : JASN.
[3] G. Dorn,et al. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death , 2022 .
[4] T. Uliasz,et al. A microtiter trypan blue absorbance assay for the quantitative determination of excitotoxic neuronal injury in cell culture , 2000, Journal of Neuroscience Methods.
[5] H. Humes. Role of calcium in pathogenesis of acute renal failure. , 1986, The American journal of physiology.
[6] P. Vandenabeele,et al. Inhibition of apoptosis induced by ischemia-reperfusion prevents inflammation. , 1999, The Journal of clinical investigation.
[7] P. Bernardi,et al. Properties of the Permeability Transition Pore in Mitochondria Devoid of Cyclophilin D* , 2005, Journal of Biological Chemistry.
[8] K. Ueda,et al. Mitochondrial impairment induced by poly(ADP‐ribose) polymerase‐1 activation in cortical neurons after oxygen and glucose deprivation , 2005, Journal of neurochemistry.
[9] L. Mandel,et al. Bulk isolation of renal PCT and PST. I. Glucose-dependent metabolic differences. , 1990, The American journal of physiology.
[10] Dean P. Jones,et al. The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore , 2004, Nature.
[11] M. Crompton. Mitochondrial intermembrane junctional complexes and their role in cell death , 2000, The Journal of physiology.
[12] Jean-Claude Martinou,et al. Breaking the mitochondrial barrier , 2001, Nature Reviews Molecular Cell Biology.
[13] P. Paty,et al. Mitochondrial Proliferation and Paradoxical Membrane Depolarization during Terminal Differentiation and Apoptosis in a Human Colon Carcinoma Cell Line , 1997, The Journal of cell biology.
[14] M. Crompton,et al. Cyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosis. , 2004, The Biochemical journal.
[15] W. Craigen,et al. Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death , 2007, Nature Cell Biology.
[16] I. Housini,et al. Alpha-melanocyte-stimulating hormone protects against renal injury after ischemia in mice and rats. , 1997, The Journal of clinical investigation.
[17] N. Rothwell,et al. Cleavage of the Plasma Membrane Na+/Ca2+ Exchanger in Excitotoxicity , 2005, Cell.
[18] Y. Tsujimoto,et al. Role of the mitochondrial membrane permeability transition in cell death , 2007, Apoptosis.
[19] W. Lieberthal,et al. Mouse proximal tubular cell-cell adhesion inhibits apoptosis by a cadherin-dependent mechanism. , 2000, American journal of physiology. Renal physiology.
[20] A. Halestrap,et al. Recent progress in elucidating the molecular mechanism of the mitochondrial permeability transition pore. , 2008, Biochimica et biophysica acta.
[21] S. Korsmeyer,et al. Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[22] B. Padanilam,et al. Poly(ADP-ribose) polymerase-mediated cell injury in acute renal failure. , 2005, Pharmacological research.
[23] A. Halestrap,et al. Direct demonstration of a specific interaction between cyclophilin-D and the adenine nucleotide translocase confirms their role in the mitochondrial permeability transition. , 1998, The Biochemical journal.
[24] M. Yaqoob,et al. Modulation of hypoxia-induced calpain activity in rat renal proximal tubules. , 1996, Kidney international.
[25] M. Crompton,et al. Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress. , 1988, The Biochemical journal.
[26] D. Yellon,et al. Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore. , 2007, Cardiovascular research.
[27] B. Padanilam,et al. Inhibition of poly(ADP-ribose) polymerase attenuates ischemic renal injury in rats. , 2000, American journal of physiology. Regulatory, integrative and comparative physiology.
[28] P. Dagher. Apoptosis in ischemic renal injury: roles of GTP depletion and p53. , 2004, Kidney international.
[29] A. Halestrap,et al. The role of mitochondria in protection of the heart by preconditioning , 2007, Biochimica et biophysica acta.
[30] D. Kintner,et al. Calcium dysregulation induces apoptosis-inducing factor release: Cross-talk between PARP-1- and calpain- signaling pathways , 2009, Experimental Neurology.
[31] M. Duchen,et al. On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury. , 1993, Cardiovascular research.
[32] A. Halestrap,et al. Sanglifehrin A Acts as a Potent Inhibitor of the Mitochondrial Permeability Transition and Reperfusion Injury of the Heart by Binding to Cyclophilin-D at a Different Site from Cyclosporin A* , 2002, The Journal of Biological Chemistry.
[33] W. Buurman,et al. Apoptosis and inflammation in renal reperfusion injury , 2002, Transplantation.
[34] E. Doran,et al. Cytochrome c release from isolated rat liver mitochondria can occur independently of outer-membrane rupture: possible role of contact sites. , 2000, The Biochemical journal.
[35] M. Crompton,et al. Import and processing of heart mitochondrial cyclophilin D. , 1999, European journal of biochemistry.
[36] A. Halestrap,et al. Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition pore. , 2002, The Biochemical journal.
[37] A. Halestrap. Calcium, mitochondria and reperfusion injury: a pore way to die. , 2006, Biochemical Society transactions.
[38] W. B. Reeves,et al. Inhibition of PARP prevents oxidant-induced necrosis but not apoptosis in LLC-PK1 cells. , 1999, The American journal of physiology.
[39] A. Kribben,et al. Assessment of mitochondrial membrane potential in proximal tubules after hypoxia-reoxygenation. , 2005, American journal of physiology. Renal physiology.
[40] G. Steele,et al. Intracellular heterogeneity in mitochondrial membrane potentials revealed by a J-aggregate-forming lipophilic cation JC-1. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[41] P. Bernardi,et al. Genetic Dissection of the Permeability Transition Pore , 2005, Journal of bioenergetics and biomembranes.
[42] Y. S. Kim,et al. Pharmacological preconditioning with low-dose cyclosporine or FK506 reduces subsequent ischemia/reperfusion injury in rat kidney. , 2001, Transplantation.
[43] B. Padanilam. Cell death induced by acute renal injury: a perspective on the contributions of apoptosis and necrosis. , 2003, American journal of physiology. Renal physiology.
[44] B. Padanilam,et al. Poly(ADP-ribose) polymerase-1 gene ablation protects mice from ischemic renal injury. , 2005, American journal of physiology. Renal physiology.
[45] D. Green,et al. Pharmacological manipulation of cell death: clinical applications in sight? , 2005, The Journal of clinical investigation.
[46] B. Padanilam,et al. Activation of Protein Kinase C Isozymes Protects LLCPK1 Cells from H2O2 Induced Necrotic Cell Death , 2003, American Journal of Nephrology.
[47] L. Scorrano,et al. Mitochondria and cell death. Mechanistic aspects and methodological issues. , 1999, European journal of biochemistry.
[48] M. Duchen,et al. Mitochondrial permeability transition pore as a target for cardioprotection in the human heart. , 2005, American journal of physiology. Heart and circulatory physiology.
[49] Jeffrey Robbins,et al. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death , 2005, Nature.
[50] A. Goldman,et al. Role of cyclophilin D in the resistance of brain mitochondria to the permeability transition , 2007, Neurobiology of Aging.
[51] Changlian Zhu,et al. Developmental Shift of Cyclophilin D Contribution to Hypoxic-Ischemic Brain Injury , 2009, The Journal of Neuroscience.
[52] H. I. Chen,et al. The protective effect of niacinamide on ischemia-reperfusion-induced liver injury. , 2001, Journal of biomedical science.
[53] H. Liu,et al. HIF-1alpha activation by a redox-sensitive pathway mediates cyanide-induced BNIP3 upregulation and mitochondrial-dependent cell death. , 2007, Free radical biology & medicine.
[54] M. Periasamy. Calcineurin and the heartbeat, an evolving story. , 2002, Journal of molecular and cellular cardiology.
[55] Luca Scorrano,et al. Mitochondria and cell death. Mechanistic aspects and methodological issues. , 1999, European journal of biochemistry.