The immunology of psoriasis and biologic immunotherapy.
暂无分享,去创建一个
[1] D. West,et al. Biologic therapy for psoriasis: the new therapeutic frontier. , 2002, Archives of dermatology.
[2] P. Bongrand,et al. Intercellular transfer of antigen‐presenting cell determinants onto T cells: molecular mechanisms and biological significance , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[3] M. Aringer. T lymphocyte activation--an inside overview. , 2002, Acta medica Austriaca.
[4] G. Krueger,et al. Treatment of chronic plaque psoriasis by selective targeting of memory effector T lymphocytes. , 2001, The New England journal of medicine.
[5] P. Friedl,et al. Interaction of T cells with APCs: the serial encounter model. , 2001, Trends in immunology.
[6] James G Krueger,et al. Potential Role of the Chemokine Receptors CXCR3, CCR4, and the Integrin αEβ7 in the Pathogenesis of Psoriasis Vulgaris , 2001, Laboratory Investigation.
[7] B. Nickoloff,et al. Keratinocyte CDw60 expression is modulated by both a Th-1 type cytokine IFN-gamma and Th-2 cytokines IL-4 and IL-13: relevance to psoriasis. , 2001, The Journal of investigative dermatology.
[8] B. Nickoloff,et al. Characterization of lymphocyte-dependent angiogenesis using a SCID mouse: human skin model of psoriasis. , 2000, The journal of investigative dermatology. Symposium proceedings.
[9] C. Caux,et al. Up-Regulation of Macrophage Inflammatory Protein-3α/CCL20 and CC Chemokine Receptor 6 in Psoriasis1 , 2000, The Journal of Immunology.
[10] M. Lebwohl,et al. Effects of administration of a single dose of a humanized monoclonal antibody to CD11a on the immunobiology and clinical activity of psoriasis. , 2000, Journal of the American Academy of Dermatology.
[11] T. Kupper,et al. Inflammatory skin diseases, T cells, and immune surveillance. , 1999, The New England journal of medicine.
[12] J. Koo. Current Consensus and Update on Psoriasis Therapy: A Perspective from the U.S. , 1999, The Journal of dermatology.
[13] M. Müller,et al. Combined analysis of polymorphisms of the tumor necrosis factor-alpha and interleukin-10 promoter regions and polymorphic xenobiotic metabolizing enzymes in psoriasis. , 1999, The Journal of investigative dermatology.
[14] Franco Ameglio,et al. Cytokines in psoriasis , 1999, International journal of dermatology.
[15] G. Tydén,et al. Zenapax (daclizumab) reduces the incidence of acute rejection episodes and improves patient survival following renal transplantation. No 14874 and No 14393 Zenapax Study Groups. , 1999, Transplantation proceedings.
[16] N. Yawalkar,et al. Expression of interleukin-12 is increased in psoriatic skin. , 1998, The Journal of investigative dermatology.
[17] H. Moon,et al. Psoriasiform eruption triggered by recombinant granulocyte-macrophage colony stimulating factor (rGM-CSF) and exacerbated by granulocyte colony stimulating factor (rG-CSF) in a patient with breast cancer. , 1998, Journal of Korean medical science.
[18] Y. Ueyama,et al. Histological and immunocytochemical studies of human psoriatic lesions transplanted onto SCID mice. , 1998, Journal of dermatological science.
[19] M. Holick,et al. Administration of DAB389IL-2 to patients with recalcitrant psoriasis: a double-blind, phase II multicenter trial. , 1998, Journal of the American Academy of Dermatology.
[20] K. Nishioka,et al. Repeated subcutaneous injection of staphylococcal enterotoxin B-stimulated lymphocytes retains epidermal thickness of psoriatic skin-graft onto severe combined immunodeficient mice. , 1998, Journal of dermatological science.
[21] W Sterry,et al. IL-10 is a key cytokine in psoriasis. Proof of principle by IL-10 therapy: a new therapeutic approach. , 1998, The Journal of clinical investigation.
[22] Sato,et al. RANTES expression in psoriatic skin, and regulation of RANTES and IL‐8 production in cultured epidermal keratinocytes by active vitamin D3 (tacalcitol) , 1998, The British journal of dermatology.
[23] J. Kieffer,et al. Cutaneous lymphocyte antigen is a specialized form of PSGL-1 expressed on skin-homing T cells , 1997, Nature.
[24] Y. Ullmann,et al. T-lymphocyte dependence of psoriatic pathology in human psoriatic skin grafted to SCID mice. , 1997, The Journal of investigative dermatology.
[25] L. Adorini,et al. Immune deviation towards Th2 inhibits Th-1-mediated autoimmune diabetes. , 1997, Biochemical Society transactions.
[26] E. Bröcker,et al. Differential Expression of GRO-α and IL-8 mRNA Psoriasis: A Model for Neutrophil Migration and Accumulation In Vivo , 1996 .
[27] B. Nickoloff,et al. Dermal injection of immunocytes induces psoriasis. , 1996, The Journal of clinical investigation.
[28] T. Zollner,et al. Pulling the trigger on psoriasis , 1996, Nature.
[29] F. Finkelman. Relationships among antigen presentation, cytokines, immune deviation, and autoimmune disease , 1995, The Journal of experimental medicine.
[30] James G. Krueger,et al. Response of psoriasis to a lymphocyte-selective toxin (DAB389IL-2) suggests a primary immune, but not keratinocyte, pathogenic basis , 1995, Nature Genetics.
[31] M. Burdick,et al. Severe combined immunodeficiency mouse and human psoriatic skin chimeras. Validation of a new animal model. , 1995, The American journal of pathology.
[32] D. Wallach,et al. Elevated tumour necrosis factor‐alpha (TNF‐α) biological activity in psoriatic skin lesions , 1994, Clinical and experimental immunology.
[33] W. Jochum,et al. T cells involved in psoriasis vulgaris belong to the Th1 subset. , 1994, The Journal of investigative dermatology.
[34] J. Trowsdale,et al. COMPLEXITY IN THE MAJOR HISTOCOMPATIBILITY COMPLEX , 1992, European journal of immunogenetics : official journal of the British Society for Histocompatibility and Immunogenetics.
[35] T. Strom,et al. DAB486IL‐2 (IL‐2 Toxin) Selectively Inactivates High‐Affinity IL‐2 Receptor‐Bearing Human Peripheral Blood Mononuclear Cells , 1991, Annals of the New York Academy of Sciences.
[36] L. Picker,et al. ELAM-1 is an adhesion molecule for skin-homing T cells , 1991, Nature.
[37] J. Billings,et al. Cyclosporine improves psoriasis in a double-blind study. , 1986, JAMA.
[38] J. Bos,et al. Immunocompetent cells in psoriasis , 1983, Archives of Dermatological Research.
[39] W. Mueller,et al. Cyclosporin A for psoriasis. , 1979, The New England journal of medicine.
[40] R. Matre,et al. Characterization of mononuclear cell infiltrates in psoriatic lesions. , 1978, The Journal of investigative dermatology.
[41] C. Pert,et al. Upregulation of RANTES in psoriatic keratinocytes: a possible pathogenic mechanism for psoriasis. , 1999, Acta dermato-venereologica.
[42] M. Sekimata,et al. Different growth properties in response to epidermal growth factor and interleukin-6 of primary keratinocytes derived from normal and psoriatic lesional skin. , 1998, Journal of dermatological science.
[43] M. Berridge. Lymphocyte activation in health and disease. , 1997, Critical reviews in immunology.
[44] E. Parra,et al. T cell activation pathways: B7, LFA-3, and ICAM-1 shape unique T cell profiles. , 1995, Critical reviews in immunology.
[45] B. Julian,et al. OKT3 first-dose reaction: association with T cell subsets and cytokine release. , 1991, Kidney international.