Pathophysiology of Acute Kidney Injury Class I histone deacetylase activity is required for proliferation of renal epithelial cells
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S. Zhuang | G. Bayliss | Haidong Yan | T. Zhao | Jinhua Tang | Yanli Yan
[1] Jiansheng Huang,et al. Characterization of the regulation and function of zinc‐dependent histone deacetylases during rodent liver regeneration , 2013, Hepatology.
[2] Na Liu,et al. EGFR activity is required for renal tubular cell dedifferentiation and proliferation in a murine model of folic acid-induced acute kidney injury. , 2013, American journal of physiology. Renal physiology.
[3] S. Zhuang,et al. Blocking the Class I Histone Deacetylase Ameliorates Renal Fibrosis and Inhibits Renal Fibroblast Activation via Modulating TGF-Beta and EGFR Signaling , 2013, PloS one.
[4] Na Liu,et al. Role of epidermal growth factor receptor in acute and chronic kidney injury , 2013, Kidney international.
[5] R. Raghow,et al. Pan-histone deacetylase inhibitors regulate signaling pathways involved in proliferative and pro-inflammatory mechanisms in H9c2 cells , 2012, BMC Genomics.
[6] S. Kassen,et al. Stat3 defines three populations of müller glia and is required for initiating maximal müller glia proliferation in the regenerating zebrafish retina , 2012, The Journal of comparative neurology.
[7] E. Avner,et al. HDAC6 Regulates Epidermal Growth Factor Receptor (EGFR) Endocytic Trafficking and Degradation in Renal Epithelial Cells , 2012, PloS one.
[8] Qiong Wu,et al. Impairment of liver regeneration by the histone deacetylase inhibitor valproic acid in mice , 2012, Journal of Zhejiang University SCIENCE B.
[9] Jian-Kang Chen,et al. Deletion of the epidermal growth factor receptor in renal proximal tubule epithelial cells delays recovery from acute kidney injury , 2012, Kidney international.
[10] D. Bachvarov,et al. Histone Deacetylase (HDAC) Activity Is Critical for Embryonic Kidney Gene Expression, Growth, and Differentiation* , 2011, The Journal of Biological Chemistry.
[11] D. Marsh,et al. Involvement of Insulin-like Growth Factor-binding Protein-3 in the Effects of Histone Deacetylase Inhibitor MS-275 in Hepatoma Cells , 2011, The Journal of Biological Chemistry.
[12] M. Cui,et al. Knockdown of stat3 expression by RNAi inhibits in vitro growth of human ovarian cancer , 2011, Radiology and oncology.
[13] Ming-Shiang Wu,et al. HDAC Inhibition Decreases the Expression of EGFR in Colorectal Cancer Cells , 2011, PloS one.
[14] Steven M. Lipkin,et al. The Class I Hdac Inhibitor Mgcd0103 Induces Cell Cycle Arrest and Apoptosis in Colon Cancer Initiating Cells by Upregulating Dickkopf-1 and Non-Canonical Wnt Signaling , 2010, Oncotarget.
[15] S. Zhuang,et al. A novel STAT3 inhibitor, S3I-201, attenuates renal interstitial fibroblast activation and interstitial fibrosis in obstructive nephropathy. , 2010, Kidney international.
[16] O. Dovey,et al. Histone deacetylase 1 (HDAC1), but not HDAC2, controls embryonic stem cell differentiation , 2010, Proceedings of the National Academy of Sciences.
[17] D. Threadgill,et al. Targeted inactivation of EGF receptor inhibits renal collecting duct development and function. , 2010, Journal of the American Society of Nephrology : JASN.
[18] H. Kohler,et al. Histone deacetylases 1 and 2 act in concert to promote the G1-to-S progression. , 2010, Genes & development.
[19] Julia Tischler,et al. The Cyclin-Dependent Kinase Inhibitor p21 Is a Crucial Target for Histone Deacetylase 1 as a Regulator of Cellular Proliferation , 2009, Molecular and Cellular Biology.
[20] S. Zhuang,et al. Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy. , 2009, American journal of physiology. Renal physiology.
[21] P. Munster,et al. HDAC2 regulates chromatin plasticity and enhances DNA vulnerability , 2009, Molecular Cancer Therapeutics.
[22] S. Zhuang,et al. Transglutaminase-1 Regulates Renal Epithelial Cell Proliferation through Activation of Stat-3* , 2009, Journal of Biological Chemistry.
[23] T. Tiganis,et al. Replication checkpoint control by a PTK/STAT3/Cyclin D1 axis , 2009, Cell cycle.
[24] Barbara Hero,et al. Histone Deacetylase 8 in Neuroblastoma Tumorigenesis , 2009, Clinical Cancer Research.
[25] B. Liu,et al. Activation of Signal Transducers and Activators of Transcription 3 and Overexpression of its Target Gene CyclinD1 in Laryngeal Carcinomas , 2008, The Laryngoscope.
[26] T. Nielsen,et al. Histone deacetylase inhibitors induce growth arrest, apoptosis, and differentiation in clear cell sarcoma models , 2008, Molecular Cancer Therapeutics.
[27] D. Cortez,et al. Deletion of histone deacetylase 3 reveals critical roles in S phase progression and DNA damage control. , 2008, Molecular cell.
[28] A. McMahon,et al. Intrinsic epithelial cells repair the kidney after injury. , 2008, Cell stem cell.
[29] E. Biçaku,et al. Selective inhibition of histone deacetylase 2 silences progesterone receptor-mediated signaling. , 2008, Cancer research.
[30] P. Finn,et al. Determination of the class and isoform selectivity of small-molecule histone deacetylase inhibitors. , 2008, The Biochemical journal.
[31] G. Draetta,et al. Role for Histone Deacetylase 1 in Human Tumor Cell Proliferation , 2007, Molecular and Cellular Biology.
[32] John A Kellum,et al. Acute Kidney Injury Network: report of an initiative to improve outcomes in acute kidney injury , 2007, Critical care.
[33] Sang Gyun Kim,et al. Antitumor activity of SK-7041, a novel histone deacetylase inhibitor, in human lung and breast cancer cells. , 2006, Anticancer research.
[34] A. Rustgi,et al. Cyclin D1 Induction through IκB Kinase β/Nuclear Factor-κB Pathway Is Responsible for Arsenite-Induced Increased Cell Cycle G1-S Phase Transition in Human Keratinocytes , 2005 .
[35] H. Lane,et al. ERBB receptors and cancer: the complexity of targeted inhibitors , 2005, Nature Reviews Cancer.
[36] M. Whitaker,et al. GFP—PCNA as an S‐phase marker in embryos during the first and subsequent cell cycles , 2005, Biology of the cell.
[37] R. Schnellmann,et al. Requirement of the epidermal growth factor receptor in renal epithelial cell proliferation and migration. , 2004, American journal of physiology. Renal physiology.
[38] Sang Gyun Kim,et al. Class I Histone Deacetylase-Selective Novel Synthetic Inhibitors Potently Inhibit Human Tumor Proliferation , 2004, Clinical Cancer Research.
[39] Jian-Kang Chen,et al. Importance of functional EGF receptors in recovery from acute nephrotoxic injury. , 2003, Journal of the American Society of Nephrology : JASN.
[40] Ricardo Macarron,et al. Identification of Novel Isoform-Selective Inhibitors within Class I Histone Deacetylases , 2003, Journal of Pharmacology and Experimental Therapeutics.
[41] J. Bonventre. Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure. , 2003, Journal of the American Society of Nephrology : JASN.
[42] Joseph Schlessinger,et al. Ligand-Induced, Receptor-Mediated Dimerization and Activation of EGF Receptor , 2002, Cell.
[43] Wei Li,et al. STAT3 Contributes to the Mitogenic Response of Hepatocytes during Liver Regeneration* , 2002, The Journal of Biological Chemistry.
[44] M. Masuda,et al. Constitutive activation of signal transducers and activators of transcription 3 correlates with cyclin D1 overexpression and may provide a novel prognostic marker in head and neck squamous cell carcinoma. , 2002, Cancer research.
[45] D. O’Carroll,et al. Essential function of histone deacetylase 1 in proliferation control and CDK inhibitor repression , 2002, The EMBO journal.
[46] Y. Yarden,et al. Untangling the ErbB signalling network , 2001, Nature Reviews Molecular Cell Biology.
[47] J. Turkson,et al. Induction of p21WAF1/CIP1 and cyclin D1 expression by the Src oncoprotein in mouse fibroblasts: role of activated STAT3 signaling , 2000, Oncogene.
[48] E. Bello‐Reuss,et al. Expression and function of P-glycoprotein in a mouse kidney cell line. , 1995, The American journal of physiology.
[49] K. Herrup,et al. Targeted disruption of mouse EGF receptor: effect of genetic background on mutant phenotype. , 1995, Science.
[50] J. Kellum,et al. Pathophysiology of acute kidney injury: a new perspective. , 2010, Contributions to nephrology.
[51] 荒川 哲次. Activation of signal transducer and activator of transcription 3 correlates with cell proliferation and renal injury in human glomerulonephritis , 2008 .
[52] W. Gerald,et al. Activator of Transcription 3 Transformation by Activated Signal Transducer and Cyclin D 1 Is Transcriptionally Regulated by and Required for Updated , 2006 .