A novel small compound accelerates dermal wound healing by modifying infiltration, proliferation and migration of distinct cellular components in mice.

[1]  Koichi Saito,et al.  Dermokine‐β impairs ERK signaling through direct binding to GRP78 , 2012, FEBS letters.

[2]  Raphael Rubin,et al.  Rubin's Pathology: Clinicopathologic Foundations of Medicine. , 2011 .

[3]  Y. Tabata,et al.  Differential contribution of dermal resident and bone marrow-derived cells to collagen production during wound healing and fibrogenesis in mice. , 2011, The Journal of investigative dermatology.

[4]  Y. Inagaki,et al.  A Novel Small Compound That Promotes Nuclear Translocation of YB-1 Ameliorates Experimental Hepatic Fibrosis in Mice* , 2010, The Journal of Biological Chemistry.

[5]  Hiroyuki Osada,et al.  A small-molecule inhibitor shows that pirin regulates migration of melanoma cells. , 2010, Nature chemical biology.

[6]  Y. Inagaki,et al.  Negligible contribution of bone marrow-derived cells to collagen production during hepatic fibrogenesis in mice. , 2009, Gastroenterology.

[7]  M. Fujimoto,et al.  A novel inhibitor of Smad-dependent transcriptional activation suppresses tissue fibrosis in mouse models of systemic sclerosis. , 2009, Arthritis and rheumatism.

[8]  C. Wicke,et al.  Aging influences wound healing in patients with chronic lower extremity wounds treated in a specialized wound care center , 2009, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

[9]  R. Hipskind,et al.  Erk5 controls Slug expression and keratinocyte activation during wound healing. , 2008, Molecular biology of the cell.

[10]  Olivera Stojadinovic,et al.  PERSPECTIVE ARTICLE: Growth factors and cytokines in wound healing , 2008, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

[11]  Y. Inagaki,et al.  Emerging insights into Transforming growth factor β Smad signal in hepatic fibrogenesis , 2007, Gut.

[12]  H. Okano,et al.  Bone marrow–derived cells express matrix metalloproteinases and contribute to regression of liver fibrosis in mice , 2007, Hepatology.

[13]  H. Kiyama,et al.  Cell type-specific intervention of transforming growth factor beta/Smad signaling suppresses collagen gene expression and hepatic fibrosis in mice. , 2005, Gastroenterology.

[14]  L. Hudson,et al.  Developmental transcription factor slug is required for effective re‐epithelialization by adult keratinocytes , 2005, Journal of cellular physiology.

[15]  Thomas Mustoe,et al.  Understanding chronic wounds: a unifying hypothesis on their pathogenesis and implications for therapy. , 2004, American journal of surgery.

[16]  Y. Inagaki,et al.  Interferon-γ Interferes with Transforming Growth Factor-β Signaling through Direct Interaction of YB-1 with Smad3* , 2003, Journal of Biological Chemistry.

[17]  Y. Inagaki,et al.  Y-box-binding Protein YB-1 Mediates Transcriptional Repression of Human α2(I) Collagen Gene Expression by Interferon-γ* , 2003, The Journal of Biological Chemistry.

[18]  T. Ling,et al.  Synthetic TGF‐β antagonist accelerates wound healing and reduces scarring , 2002 .

[19]  David R. Thomas Age-Related Changes in Wound Healing , 2001, Drugs & aging.

[20]  Anita B. Roberts,et al.  Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response , 1999, Nature Cell Biology.

[21]  P. Greenwel,et al.  Activation of Proalpha2(I) collagen promoter during hepatic fibrogenesis in transgenic mice. , 1998, Biochemical and biophysical research communications.

[22]  E. Kremmer,et al.  Identification of Pirin, a Novel Highly Conserved Nuclear Protein* , 1997, The Journal of Biological Chemistry.

[23]  Sin-Daw Lin,et al.  Effect of growth factors on dermal fibroblast contraction in normal skin and hypertrophic scar. , 1997, Journal of dermatological science.

[24]  K. Niederreither,et al.  A potent far-upstream enhancer in the mouse pro alpha 2(I) collagen gene regulates expression of reporter genes in transgenic mice , 1996, The Journal of cell biology.

[25]  Y. Inagaki,et al.  Transforming growth factor-beta stimulates alpha 2(I) collagen gene expression through a cis-acting element that contains an Sp1-binding site. , 1994, The Journal of biological chemistry.

[26]  D. Foreman,et al.  Neutralising antibody to TGF-beta 1,2 reduces cutaneous scarring in adult rodents. , 1994, Journal of cell science.

[27]  K. Yoshizato,et al.  A new type of biomaterial for artificial skin: dehydrothermally cross-linked composites of fibrillar and denatured collagens. , 1993, Journal of biomedical materials research.

[28]  H. Moses,et al.  Reversible inhibition of normal human prokeratinocyte proliferation by type beta transforming growth factor-growth inhibitor in serum-free medium. , 1986, Cancer research.