GABA induces Ca2+ transients in astrocytes
暂无分享,去创建一个
P. Eriksson | E. Hansson | L. Rönnbäck | M. Nilsson | M. Nilsson | P. S. Eriksson | L. Rönnbäck | E. Hansson
[1] N. Kasai,et al. The monoclonal antibody A2B5 is specific to ganglioside GQ1c , 1983, Brain Research.
[2] D. Puro,et al. Stretch‐activated channels in human retinal muller cells , 1991, Glia.
[3] R Y Tsien,et al. Sequential activation and lethal hit measured by [Ca2+]i in individual cytolytic T cells and targets. , 1987, The EMBO journal.
[4] M. Raiteri,et al. Release‐regulating autoreceptors of the GABAB‐type in human cerebral cortex , 1989, British journal of pharmacology.
[5] J. Bormann,et al. Patch-clamp study of gamma-aminobutyric acid receptor Cl- channels in cultured astrocytes. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[6] R. Tsien,et al. A new generation of Ca2+ indicators with greatly improved fluorescence properties. , 1985, The Journal of biological chemistry.
[7] D. Corey,et al. Ion channel expression by white matter glia: The type-1 astrocyte , 1990, Neuron.
[8] S. Murphy,et al. Evidence for an interaction between GABAB and glutamate receptors in astrocytes as revealed by changes in Ca2+ flux. , 1986, European journal of pharmacology.
[9] N. Bowery,et al. Characteristics of GABAB receptor binding sites on rat whole brain synaptic membranes , 1997 .
[10] L. Sivilotti,et al. GABA receptor mechanisms in the central nervous system , 1991, Progress in Neurobiology.
[11] M. Raiteri,et al. Regional Selectivity of a γ‐Aminobutyric Acid‐Induced [3H]Acetylcholine Release Sensitive to Inhibitors of γ‐Aminobutyric Acid Uptake , 1987 .
[12] G. Holz,et al. GTP-binding proteins mediate transmitter inhibition of voltage-dependent calcium channels , 1986, Nature.
[13] T. Yakushiji,et al. Intracellular calcium ions decrease the affinity of the GABA receptor , 1986, Nature.
[14] B. MacVicar,et al. Voltage-dependent calcium channels in glial cells. , 1984, Science.
[15] P. Eriksson,et al. Kappa-opioid receptors on astrocytes stimulate l-type Ca2+ channels , 1993, Neuroscience.
[16] Ito Seiji,et al. INVOLVEMENT OF PHOSPHOINOSITIDE METABOLISM IN GABA-INDUCED CATECHOLAMINE RELEASE FROM CULTURED BOVINE ADRENAL CHROMAFFIN CELLS , 1990 .
[17] R. Tapia,et al. Chelation of endogenous membrane calcium inhibits γ‐aminobutyric acid uptake in synaptosomes , 1989 .
[18] M. Crawford,et al. Potentiation by γ‐Aminobutyric Acid of α1‐Agonist‐Induced Accumulation of Inositol Phosphates in Slices of Rat Cerebral Cortex , 1990 .
[19] R. Nicoll,et al. A G protein couples serotonin and GABAB receptors to the same channels in hippocampus. , 1986, Science.
[20] S. Weiss,et al. Modulation of intracellular Ca++ in cultured astrocytes by influx through voltage‐activated Ca++ channels , 1991, Glia.
[21] H. Kettenmann,et al. GABA-activated Cl- channels in astrocytes of hippocampal slices , 1989, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[22] K. Morita,et al. GABAA receptor-mediated increase of cytosolic Ca2+ in isolated bovine adrenal chromaffin cells. , 1990, Biochimica et biophysica acta.
[23] E. Hansson,et al. Agonist‐evoked Ca2+ transients in primary astroglial cultures–modulatory effects of valproic acid , 1992, Glia.
[24] B. MacVicar,et al. Calcium activated potassium channels in cultured astrocytes , 1986, Neuroscience.
[25] M. Raff,et al. Glial cell development in the rat optic nerve , 1984, Trends in Neurosciences.
[26] A. Shahar. A Dissection and tissue culture manual of the nervous system , 1989 .
[27] J. Connor,et al. Depolarization- and transmitter-induced changes in intracellular Ca2+ of rat cerebellar granule cells in explant cultures , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[28] Börje Karlsson,et al. Cellular composition of primary cultures from cerebral cortex, striatum, hippocampus, brainstem and cerebellum , 1984, Brain Research.
[29] D. R. Curtis,et al. Phaclofen: a peripheral and central baclofen antagonist , 1987, Brain Research.
[30] K. McCarthy,et al. Schwann cells influence the expression of ganglioside GD3 by rat dorsal root ganglion neurons , 1989, Journal of Neuroimmunology.
[31] M. Davies,et al. gamma‐Aminobutyric acid hyperpolarizes rat hippocampal pyramidal cells through a calcium‐dependent potassium conductance. , 1986, The Journal of physiology.
[32] R. Wong,et al. GABAA receptor function is regulated by phosphorylation in acutely dissociated guinea‐pig hippocampal neurones. , 1990, The Journal of physiology.
[33] S. Mcguirk,et al. An investigation into the mechanisms of inhibition of calcium channel currents in cultured sensory neurones of the rat by guanine nucleotide analogues and (−)− baclofen , 1989, British journal of pharmacology.
[34] A. Dolphin,et al. Inhibition of calcium currents in cultured rat dorsal root ganglion neurones by (−)‐baclofen , 1986, British journal of pharmacology.
[35] R. Nicoll,et al. Direct hyperpolarizing action of baclofen on hippocampal pyramidal cells , 1984, Nature.
[36] K. Dunlap. TWO TYPES OF γ‐AMINOBUTYRIC ACID RECEPTOR ON EMBRYONIC SENSORY NEURONES , 1981 .
[37] P. Seeburg,et al. Molecular biology of the GABAA receptor: the receptor/channel superfamily , 1987, Trends in Neurosciences.
[38] E. Fedele,et al. Release of gamma-[3H]aminobutyric acid (GABA) from electrically stimulated rat cortical slices and its modulation by GABAB autoreceptors. , 1989, Journal of Pharmacology and Experimental Therapeutics.
[39] J. Young,et al. Ca2(+)-dependence provides evidence for differing mechanisms of GABA-induced inositol phosphate formation and GABA potentiation of inositol phosphate formation induced by noradrenaline in rat cerebral cortex. , 1990, Brain research. Molecular brain research.
[40] A. Schousboe,et al. GABA uptake inhibitors: relevance to antiepileptic drug research , 1987, Epilepsy Research.