Intraabdominal adhesion formation is associated with differential mRNA expression of metabolic genes PDHb and SDHa
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[1] M. Wallwiener,et al. An Improved Model for the Induction of Experimental Adhesions , 2010, Journal of investigative surgery : the official journal of the Academy of Surgical Research.
[2] E. Stanbridge,et al. Regulation of gene expression by hypoxia: integration of the HIF-transduced hypoxic signal at the hypoxia-responsive element. , 2008, Clinica chimica acta; international journal of clinical chemistry.
[3] J. Hubbell,et al. RNA interference targeting hypoxia inducible factor 1alpha reduces post-operative adhesions in rats. , 2007, The Journal of surgical research.
[4] J. Becker,et al. Statins (HMG-CoA Reductase Inhibitors) Decrease Postoperative Adhesions by Increasing Peritoneal Fibrinolytic Activity , 2007, Annals of surgery.
[5] M. Wallwiener,et al. Innovative barriers for peritoneal adhesion prevention: liquid or solid? A rat uterine horn model. , 2006, Fertility and sterility.
[6] J. Becker,et al. A neurokinin 1 receptor antagonist decreases postoperative peritoneal adhesion formation and increases peritoneal fibrinolytic activity , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[7] D. Ann,et al. Crosstalk of hypoxia‐mediated signaling pathways in upregulating plasminogen activator inhibitor‐1 expression in keloid fibroblasts , 2004, Journal of cellular physiology.
[8] A. Chapman-Smith,et al. The mammalian basic helix-loop-helix/PAS family of transcriptional regulators. , 2004, The international journal of biochemistry & cell biology.
[9] P. Carmeliet,et al. Role of hypoxia inducible factors 1alpha and 2alpha in basal adhesion formation and in carbon dioxide pneumoperitoneum-enhanced adhesion formation after laparoscopic surgery in transgenic mice. , 2003, Fertility and sterility.
[10] G. Saed,et al. Expression of transforming growth factor beta isoform mRNA in injured peritoneum that healed with adhesions and without adhesions and in uninjured peritoneum. , 2003, Fertility and sterility.
[11] J. Becker,et al. Neurokinin-1 receptor and substance P messenger RNA levels increase during intraabdominal adhesion formation. , 2002, The Journal of surgical research.
[12] R. Wenger,et al. Cellular adaptation to hypoxia: O2‐sensing protein hydroxylases, hypoxia‐inducible transcription factors, and O2‐regulated gene expression , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[13] G. Saed,et al. Apoptosis and proliferation of human peritoneal fibroblasts in response to hypoxia. , 2002, Fertility and sterility.
[14] J. Campeau,et al. Peritoneal repair and post-surgical adhesion formation. , 2001, Human reproduction update.
[15] G. Laurent,et al. Human peritoneal adhesions are highly cellular, innervated, and vascularized , 2000, The Journal of pathology.
[16] Ag Johnson. Making and covering of surgical footprints , 1999, The Lancet.
[17] G. Semenza,et al. Hypoxia Response Elements in the Aldolase A, Enolase 1, and Lactate Dehydrogenase A Gene Promoters Contain Essential Binding Sites for Hypoxia-inducible Factor 1* , 1996, The Journal of Biological Chemistry.
[18] G. Semenza,et al. Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[19] K. von Wilpert,et al. The Potential of Liming to Improve Drought Tolerance of Norway Spruce [Picea abies (L.) Karst.] , 2019, Front. Plant Sci..
[20] D. Milligan,et al. Observations on the pathogenesis of peritoneal adhesions: a light and electron microscopical study , 1974, The British journal of surgery.
[21] H. Ellis. Post-Operative Intra-Abdominal Adhesions , 1962, Nature.
[22] K. Hirota. Hypoxia-inducible factor 1, a master transcription factor of cellular hypoxic gene expression , 2002, Journal of Anesthesia.
[23] R. Buckman,et al. A unifying pathogenetic mechanism in the etiology of intraperitoneal adhesions;. , 1976, The Journal of surgical research.