Enhanced activity of sodium-lithium countertransport in patients with cardiac syndrome X: a potential link between cardiac and metabolic syndrome X.

[1]  C. Trani,et al.  Abnormal cardiac adrenergic nerve function in patients with syndrome X detected by [123I]metaiodobenzylguanidine myocardial scintigraphy. , 1997, Circulation.

[2]  E. Peleg,et al.  Enhanced red cell sodium-hydrogen exchange in microvascular angina. , 1997, European heart journal.

[3]  T. Hardman,et al.  Controversies surrounding erythrocyte sodium-lithium countertransport , 1996, Journal of hypertension.

[4]  Agostoni Pg The lymphatics and lung fluid resorption , 1996 .

[5]  H. Crijns,et al.  Mechanisms of angina pectoris in syndrome X assessed by myocardial perfusion dynamics and heart rate variability. , 1995, European heart journal.

[6]  M. Canessa Erythrocyte sodium-lithium countertransport: another link between essential hypertension and diabetes. , 1994, Current opinion in nephrology and hypertension.

[7]  A. Maseri,et al.  Left ventricular hypercontractility and ST segment depression in patients with syndrome X. , 1993, Journal of the American College of Cardiology.

[8]  H. Bøtker,et al.  Insulin resistance in microvascular angina (syndrome X) , 1993, The Lancet.

[9]  A. Lammertsma,et al.  Comparison of regional myocardial blood flow in syndrome X and one-vessel coronary artery disease. , 1993, The American journal of cardiology.

[10]  A. Takeshita,et al.  Evidence of impaired endothelium-dependent coronary vasodilatation in patients with angina pectoris and normal coronary angiograms. , 1993, The New England journal of medicine.

[11]  D. Rosskopf,et al.  Membrane sodium-proton exchange and primary hypertension. , 1993, Hypertension.

[12]  A. Aviv The roles of cell Ca2+, protein kinase C and the Na(+)-H+ antiport in the development of hypertension and insulin resistance. , 1992, Journal of the American Society of Nephrology : JASN.

[13]  N. D. De Cesare,et al.  Coronary adrenergic hyperreactivity in patients with syndrome X and abnormal electrocardiogram at rest. , 1991, The American journal of cardiology.

[14]  E A Anderson,et al.  Hyperinsulinemia produces both sympathetic neural activation and vasodilation in normal humans. , 1991, The Journal of clinical investigation.

[15]  C. Jones,et al.  Hyperinsulinaemia and microvascular angina ("syndrome X") , 1991, The Lancet.

[16]  F. Crea,et al.  Mechanisms of angina pectoris in syndrome X. , 1991, Journal of the American College of Cardiology.

[17]  A. Lahey Airway Hyperresponsiveness in Patients With Microvascular Angina. , 1991 .

[18]  M. Kaliner,et al.  Airway hyperresponsiveness in patients with microvascular angina. Evidence for a diffuse disorder of smooth muscle responsiveness. , 1990, Circulation.

[19]  R. Cannon,et al.  Coronary flow reserve, esophageal motility, and chest pain in patients with angiographically normal coronary arteries. , 1990, The American journal of medicine.

[20]  L. Poston,et al.  Endothelin-induced contraction of human peripheral resistance vessels is partly dependent on stimulation of sodium-hydrogen exchange. , 1989, Journal of hypertension.

[21]  E. Cragoe,et al.  Sodium-Lithium Exchange and Sodium-Proton Exchange Are Mediated by the Same Transport System in Sarcolemmal Vesicles From Bovine Superior Mesenteric Artery , 1989, Circulation research.

[22]  L. Ng,et al.  Leucocyte intracellular pH and Na+/H+ antiport activity in human hypertension. , 1989, Journal of hypertension.

[23]  R. Cannon,et al.  "Microvascular angina" as a cause of chest pain with angiographically normal coronary arteries. , 1988, The American journal of cardiology.

[24]  R. Cannon,et al.  Impaired forearm vasodilator reserve in patients with microvascular angina. Evidence of a generalized disorder of vascular function? , 1987, The New England journal of medicine.

[25]  R. Cannon,et al.  Angina caused by reduced vasodilator reserve of the small coronary arteries. , 1983, Journal of the American College of Cardiology.

[26]  D. Tosteson,et al.  Increased sodium-lithium countertransport in red cells of patients with essential hypertension. , 1980, The New England journal of medicine.