Kinesin family member 14 is a candidate prognostic marker for outcome of glioma patients.

[1]  Gang Zhao,et al.  Kinesin family member 2C (KIF2C/MCAK) is a novel marker for prognosis in human gliomas , 2012, Clinical Neurology and Neurosurgery.

[2]  B. Gallie,et al.  Kinesin family member 14: An independent prognostic marker and potential therapeutic target for ovarian cancer , 2012, International journal of cancer.

[3]  Y. Tu,et al.  Increased Expression of FAT10 is Correlated with Progression and Prognosis of Human Glioma , 2012, Pathology & Oncology Research.

[4]  Gang Li,et al.  Downregulation of N-Myc downstream-regulated gene 4 influences patient survival in gliomas , 2012, Brain Tumor Pathology.

[5]  Y. Tu,et al.  Overexpression of a disintegrin and metalloprotease 8 in human gliomas is implicated in tumor progression and prognosis , 2012, Medical Oncology.

[6]  Masahiro Toda,et al.  Downregulation of KIF23 suppresses glioma proliferation , 2012, Journal of Neuro-Oncology.

[7]  M. J. van de Vijver,et al.  External Validation of Adjuvant! Online Breast Cancer Prognosis Tool. Prioritising Recommendations for Improvement , 2011, PloS one.

[8]  J. J. Thompson,et al.  Canine Subcutaneous Mast Cell Tumors , 2011, Veterinary pathology.

[9]  Yue Yu,et al.  The role of kinesin family proteins in tumorigenesis and progression , 2010, Cancer.

[10]  L. Taylor.  Diagnosis, treatment, and prognosis of glioma , 2010, Neurology.

[11]  R. Roesler,et al.  Current and emerging molecular targets in glioma , 2010, Expert review of anticancer therapy.

[12]  E. Dunbar,et al.  Glioma Diagnosis: Immunohistochemistry and Beyond , 2010, Advances in anatomic pathology.

[13]  B. Jarzab,et al.  Metal-proteinase ADAM12, kinesin 14 and checkpoint suppressor 1 as new molecular markers of laryngeal carcinoma , 2009, European Archives of Oto-Rhino-Laryngology.

[14]  K. Mallikarjuna,et al.  KIF14 and E2F3 mRNA expression in human retinoblastoma and its phenotype association , 2009, Molecular vision.

[15]  Tae-Min Kim,et al.  Clinical implication of recurrent copy number alterations in hepatocellular carcinoma and putative oncogenes in recurrent gains on 1q , 2008, International journal of cancer.

[16]  T. Corson,et al.  High expression of KIF14 in retinoblastoma: association with older age at diagnosis. , 2007, Investigative ophthalmology & visual science.

[17]  T. Corson,et al.  KIF14 Messenger RNA Expression Is Independently Prognostic for Outcome in Lung Cancer , 2007, Clinical Cancer Research.

[18]  T. Corson,et al.  KIF14 mRNA expression is a predictor of grade and outcome in breast cancer , 2006, International journal of cancer.

[19]  T. Misteli,et al.  Tumor Formation via Loss of a Molecular Motor Protein , 2006, Current Biology.

[20]  Peter S. Linsley,et al.  RNA Interference-Mediated Silencing of Mitotic Kinesin KIF14 Disrupts Cell Cycle Progression and Induces Cytokinesis Failure , 2006, Molecular and Cellular Biology.

[21]  Erich A. Nigg,et al.  KIF14 and citron kinase act together to promote efficient cytokinesis , 2006, The Journal of cell biology.

[22]  Nobutaka Hirokawa,et al.  Analysis of the kinesin superfamily: insights into structure and function. , 2005, Trends in cell biology.

[23]  Timothy W Corson,et al.  KIF14 is a candidate oncogene in the 1q minimal region of genomic gain in multiple cancers , 2005, Oncogene.

[24]  Russell L. Malmberg,et al.  A standardized kinesin nomenclature , 2004, The Journal of cell biology.

[25]  T. Misteli,et al.  Human chromokinesin KIF4A functions in chromosome condensation and segregation , 2004, The Journal of cell biology.

[26]  N. Hirokawa,et al.  All kinesin superfamily protein, KIF, genes in mouse and human , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[27]  N. Nomura,et al.  Prediction of the coding sequences of unidentified human genes. IV. The coding sequences of 40 new genes (KIAA0121-KIAA0160) deduced by analysis of cDNA clones from human cell line KG-1. , 1995, DNA research : an international journal for rapid publication of reports on genes and genomes.

[28]  Michael P. Sheetz,et al.  Identification of a novel force-generating protein, kinesin, involved in microtubule-based motility , 1985, Cell.