GTPγ causes contraction of skinned frog skeletal muscle via the DHP-sensitive Ca2+ channels of sealed T-tubules
暂无分享,去创建一个
[1] E. Stefani,et al. Charge movement and calcium currents in skeletal muscle fibers are enhanced by GTPγS , 1990, Pflügers Archiv.
[2] G. P. Reid,et al. Synthesis and properties of caged nucleotides. , 1990, Methods in enzymology.
[3] G. Lamb,et al. Calcium release in skinned muscle fibres of the toad by transverse tubule depolarization or by direct stimulation. , 1990, The Journal of physiology.
[4] J. Bockaert,et al. Cellular distribution and biochemical characterization of G proteins in skeletal muscle: comparative location with voltage‐dependent calcium channels. , 1990, The EMBO journal.
[5] A. Caswell,et al. Does muscle activation occur by direct mechanical coupling of transverse tubules to sarcoplasmic reticulum? , 1989, Trends in Biochemical Sciences.
[6] M. Fill,et al. Block of contracture in skinned frog skeletal muscle fibers by calcium antagonists , 1989, The Journal of general physiology.
[7] K. Campbell,et al. Structural evidence for direct interaction between the molecular components of the transverse tubule/sarcoplasmic reticulum junction in skeletal muscle , 1988, The Journal of cell biology.
[8] M. Trabucchi,et al. Direct coupling of a G-protein to dihydropyridine binding sites. , 1988, Biochemical and biophysical research communications.
[9] G. P. Reid,et al. Photolabile 1-(2-nitrophenyl)ethyl phosphate esters of adenine nucleotide analogs. Synthesis and mechanism of photolysis , 1988 .
[10] J. Bockaert,et al. G-proteins in skeletal muscle. Evidence for a 40 kDa pertussis-toxin substrate in purified transverse tubules. , 1988, The Biochemical journal.
[11] T. Walseth,et al. Voltage dependence of inositol 1,4,5-trisphosphate-induced Ca2+ release in peeled skeletal muscle fibers. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[12] S. Cockcroft,et al. G-proteins, the inositol lipid signalling pathway, and secretion. , 1988, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.
[13] A. Brown,et al. The stimulatory G protein of adenylyl cyclase, Gs, also stimulates dihydropyridine-sensitive Ca2+ channels. Evidence for direct regulation independent of phosphorylation by cAMP-dependent protein kinase or stimulation by a dihydropyridine agonist. , 1988, The Journal of biological chemistry.
[14] A. Dolphin,et al. Photoactivation of intracellular guanosine triphosphate analogues reduces the amplitude and slows the kinetics of voltage-activated calcium channel currents in sensory neurones , 1988, Pflügers Archiv.
[15] R. Oswald,et al. Functional reconstitution of skeletal muscle Ca2+ channels: separation of regulatory and channel components. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[16] E. Stefani,et al. Effects of extracellular calcium on calcium movements of excitation‐contraction coupling in frog skeletal muscle fibres. , 1988, The Journal of physiology.
[17] G. Rapp,et al. A low cost high intensity flash device for photolysis experiments , 1988, Pflügers Archiv.
[18] M. Entman,et al. G-protein distribution in canine cardiac sarcoplasmic reticulum and sarcolemma: comparison to rabbit skeletal muscle membranes and to brain and erythrocyte G-proteins. , 1987, Archives of biochemistry and biophysics.
[19] G. Lamb,et al. Calcium currents, charge movement and dihydropyridine binding in fast‐ and slow‐twitch muscles of rat and rabbit. , 1987, The Journal of physiology.
[20] Y. E. Goldman,et al. Kinetics of smooth and skeletal muscle activation by laser pulse photolysis of caged inositol 1,4,5-trisphosphate , 1987, Nature.
[21] E. Ríos,et al. Involvement of dihydropyridine receptors in excitation–contraction coupling in skeletal muscle , 1987, Nature.
[22] P. J. Griffiths,et al. An examination of the ability ofinositol 1,4,5‐trisphosphate to induce calcium release and tension development in skinned skeletal muscle fibres of frog and crustacea , 1986, FEBS letters.
[23] A. Dolphin,et al. Regulation of calcium currents by a GTP analogue: Potentiation of (−)-baclofen-mediated inhibition , 1986, Neuroscience Letters.
[24] F. Di Virgilio,et al. Is a guanine nucleotide‐binding protein involved in excitation‐contraction coupling in skeletal muscle? , 1986, The EMBO journal.
[25] S. K. Donaldson,et al. Peeled mammalian skeletal muscle fibers. Possible stimulation of Ca2+ release via a transverse tubule-sarcoplasmic reticulum mechanism , 1985, The Journal of general physiology.
[26] R. Eisenberg,et al. Paralysis of frog skeletal muscle fibres by the calcium antagonist D‐600. , 1983, The Journal of physiology.
[27] G. Meissner,et al. Sodium-calcium ion exchange in skeletal muscle sarcolemmal vesicles , 1982, The Journal of Membrane Biology.
[28] E. Stefani,et al. Effect of glycerol treatment on the calcium current of frog skeletal muscle. , 1980, The Journal of physiology.
[29] M. Endo,et al. Calcium release from the sarcoplasmic reticulum. , 1977, Physiological reviews.
[30] A. Somlyo,et al. Flash photolysis studies of excitation-contraction coupling, regulation, and contraction in smooth muscle. , 1990, Annual review of physiology.
[31] G. P. Reid,et al. [16] Synthesis and properties of caged nucleotides , 1989 .
[32] M. Toutant,et al. G-protein dependent potentiation of calcium release from sarcoplasmic reticulum of skeletal muscle. , 1989, Cellular signalling.
[33] H. Kalbitzer,et al. Characterisation of the metal-ion-GDP complex at the active sites of transforming and nontransforming p21 proteins by observation of the 17O-Mn superhyperfine coupling and by kinetic methods. , 1987, European journal of biochemistry.
[34] Arthur E. Martell,et al. Stability constants of metal-ion complexes , 1964 .