Possible involvement of nitric oxide in long-term potentiation.

[1]  K. Shibuki,et al.  Endogenous nitric oxide release required for long-term synaptic depression in the cerebellum , 1991, Nature.

[2]  S. Snyder,et al.  Localization of nitric oxide synthase indicating a neural role for nitric oxide , 1990, Nature.

[3]  J. Garthwaite,et al.  Nanomolar N(G)-nitroarginine inhibits NMDA-induced cyclic GMP formation in rat cerebellum. , 1990, European journal of pharmacology.

[4]  Richard Graham Knowles,et al.  Kinetic characteristics of nitric oxide synthase from rat brain. , 1990, The Biochemical journal.

[5]  S. Snyder,et al.  Isolation of nitric oxide synthetase, a calmodulin-requiring enzyme. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[6]  J. Garthwaite,et al.  NMDA receptor activation induces nitric oxide synthesis from arginine in rat brain slices. , 1989, European journal of pharmacology.

[7]  J. Garthwaite,et al.  Endothelium-derived relaxing factor release on activation of NMDA receptors suggests role as intercellular messenger in the brain , 1988, Nature.

[8]  M. Feelisch,et al.  Correlation between nitric oxide formation during degradation of organic nitrates and activation of guanylate cyclase. , 1987, European journal of pharmacology.

[9]  S. Moncada,et al.  Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor , 1987, Nature.

[10]  G. Collingridge,et al.  Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus. , 1983, The Journal of physiology.