Essential myosin light chain as a target for caspase-3 in failing myocardium
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Martin Ungerer | Karl-Ludwig Laugwitz | Albert Schömig | Melchior Seyfarth | M. Seyfarth | A. Schömig | K. Laugwitz | T. Ott | D. Grewe | Lorenz Bott-Flügel | Alessandra Moretti | A. Moretti | Hans-Peter Holthoff | Hans-Jörg Weig | Thomas Ott | Diana Grewe | Angelika Gillitzer | Lorenz Bott-Flügel | M. Ungerer | H. Holthoff | H. Weig | A. Gillitzer
[1] Emad S. Alnemri,et al. A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosis , 2001, Nature.
[2] O. Hess,et al. Hemodynamic performance and myosin light chain-1 expression of the hypertrophied left ventricle in aortic valve disease before and after valve replacement. , 1992, Circulation research.
[3] K. Trybus,et al. Skeletal muscle myosin light chains are essential for physiological speeds of shortening , 1993, Nature.
[4] P. Anversa,et al. Oxidative Stress–Mediated Cardiac Cell Death Is a Major Determinant of Ventricular Dysfunction and Failure in Dog Dilated Cardiomyopathy , 2001, Circulation research.
[5] R. Kraft,et al. Targeting of the transcription factor Max during apoptosis: phosphorylation-regulated cleavage by caspase-5 at an unusual glutamic acid residue in position P1. , 2001 .
[6] M. Hengartner. The biochemistry of apoptosis , 2000, Nature.
[7] Y. Tsujimoto,et al. A cloning method for caspase substrates that uses the yeast two-hybrid system: cloning of the antiapoptotic gene gelsolin. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[8] R. Frank,et al. Evidence of apoptosis in arrhythmogenic right ventricular dysplasia. , 1996, The New England journal of medicine.
[9] N. Thornberry,et al. A Combinatorial Approach Defines Specificities of Members of the Caspase Family and Granzyme B , 1997, The Journal of Biological Chemistry.
[10] R. Virmani,et al. Apoptosis in heart failure: release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[11] D A Winkelmann,et al. Three-dimensional structure of myosin subfragment-1: a molecular motor. , 1993, Science.
[12] M. Whittaker,et al. Molecular structure of F-actin and location of surface binding sites , 1990, Nature.
[13] P. D. de Tombe,et al. Functional consequences of caspase activation in cardiac myocytes , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[14] K. Trybus,et al. The essential light chain is required for full force production by skeletal muscle myosin. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[15] C A Beltrami,et al. Apoptosis in the failing human heart. , 1997, The New England journal of medicine.
[16] T. Schöneberg,et al. Adenoviral gene transfer of the human V2 vasopressin receptor improves contractile force of rat cardiomyocytes. , 1999, Circulation.
[17] K. Chien. Genomic circuits and the integrative biology of cardiac diseases , 2000, Nature.
[18] E. Peterson,et al. The Drosophila caspase DRONC cleaves following glutamate or aspartate and is regulated by DIAP1, HID, and GRIM. , 2000, The Journal of biological chemistry.
[19] Jean X. Jiang,et al. The Development-associated Cleavage of Lens Connexin 45.6 by Caspase-3-like Protease Is Regulated by Casein Kinase II-mediated Phosphorylation* , 2001, The Journal of Biological Chemistry.
[20] I. Morano. Tuning the human heart molecular motors by myosin light chains , 1999, Journal of Molecular Medicine.
[21] E. Bossy‐Wetzel,et al. Caspase 7 can cleave tumor necrosis factor receptor-I (p60) at a non-consensus motif, in vitro. , 2001, Biochimica et biophysica acta.
[22] M. Seyfarth,et al. Blocking caspase-activated apoptosis improves contractility in failing myocardium. , 2001, Human gene therapy.
[23] G M Cohen,et al. Caspases: the executioners of apoptosis. , 1997, The Biochemical journal.
[24] S. Izumo,et al. Apoptosis: basic mechanisms and implications for cardiovascular disease. , 1998, Circulation research.
[25] J. Mankovich,et al. Substrate Specificities of Caspase Family Proteases* , 1997, The Journal of Biological Chemistry.
[26] M. Böhm,et al. Changes in essential myosin light chain isoform expression provide a molecular basis for isometric force regulation in the failing human heart. , 1997, Journal of molecular and cellular cardiology.
[27] W. Littler,et al. Human atrial and ventricular myosin light-chains subunits in the adult and during development. , 1980, The Biochemical journal.
[28] I. Rayment,et al. Mutations in either the essential or regulatory light chains of myosin are associated with a rare myopathy in human heart and skeletal muscle , 1996, Nature Genetics.