Molecular and cellular mechanisms of myocardial failure.
暂无分享,去创建一个
[1] G. Feuerstein,et al. Protective effects of carvedilol in the myocardium. , 1997, The American journal of cardiology.
[2] M. Bristow. Mechanism of action of beta-blocking agents in heart failure. , 1997, The American journal of cardiology.
[3] N. Bishopric,et al. Atrial Natriuretic Peptide Induces Apoptosis in Neonatal Rat Cardiac Myocytes* , 1997, The Journal of Biological Chemistry.
[4] W. Lederer,et al. Defective excitation-contraction coupling in experimental cardiac hypertrophy and heart failure. , 1997, Science.
[5] C A Beltrami,et al. Apoptosis in the failing human heart. , 1997, The New England journal of medicine.
[6] E. Pennisi. Superoxides Relay Ras Protein's Oncogenic Message , 1997, Science.
[7] A. Takeshita,et al. Role of Myocyte Nitric Oxide in β-Adrenergic Hyporesponsiveness in Heart Failure , 1997 .
[8] P. Anversa,et al. Angiotensin II Induces Apoptosis of Adult Ventricular MyocytesIn Vitro , 1997 .
[9] A. Saraste,et al. Apoptosis in human acute myocardial infarction. , 1997, Circulation.
[10] D. Zechner,et al. Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes. Involvement of the sphingolipid signaling cascade in cardiac cell death. , 1996, The Journal of clinical investigation.
[11] Y. Sugishita,et al. Inhibition of myocardial endothelin pathway improves long-term survival in heart failure , 1996, Nature.
[12] R. Virmani,et al. Apoptosis in myocytes in end-stage heart failure. , 1996, The New England journal of medicine.
[13] W. Colucci. Apoptosis in the heart. , 1996, The New England journal of medicine.
[14] P. Singal,et al. Role of oxidative stress in transition of hypertrophy to heart failure. , 1996, Journal of the American College of Cardiology.
[15] L. Gaboury,et al. Apoptosis in pressure overload-induced heart hypertrophy in the rat. , 1996, The Journal of clinical investigation.
[16] J. Heath,et al. Cardiotrophin-1 Activates a Distinct Form of Cardiac Muscle Cell Hypertrophy , 1996, The Journal of Biological Chemistry.
[17] H. Oral,et al. Proinflammatory cytokine levels in patients with depressed left ventricular ejection fraction: a report from the Studies of Left Ventricular Dysfunction (SOLVD). , 1996, Journal of the American College of Cardiology.
[18] P. Tsao,et al. Expression of inducible nitric oxide synthase in human heart failure. , 1996, Circulation.
[19] W. Colucci. Myocardial endothelin. Does it play a role in myocardial failure? , 1996, Circulation.
[20] Y. Sugishita,et al. Endogenous endothelin-1 participates in the maintenance of cardiac function in rats with congestive heart failure. Marked increase in endothelin-1 production in the failing heart. , 1996, Circulation.
[21] T. Ogihara,et al. Nitric oxide induces upregulation of Fas and apoptosis in vascular smooth muscle. , 1996, Hypertension.
[22] D. Mann,et al. Tumor Necrosis Factor-α and Tumor Necrosis Factor Receptors in the Failing Human Heart , 1996 .
[23] R Aikawa,et al. Endothelin-1 Is Involved in Mechanical Stress-induced Cardiomyocyte Hypertrophy (*) , 1996, The Journal of Biological Chemistry.
[24] John Calvin Reed,et al. Apoptotic and necrotic myocyte cell deaths are independent contributing variables of infarct size in rats. , 1996, Laboratory investigation; a journal of technical methods and pathology.
[25] P. Anversa,et al. Myocyte nuclear mitotic division and programmed myocyte cell death characterize the cardiac myopathy induced by rapid ventricular pacing in dogs. , 1995, Laboratory investigation; a journal of technical methods and pathology.
[26] E. Sonnenblick,et al. Stretch-induced programmed myocyte cell death. , 1995, The Journal of clinical investigation.
[27] N. Takahashi,et al. Pressure- and volume-induced left ventricular hypertrophies are associated with distinct myocyte phenotypes and differential induction of peptide growth factor mRNAs. , 1995, Circulation.
[28] M. Creager,et al. Nitric Oxide Inhibits the Positive Inotropic Response to β-Adrenergic Stimulation in Humans With Left Ventricular Dysfunction , 1995 .
[29] M. Radomski,et al. Myocardial calcium-independent nitric oxide synthase activity is present in dilated cardiomyopathy, myocarditis, and postpartum cardiomyopathy but not in ischaemic or valvar heart disease. , 1995, British heart journal.
[30] K. Teng,et al. p21ras as a Common Signaling Target of Reactive Free Radicals and Cellular Redox Stress (*) , 1995, The Journal of Biological Chemistry.
[31] J. Hare,et al. Role of nitric oxide in the regulation of myocardial function. , 1995, Progress in cardiovascular diseases.
[32] N. Takahashi,et al. Interleukin-1 beta modulates the growth and phenotype of neonatal rat cardiac myocytes. , 1995, The Journal of clinical investigation.
[33] J. Cohn. Structural basis for heart failure. Ventricular remodeling and its pharmacological inhibition. , 1995, Circulation.
[34] P. Cannon,et al. The lethal effects of cytokine-induced nitric oxide on cardiac myocytes are blocked by nitric oxide synthase antagonism or transforming growth factor beta. , 1995, The Journal of clinical investigation.
[35] C. H. Conrad,et al. Myocardial fibrosis and stiffness with hypertrophy and heart failure in the spontaneously hypertensive rat. , 1995, Circulation.
[36] C. Lowenstein,et al. Cytokine-inducible nitric oxide synthase (iNOS) expression in cardiac myocytes. Characterization and regulation of iNOS expression and detection of iNOS activity in single cardiac myocytes in vitro. , 1994, The Journal of biological chemistry.
[37] R. Kloner,et al. Reperfusion injury induces apoptosis in rabbit cardiomyocytes. , 1994, The Journal of clinical investigation.
[38] N. Takahashi,et al. Hypertrophic stimuli induce transforming growth factor-beta 1 expression in rat ventricular myocytes. , 1994, The Journal of clinical investigation.
[39] A. Katz. The Cardiomyopathy of Overload: An Unnatural Growth Response in the Hypertrophied Heart , 1994, Annals of Internal Medicine.
[40] C. H. Conrad,et al. Alterations in cardiac gene expression during the transition from stable hypertrophy to heart failure. Marked upregulation of genes encoding extracellular matrix components. , 1994, Circulation research.
[41] U. Ikeda,et al. Neutrophil Adherence to Rat Cardiac Myocyte by Proinflammatory Cytokines , 1994, Journal of cardiovascular pharmacology.
[42] P. Singal,et al. Antioxidant changes in hypertrophied and failing guinea pig hearts. , 1994, The American journal of physiology.
[43] K. Webster,et al. Regulation of fos and jun immediate-early genes by redox or metabolic stress in cardiac myocytes. , 1994, Circulation research.
[44] M. Wilson,et al. Integrin-mediated collagen gel contraction by cardiac fibroblasts. Effects of angiotensin II. , 1994, Circulation research.
[45] J. Sadoshima,et al. Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro , 1993, Cell.
[46] K. Weber,et al. Effects of endothelins on collagen turnover in cardiac fibroblasts. , 1993, Cardiovascular research.
[47] J. LaPres,et al. Regulation of hypertrophy and atrophy in cultured adult heart cells. , 1993, Circulation research.
[48] M. Bristow. Changes in myocardial and vascular receptors in heart failure. , 1993, Journal of the American College of Cardiology.
[49] J. Sadoshima,et al. Critical Role of the AT1 Receptor Subtype , 2005 .
[50] K. Chien,et al. The alpha 1A-adrenergic receptor subtype mediates biochemical, molecular, and morphologic features of cultured myocardial cell hypertrophy. , 1993, The Journal of biological chemistry.
[51] B. Lorell,et al. Selective changes in cardiac gene expression during compensated hypertrophy and the transition to cardiac decompensation in rats with chronic aortic banding. , 1993, Circulation research.
[52] T. Yue,et al. Myocardial protection by the novel vasodilating beta-blocker, carvedilol: potential relevance of anti-oxidant activity. , 1993, Journal of hypertension. Supplement : official journal of the International Society of Hypertension.
[53] P. Anversa,et al. Regulation of angiotensin II receptors on ventricular myocytes after myocardial infarction in rats. , 1993, Circulation research.
[54] J. Balligand,et al. Abnormal contractile function due to induction of nitric oxide synthesis in rat cardiac myocytes follows exposure to activated macrophage-conditioned medium. , 1993, The Journal of clinical investigation.
[55] D. Ganten,et al. Selective activation of cardiac angiotensinogen gene expression in post-infarction ventricular remodeling in the rat. , 1993, Journal of molecular and cellular cardiology.
[56] N. Alpert,et al. Alterations in sarcoplasmic reticulum gene expression in human heart failure. A possible mechanism for alterations in systolic and diastolic properties of the failing myocardium. , 1993, Circulation research.
[57] P. Poole‐Wilson,et al. Nitric oxide production within cardiac myocytes reduces their contractility in endotoxemia. , 1992, The American journal of physiology.
[58] W Grossman,et al. Expression of dihydropyridine receptor (Ca2+ channel) and calsequestrin genes in the myocardium of patients with end-stage heart failure. , 1992, The Journal of clinical investigation.
[59] S. Izumo,et al. Expression of A-, B-, and C-type natriuretic peptide genes in failing and developing human ventricles. Correlation with expression of the Ca(2+)-ATPase gene. , 1992, Circulation research.
[60] T. Takahashi,et al. Differences in cardiac calcium release channel (ryanodine receptor) expression in myocardium from patients with end-stage heart failure caused by ischemic versus dilated cardiomyopathy. , 1992, Circulation research.
[61] J. Sadoshima,et al. Molecular characterization of the stretch-induced adaptation of cultured cardiac cells. An in vitro model of load-induced cardiac hypertrophy. , 1992, The Journal of biological chemistry.
[62] S. Moncada,et al. Induction and potential biological relevance of a Ca2+‐independent nitric oxide synthase in the myocardium , 1992, British journal of pharmacology.
[63] E Erdmann,et al. Intracellular Calcium Handling in Isolated Ventricular Myocytes From Patients With Terminal Heart Failure , 1992, Circulation.
[64] M. Entman,et al. Neutrophil adherence to isolated adult cardiac myocytes. Induction by cardiac lymph collected during ischemia and reperfusion. , 1992, The Journal of clinical investigation.
[65] B. Parsons,et al. Adrenergic Effects on the Biology of the Adult Mammalian Cardiocyte , 1992, Circulation.
[66] P. Allen,et al. Troponin T isoform expression in the normal and failing human left ventricle: a correlation with myofibrillar ATPase activity. , 1992, Basic research in cardiology.
[67] H. Schunkert,et al. Tissue-specific activation of cardiac angiotensin converting enzyme in experimental heart failure. , 1991, Circulation research.
[68] K. Weber,et al. Pathological Hypertrophy and Cardiac Interstitium: Fibrosis and Renin‐Angiotensin‐Aldosterone System , 1991, Circulation.
[69] A. Feldman,et al. Selective Gene Expression in Failing Human Heart: Quantification of Steady‐State Levels of Messenger RNA in Endomyocardial Biopsies Using the Polymerase Chain Reaction , 1991, Circulation.
[70] T K Borg,et al. Expression of collagen binding integrins during cardiac development and hypertrophy. , 1991, Circulation research.
[71] K. Chien,et al. Endothelin induction of inositol phospholipid hydrolysis, sarcomere assembly, and cardiac gene expression in ventricular myocytes. A paracrine mechanism for myocardial cell hypertrophy. , 1990, The Journal of biological chemistry.
[72] H. Fillit,et al. Elevated circulating levels of tumor necrosis factor in severe chronic heart failure. , 1990, The New England journal of medicine.
[73] M. Sporn,et al. Transforming Growth Factor‐β1 in Normal Heart and in Myocardial Infarction , 1990 .
[74] G. Murrell,et al. Modulation of fibroblast proliferation by oxygen free radicals , 1990 .
[75] T. Parker,et al. Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes. , 1990, The Journal of clinical investigation.
[76] W. Cassells. Transforming growth factor β_1 in normal heart and in myocardial infarction. , 1990 .
[77] G. Habermehl,et al. Human fibroblasts release reactive oxygen species in response to interleukin-1 or tumour necrosis factor-alpha. , 1989, The Biochemical journal.
[78] P. Ludmer,et al. Intracoronary infusion of dobutamine to patients with and without severe congestive heart failure. Dose-response relationships, correlation with circulating catecholamines, and effect of phosphodiesterase inhibition. , 1988, The Journal of clinical investigation.
[79] G Olivetti,et al. Quantitative structural analysis of the myocardium during physiologic growth and induced cardiac hypertrophy: a review. , 1986, Journal of the American College of Cardiology.
[80] P. Simpson. Norepinephrine-stimulated hypertrophy of cultured rat myocardial cells is an alpha 1 adrenergic response. , 1983, The Journal of clinical investigation.
[81] T. Kiss,et al. Effect of Ca-free medium on the ultrastructure and excitability of the myocardial cells of the snail, Helix pomatia L. , 1973, Journal of molecular and cellular cardiology.