Anandamide-induced cell death in primary neuronal cultures: role of calpain and caspase pathways
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R. Vink | A. Yakovlev | B. Stoica | A. Faden | P. M. Lea | S. Knoblach | V. Movsesyan | I. Cernak | Ibolja Cernak | Alexander G. Yakovlev | Paul M Lea | Robert Vink
[1] I. Maruyama,et al. Anandamide induces cell death independently of cannabinoid receptors or vanilloid receptor 1: possible involvement of lipid rafts , 2003, Cellular and Molecular Life Sciences CMLS.
[2] D. Owen,et al. Comparison of effects of anandamide at recombinant and endogenous rat vanilloid receptors. , 2002, British journal of anaesthesia.
[3] A. Yakovlev,et al. Ceramide induces neuronal apoptosis through the caspase-9/caspase-3 pathway. , 2002, Biochemical and biophysical research communications.
[4] R. Giffard,et al. Caspase inhibitors reduce the apoptotic but not necrotic component of kainate injury in primary murine cortical neuronal cultures , 2002, Neurological research.
[5] H. Hansen,et al. Putative neuroprotective actions of N-acyl-ethanolamines. , 2002, Pharmacology & therapeutics.
[6] Oliver Popp,et al. Ionomycin-activated Calpain Triggers Apoptosis , 2002, The Journal of Biological Chemistry.
[7] C. Fowler,et al. ‘Entourage’ effects of N‐acyl ethanolamines at human vanilloid receptors. Comparison of effects upon anandamide‐induced vanilloid receptor activation and upon anandamide metabolism , 2002, British journal of pharmacology.
[8] S. Milstien,et al. Ceramide‐induced cell death in primary neuronal cultures: Upregulation of ceramide levels during neuronal apoptosis , 2002, Journal of neuroscience research.
[9] E. Fride. Endocannabinoids in the central nervous system--an overview. , 2002, Prostaglandins, leukotrienes, and essential fatty acids.
[10] Kevin K W Wang,et al. Selective release of calpain produced alphalI-spectrin (alpha-fodrin) breakdown products by acute neuronal cell death. , 2002, Biological chemistry.
[11] A. Finazzi-Agro’,et al. Endocannabinoids and their actions. , 2002, Vitamins and hormones.
[12] T. Sugawara,et al. Evidence of Phosphorylation of Akt and Neuronal Survival after Transient Focal Cerebral Ischemia in Mice , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[13] C. Fowler,et al. Inhibition of rat C6 glioma cell proliferation by endogenous and synthetic cannabinoids. Relative involvement of cannabinoid and vanilloid receptors. , 2001, The Journal of pharmacology and experimental therapeutics.
[14] K. Nicolay,et al. Exogenous Anandamide Protects Rat Brain against Acute Neuronal Injury In Vivo , 2001, The Journal of Neuroscience.
[15] S. Ben-Shabat,et al. An endogenous cannabinoid (2-AG) is neuroprotective after brain injury , 2001, Nature.
[16] P. Vallet,et al. Prevention of apoptotic neuronal death by controlling procaspases? A point of view , 2001, Brain Research Reviews.
[17] A. Yakovlev,et al. Differential Expression of Apoptotic Protease-Activating Factor-1 and Caspase-3 Genes and Susceptibility to Apoptosis during Brain Development and after Traumatic Brain Injury , 2001, The Journal of Neuroscience.
[18] V. Sée,et al. Oxidative Stress Induces Neuronal Death by Recruiting a Protease and Phosphatase-gated Mechanism* , 2001, The Journal of Biological Chemistry.
[19] J. Manzanares,et al. Anandamide, but not 2‐arachidonoylglycerol, accumulates during in vivo neurodegeneration , 2001, Journal of neurochemistry.
[20] M. Iadarola,et al. Anandamide Activates Vanilloid Receptor 1 (VR1) at Acidic pH in Dorsal Root Ganglia Neurons and Cells Ectopically Expressing VR1* , 2001, The Journal of Biological Chemistry.
[21] D. Lambert,et al. Temperature-dependent activation of recombinant rat vanilloid VR1 receptors expressed in HEK293 cells by capsaicin and anandamide. , 2001, European journal of pharmacology.
[22] A. Finazzi-Agro’,et al. The Activity of Anandamide at Vanilloid VR1 Receptors Requires Facilitated Transport across the Cell Membrane and Is Limited by Intracellular Metabolism* , 2001, The Journal of Biological Chemistry.
[23] A. Randall,et al. Characterisation using FLIPR of human vanilloid VR1 receptor pharmacology. , 2001, European journal of pharmacology.
[24] C. Fowler,et al. Characterization of palmitoylethanolamide transport in mouse Neuro‐2a neuroblastoma and rat RBL‐2H3 basophilic leukaemia cells: comparison with anandamide , 2001, British journal of pharmacology.
[25] Changlian Zhu,et al. Synergistic Activation of Caspase-3 by m-Calpain after Neonatal Hypoxia-Ischemia , 2001, The Journal of Biological Chemistry.
[26] J. Gil,et al. Low concentrations of 1‐methyl‐4‐phenylpyridinium ion induce caspase‐mediated apoptosis in human SH‐SY5Y neuroblastoma cells , 2001, Journal of neuroscience research.
[27] S. Hansen,et al. Accumulation of the anandamide precursor and other N‐acylethanolamine phospholipids in infant rat models of in vivo necrotic and apoptotic neuronal death , 2001, Journal of neurochemistry.
[28] D. Green,et al. Chapter 16 The (Holey) study of mitochondria in apoptosis , 2001 .
[29] A. Faden,et al. mGluR5 antagonists 2-methyl-6-(phenylethynyl)-pyridine and (E)-2-methyl-6-(2-phenylethenyl)-pyridine reduce traumatic neuronal injury in vitro and in vivo by antagonizing N-methyl-D-aspartate receptors. , 2001, The Journal of pharmacology and experimental therapeutics.
[30] D. Green,et al. The (Holey) study of mitochondria in apoptosis. , 2001, Methods in cell biology.
[31] A. Thor,et al. Reconstitution of caspase 3 sensitizes MCF-7 breast cancer cells to doxorubicin- and etoposide-induced apoptosis. , 2001, Cancer research.
[32] M. Mezna,et al. Subtype identification and functional properties of inositol 1,4, 5-trisphosphate receptors in heart and aorta. , 2000, Pharmacological research.
[33] Honglin Zhou,et al. Akt Regulates Cell Survival and Apoptosis at a Postmitochondrial Level , 2000, The Journal of cell biology.
[34] G. Melino,et al. Anandamide Induces Apoptosis in Human Cells via Vanilloid Receptors , 2000, The Journal of Biological Chemistry.
[35] C. Fowler,et al. Differences in the pharmacological properties of rat and chicken brain fatty acid amidohydrolase , 2000, British journal of pharmacology.
[36] K. Willoughby,et al. Influence of phenylmethylsulfonyl fluoride on anandamide brain levels and pharmacological effects. , 2000, Life sciences.
[37] R. Prinjha,et al. Characterization using FLIPR of rat vanilloid receptor (rVR1) pharmacology , 2000, British journal of pharmacology.
[38] D. Smart,et al. Anandamide: an endogenous activator of the vanilloid receptor. , 2000, Trends in pharmacological sciences.
[39] D. Greenberg,et al. Endocannabinoids protect cerebral cortical neurons from in vitro ischemia in rats , 2000, Neuroscience Letters.
[40] K. Waku,et al. Evidence That 2-Arachidonoylglycerol but Not N-Palmitoylethanolamine or Anandamide Is the Physiological Ligand for the Cannabinoid CB2 Receptor , 2000, The Journal of Biological Chemistry.
[41] J. Chambers,et al. The endogenous lipid anandamide is a full agonist at the human vanilloid receptor (hVR1) , 2000, British journal of pharmacology.
[42] M. Mattson,et al. Caspase and calpain substrates: Roles in synaptic plasticity and cell death , 1999, Journal of neuroscience research.
[43] P. Blumberg,et al. The cloned rat vanilloid receptor VR1 mediates both R-type binding and C-type calcium response in dorsal root ganglion neurons. , 1999, Molecular pharmacology.
[44] W. Graier,et al. Anandamide‐induced mobilization of cytosolic Ca2+ in endothelial cells , 1999, British journal of pharmacology.
[45] R. Pertwee,et al. Agonist‐inverse agonist characterization at CB1 and CB2 cannabinoid receptors of L759633, L759656 and AM630 , 1999, British journal of pharmacology.
[46] N. Thornberry,et al. Inhibition of Human Caspases by Peptide-based and Macromolecular Inhibitors* , 1998, The Journal of Biological Chemistry.
[47] Eugene M. Johnson,et al. Evidence of a Novel Event during Neuronal Death Development of Competence-to-Die in Response to Cytoplasmic Cytochrome c , 1998, Neuron.
[48] B. Pike,et al. Regional calpain and caspase‐3 proteolysis of α‐spectrin after traumatic brain injury , 1998, Neuroreport.
[49] Alan G. Porter,et al. Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with Apoptosis* , 1998, The Journal of Biological Chemistry.
[50] V. Dixit,et al. Caspase-9, Bcl-XL, and Apaf-1 Form a Ternary Complex* , 1998, The Journal of Biological Chemistry.
[51] M. Scanziani,et al. Dual Effects of Anandamide on NMDA Receptor‐Mediated Responses and Neurotransmission , 1998, Journal of neurochemistry.
[52] J. Sutcliffe,et al. Fatty acid amide hydrolase, the degradative enzyme for anandamide and oleamide, has selective distribution in neurons within the rat central nervous system , 1997, Journal of neuroscience research.
[53] S. Srinivasula,et al. Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade , 1997, Cell.
[54] A. Yakovlev,et al. The Role of CED-3-Related Cysteine Proteases in Apoptosis of Cerebellar Granule Cells , 1997, The Journal of Neuroscience.
[55] N. Volkow,et al. 123I-labeled AM251: a radioiodinated ligand which binds in vivo to mouse brain cannabinoid CB1 receptors. , 1996, European journal of pharmacology.
[56] K. Mackie,et al. Comparison of the pharmacology and signal transduction of the human cannabinoid CB1 and CB2 receptors. , 1995, Molecular pharmacology.
[57] Y. Saito,et al. Identification and characterization of membrane-bound calpains in clathrin-coated vesicles from bovine brain. , 1995, European journal of biochemistry.
[58] Keizo Yoshida,et al. Non-competitive NMDA antagonists, FR115427 and MK801, enhance neuronal survival in primary culture , 1995, Neuroscience Letters.
[59] A. Leiser,et al. Oxidative stress aspects of the cytotoxicity of carbamide peroxide: in vitro studies. , 1995, Toxicology letters.
[60] J. Schwartz,et al. Formation and inactivation of endogenous cannabinoid anandamide in central neurons , 1994, Nature.
[61] S. Furukawa,et al. Etoposide induces programmed death in neurons cultured from the fetal rat central nervous system , 1994, Brain Research.