Development of an inhaled endotoxin challenge protocol for characterizing evoked cell surface phenotype and genomic responses of airway cells in allergic individuals.
暂无分享,去创建一个
J. Ting | N. Alexis | D. Peden | J. C. Lay | W. J. Brickey | Ying Wang | R. Roubey | W. Brickey
[1] J. Cadranel,et al. Safety and tolerability of three consecutive bronchoscopies after allergen challenge in volunteers with mild asthma , 2007, Thorax.
[2] J. Kongerud,et al. Macrophage enrichment from induced sputum , 2007, Thorax.
[3] D. Zeldin,et al. Endotoxin exposure is a risk factor for asthma: the national survey of endotoxin in United States housing. , 2005, American journal of respiratory and critical care medicine.
[4] Donald MacGlashan. IgE and FcɛRI regulation , 2005 .
[5] Donald MacGlashan. IgE and FcεRI Regulation , 2005 .
[6] D. Patel,et al. Acute LPS inhalation in healthy volunteers induces dendritic cell maturation in vivo. , 2005, The Journal of allergy and clinical immunology.
[7] N. Alexis,et al. Inhalation of low-dose endotoxin favors local T(H)2 response and primes airway phagocytes in vivo. , 2004, The Journal of allergy and clinical immunology.
[8] M. Hazucha,et al. Low-dose airborne endotoxin exposure enhances bronchial responsiveness to inhaled allergen in atopic asthmatics. , 2003, The Journal of allergy and clinical immunology.
[9] E. D. de KLERK,et al. Interleukin‐1β and interleukin‐1ra levels in nasal lavages during experimental rhinovirus infection in asthmatic and non‐asthmatic subjects , 2003, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[10] Kim Bottomly,et al. Lipopolysaccharide-enhanced, Toll-like Receptor 4–dependent T Helper Cell Type 2 Responses to Inhaled Antigen , 2002, The Journal of experimental medicine.
[11] V. Ridger,et al. Caspase-1-Deficient Mice Have Delayed Neutrophil Apoptosis and a Prolonged Inflammatory Response to Lipopolysaccharide-Induced Acute Lung Injury1 , 2002, The Journal of Immunology.
[12] N. Alexis,et al. Induced sputum derives from the central airways: confirmation using a radiolabeled aerosol bolus delivery technique. , 2001, American journal of respiratory and critical care medicine.
[13] C. Adra,et al. Imbalance production between interleukin-1beta (IL-1beta) and IL-1 receptor antagonist (IL-1Ra) in bronchial asthma. , 2000, Biochemical and biophysical research communications.
[14] D. Peden,et al. Allergen provocation augments endotoxin-induced nasal inflammation in subjects with atopic asthma. , 2000, The Journal of allergy and clinical immunology.
[15] R. Sergysels,et al. Dose-response relationship to inhaled endotoxin in normal subjects. , 1997, American journal of respiratory and critical care medicine.
[16] J. Kinet,et al. Peripheral blood dendritic cells express Fc epsilon RI as a complex composed of Fc epsilon RI alpha- and Fc epsilon RI gamma-chains and can use this receptor for IgE-mediated allergen presentation. , 1996, Journal of immunology.
[17] J. Denburg,et al. Use of induced sputum cell counts to investigate airway inflammation in asthma. , 1992, Thorax.
[18] M. Dentener,et al. Evaluation of oral corticosteroids and phosphodiesterase-4 inhibitor on the acute inflammation induced by inhaled lipopolysaccharide in human. , 2007, Pulmonary pharmacology & therapeutics.
[19] Andrew H Liu,et al. Something old, something new: indoor endotoxin, allergens and asthma. , 2004, Paediatric respiratory reviews.