Sodium pivalate reduces cardiac carnitine content and increases glucose oxidation without affecting cardiac functional capacity.

[1]  B. Wolf,et al.  Fatty acid oxidation and cardiac function in the sodium pivalate model of secondary carnitine deficiency. , 1995, Metabolism: clinical and experimental.

[2]  R. Barnard,et al.  Exercise capacity of rats remains unaffected by a chronic pressure overload. , 1993, Cardiovascular Research.

[3]  F. Haddad,et al.  Food restriction-induced transformations in cardiac functional and biochemical properties in rats. , 1993, Journal of applied physiology.

[4]  Q. Diep,et al.  Reduced carnitine and ketogenesis in the pivampicillin treated rat. , 1992, Biochemical pharmacology.

[5]  R. Jacob,et al.  Metabolically-modulated growth and phenotype of the rat heart. , 1992, European heart journal.

[6]  S. Lobatto,et al.  Amelioration of cardiac function by L-carnitine administration in patients on haemodialysis. , 1992, Contributions to nephrology.

[7]  N. Dhalla,et al.  Modification of subcellular organelles in pressure‐overloaded heart by etomoxir, a carnitine palmitoyltransferase I inhibitor , 1992, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[8]  H. Quinney,et al.  Carnitine stimulation of glucose oxidation in the fatty acid perfused isolated working rat heart. , 1992, The Journal of biological chemistry.

[9]  A. Davis,et al.  Sodium pivalate treatment reduces tissue carnitines and enhances ketosis in rats. , 1991, The Journal of nutrition.

[10]  G. Morris,et al.  Dietary effects on cardiac metabolic properties in rodents. , 1990, Journal of molecular and cellular cardiology.

[11]  S. Lindstedt,et al.  CARNITINE DEFICIENCY INDUCED BY PIVAMPICILLIN AND PIVMECILLINAM THERAPY , 1989, The Lancet.

[12]  L L Ji,et al.  Carnitine supplementation and depletion: tissue carnitines and enzymes in fatty acid oxidation. , 1987, Journal of applied physiology.

[13]  M. Schmidt,et al.  Improvement of myocardial function in diabetic rats after treatment with L-carnitine. , 1984, Metabolism: clinical and experimental.

[14]  J. Bremer.  Carnitine--metabolism and functions. , 1983, Physiological reviews.

[15]  S. Dimauro,et al.  Carnitine deficiency presenting as familial cardiomyopathy: a treatable defect in carnitine transport. , 1982, The Journal of pediatrics.

[16]  R. Bressler,et al.  Metabolic Control of Prevention of Nephropathy by 2-Tetradecylglycidate in the Diabetic Mouse (db/db) , 1982, Diabetes.

[17]  J. Hoh,et al.  Electrophoretic analysis of multiple forms of rat cardiac myosin: effects of hypophysectomy and thyroxine replacement. , 1978, Journal of molecular and cellular cardiology.

[18]  J. McGarry,et al.  An improved and simplified radioisotopic assay for the determination of free and esterified carnitine. , 1976, Journal of lipid research.

[19]  E. Newsholme,et al.  Inhibition of the Phosphofructokinase Reaction in Perfused Rat Heart by Respiration of Ketone Bodies, Fatty Acids and Pyruvate , 1962, Nature.

[20]  J. C. Finerty,et al.  Effects of a rat-free diet on growing female rats with special reference to the endocrine system. , 1953, The Journal of nutrition.

[21]  A. L. Carter.  Current concepts in carnitine research , 1992 .

[22]  B. Sempore,et al.  Large variations in skeletal muscle carnitine level fail to modify energy metabolism in exercising rats. , 1990, Comparative biochemistry and physiology. A, Comparative physiology.

[23]  C. Rebouche,et al.  Carnitine metabolism and function in humans. , 1986, Annual review of nutrition.

[24]  W. Dillmann.  Diabetes mellitus and hypothyroidism induce changes in myosin isoenzyme distribution in the rat heart--do alterations in fuel flux mediate these changes? , 1986, Advances in experimental medicine and biology.