Effects of CYP2C19 genetic polymorphism on the pharmacokinetics and pharmacodynamics of omeprazole in Chinese people
暂无分享,去创建一个
Qiao Mei | Jianming Xu | Jianming Xu | Xiang‐peng Hu | Yong-mei Hu | Xin-hua Xu | Xiang‐peng Hu | Q. Mei | Xin-hua Xu | Yong-mei Hu
[1] P. Lundborg,et al. Effect of omeprazole treatment on diazepam plasma levels in slow versus normal rapid metabolizers of omeprazole , 1990, Clinical pharmacology and therapeutics.
[2] M O Karlsson,et al. A turnover model of irreversible inhibition of gastric acid secretion by omeprazole in the dog. , 2000, The Journal of pharmacology and experimental therapeutics.
[3] S. Higuchi,et al. Pharmacokinetics of omeprazole (a substrate of CYP2C19) and comparison with two mutant alleles, C gamma P2C19m1 in exon 5 and C gamma P2C19m2 in exon 4, in Japanese subjects. , 1996, Clinical pharmacology and therapeutics.
[4] M. Onda,et al. Genetic differences in CYP2C19 single nucleotide polymorphisms among four Asian populations , 2001, Journal of Gastroenterology.
[5] K. Safranow,et al. Effect of CYP2C19 and MDR1 polymorphisms on cure rate in patients with acid-related disorders with Helicobacter pylori infection , 2005, European Journal of Clinical Pharmacology.
[6] Na Gao,et al. Pharmacokinetics of three proton pump inhibitors in Chinese subjects in relation to the CYP2C19 genotype , 2006, European Journal of Clinical Pharmacology.
[7] H. Zhou,et al. Individual and ethnic differences in CYP2C19 activity in Chinese populations. , 2000, Acta pharmacologica Sinica.
[8] G. Sachs,et al. Pharmacological aspects of acid secretion , 1995, Digestive Diseases and Sciences.
[9] L. Bertilsson,et al. Omeprazole treatment of Korean patients: effects on gastric pH and gastrin release in relation to CYP2C19 geno- and phenotypes. , 2004, Basic & clinical pharmacology & toxicology.
[10] S. Higuchi,et al. Pharmacokinetics of omeprazole (a substrate of CYP2C19) and comparison with two mutant alleles, CYP2C19m1 in exon 5 and CYP2C19m2 in exon 4, in Japanese subjects , 1996 .
[11] G R Wilkinson,et al. Identification of a new genetic defect responsible for the polymorphism of (S)-mephenytoin metabolism in Japanese. , 1994, Molecular pharmacology.
[12] K. Ohashi,et al. Effects of CYP2C19 genotypic differences in the metabolism of omeprazole and rabeprazole on intragastric pH , 2001, Alimentary pharmacology & therapeutics.
[13] T Ishizaki,et al. CYP2C19 genotype status and effect of omeprazole on intragastric pH in humans. , 1998, Clinical pharmacology and therapeutics.
[14] M. Kasuga,et al. CYP2C19 Genotype and Pharmacokinetics of Three Proton Pump Inhibitors in Healthy Subjects , 2001, Pharmaceutical Research.
[15] Leif Bertilsson,et al. A common novel CYP2C19 gene variant causes ultrarapid drug metabolism relevant for the drug response to proton pump inhibitors and antidepressants , 2006, Clinical pharmacology and therapeutics.
[16] A. Chow,et al. Omeprazole as a CYP2C19 Marker in Chinese Subjects: Assessment of Its Gene‐Dose Effect and Intrasubject Variability , 2004, Journal of clinical pharmacology.
[17] G R Wilkinson,et al. The major genetic defect responsible for the polymorphism of S-mephenytoin metabolism in humans. , 1994, The Journal of biological chemistry.
[18] H. Mieno,et al. Effect of omeprazole 10 mg on intragastric pH in three different CYP2C19 genotypes, compared with omeprazole 20 mg and lafutidine 20 mg, a new H2‐receptor antagonist , 2003, Alimentary pharmacology & therapeutics.
[19] J. Miners,et al. Identification of human liver cytochrome P450 isoforms mediating secondary omeprazole metabolism. , 1994, British journal of clinical pharmacology.
[20] L. Bertilsson,et al. Pronounced differences between native Chinese and Swedish populations in the polymorphic hydroxylations of debrisoquin and S‐mephenytoin , 1992, Clinical pharmacology and therapeutics.
[21] T. Starzynska,et al. Effect of CYP2C19*17 gene variant on Helicobacter pylori eradication in peptic ulcer patients , 2006, European Journal of Clinical Pharmacology.
[22] T. Andersson,et al. Pharmacokinetics and bioavailability of omeprazole after single and repeated oral administration in healthy subjects. , 1990, British journal of clinical pharmacology.
[23] U. Haglund,et al. Effect of omeprazole--a gastric proton pump inhibitor--on pentagastrin stimulated acid secretion in man. , 1983, Gut.
[24] L. Bertilsson,et al. Effects of omeprazole on intragastric pH and plasma gastrin are dependent on the CYP2C19 polymorphism. , 2000, Gastroenterology.
[25] M. Fromm,et al. Interaction of omeprazole, lansoprazole and pantoprazole with P-glycoprotein , 2001, Naunyn-Schmiedeberg's Archives of Pharmacology.
[26] G. Sachs. Physiology of the Parietal Cell and Therapeutic Implications , 2003, Pharmacotherapy.
[27] L. Bertilsson,et al. Interphenotype differences in disposition and effect on gastrin levels of omeprazole--suitability of omeprazole as a probe for CYP2C19. , 1995, British journal of clinical pharmacology.
[28] O. Junghard,et al. The effect of the area under the plasma concentration vs time curve and the maximum plasma concentration of esomeprazole on intragastric pH , 2002, European Journal of Clinical Pharmacology.
[29] J. Goldstein,et al. Differences in the incidence of the CYP2C19 polymorphism affecting the S-mephenytoin phenotype in Chinese Han and Bai populations and identification of a new rare CYP2C19 mutant allele. , 1997, The Journal of pharmacology and experimental therapeutics.
[30] L. Bertilsson,et al. Slow omeprazole metabolizers are also poor S-mephenytoin hydroxylators. , 1990, Therapeutic drug monitoring.
[31] Yuan Zhang,et al. Polymorphic hydroxylation of S-mephenytoin and omeprazole metabolism in Caucasian and Chinese subjects. , 1992, Pharmacogenetics.
[32] D. Flockhart,et al. Clinical Significance of the Cytochrome P450 2C19 Genetic Polymorphism , 2002, Clinical pharmacokinetics.