Development of an inhaled endotoxin challenge protocol for characterizing evoked cell surface phenotype and genomic responses of airway cells in allergic individuals.

[1]  J. Cadranel,et al.  Safety and tolerability of three consecutive bronchoscopies after allergen challenge in volunteers with mild asthma , 2007, Thorax.

[2]  J. Kongerud,et al.  Macrophage enrichment from induced sputum , 2007, Thorax.

[3]  D. Zeldin,et al.  Endotoxin exposure is a risk factor for asthma: the national survey of endotoxin in United States housing. , 2005, American journal of respiratory and critical care medicine.

[4]  Donald MacGlashan IgE and FcɛRI regulation , 2005 .

[5]  Donald MacGlashan IgE and FcεRI Regulation , 2005 .

[6]  D. Patel,et al.  Acute LPS inhalation in healthy volunteers induces dendritic cell maturation in vivo. , 2005, The Journal of allergy and clinical immunology.

[7]  N. Alexis,et al.  Inhalation of low-dose endotoxin favors local T(H)2 response and primes airway phagocytes in vivo. , 2004, The Journal of allergy and clinical immunology.

[8]  M. Hazucha,et al.  Low-dose airborne endotoxin exposure enhances bronchial responsiveness to inhaled allergen in atopic asthmatics. , 2003, The Journal of allergy and clinical immunology.

[9]  E. D. de KLERK,et al.  Interleukin‐1β and interleukin‐1ra levels in nasal lavages during experimental rhinovirus infection in asthmatic and non‐asthmatic subjects , 2003, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[10]  Kim Bottomly,et al.  Lipopolysaccharide-enhanced, Toll-like Receptor 4–dependent T Helper Cell Type 2 Responses to Inhaled Antigen , 2002, The Journal of experimental medicine.

[11]  V. Ridger,et al.  Caspase-1-Deficient Mice Have Delayed Neutrophil Apoptosis and a Prolonged Inflammatory Response to Lipopolysaccharide-Induced Acute Lung Injury1 , 2002, The Journal of Immunology.

[12]  N. Alexis,et al.  Induced sputum derives from the central airways: confirmation using a radiolabeled aerosol bolus delivery technique. , 2001, American journal of respiratory and critical care medicine.

[13]  C. Adra,et al.  Imbalance production between interleukin-1beta (IL-1beta) and IL-1 receptor antagonist (IL-1Ra) in bronchial asthma. , 2000, Biochemical and biophysical research communications.

[14]  D. Peden,et al.  Allergen provocation augments endotoxin-induced nasal inflammation in subjects with atopic asthma. , 2000, The Journal of allergy and clinical immunology.

[15]  R. Sergysels,et al.  Dose-response relationship to inhaled endotoxin in normal subjects. , 1997, American journal of respiratory and critical care medicine.

[16]  J. Kinet,et al.  Peripheral blood dendritic cells express Fc epsilon RI as a complex composed of Fc epsilon RI alpha- and Fc epsilon RI gamma-chains and can use this receptor for IgE-mediated allergen presentation. , 1996, Journal of immunology.

[17]  J. Denburg,et al.  Use of induced sputum cell counts to investigate airway inflammation in asthma. , 1992, Thorax.

[18]  M. Dentener,et al.  Evaluation of oral corticosteroids and phosphodiesterase-4 inhibitor on the acute inflammation induced by inhaled lipopolysaccharide in human. , 2007, Pulmonary pharmacology & therapeutics.

[19]  Andrew H Liu,et al.  Something old, something new: indoor endotoxin, allergens and asthma. , 2004, Paediatric respiratory reviews.