CD19-independent instruction of murine marginal zone B-cell development by constitutive Notch2 signaling.
暂无分享,去创建一个
Franziska Hampel | S. Ehrenberg | Caroline D Hojer | A. Draeseke | Gabriele Marschall-Schröter | R. Kühn | B. Mack | O. Gires | Christoph J Vahl | M. Schmidt-Supprian | L. Strobl | U. Zimber-Strobl
[1] M. Birnbaum,et al. Akt1 and Akt2 promote peripheral B-cell maturation and survival. , 2010, Blood.
[2] J. Weis,et al. The in vitro derivation of phenotypically mature and diverse B cells from immature spleen and bone marrow precursors , 2010, European journal of immunology.
[3] H. Macdonald,et al. Notch signaling in the immune system. , 2010, Immunity.
[4] S. Pillai,et al. The follicular versus marginal zone B lymphocyte cell fate decision , 2009, Nature Reviews Immunology.
[5] U. Weidle,et al. Notch1, Notch2, and Epstein-Barr virus-encoded nuclear antigen 2 signaling differentially affects proliferation and survival of Epstein-Barr virus-infected B cells. , 2009, Blood.
[6] A. Bandaranayake,et al. Reduced c-myc Expression Levels Limit Follicular Mature B Cell Cycling in Response to TLR Signals1 , 2009, The Journal of Immunology.
[7] S. Egan,et al. Lunatic and manic fringe cooperatively enhance marginal zone B cell precursor competition for delta-like 1 in splenic endothelial niches. , 2009, Immunity.
[8] K. Ando,et al. Expression of Delta-like 1 in the splenic non-hematopoietic cells is essential for marginal zone B cell development. , 2008, Immunology letters.
[9] K. Rajewsky,et al. Constitutive CD40 signaling in B cells selectively activates the noncanonical NF-κB pathway and promotes lymphomagenesis , 2008, The Journal of experimental medicine.
[10] K. Okumura,et al. Delta-like 1 is essential for the maintenance of marginal zone B cells in normal mice but not in autoimmune mice. , 2008, International immunology.
[11] K. Rajewsky,et al. LMP1 signaling can replace CD40 signaling in B cells in vivo and has unique features of inducing class-switch recombination to IgG1. , 2008, Blood.
[12] C. Boboila,et al. The Recirculating B Cell Pool Contains Two Functionally Distinct, Long-Lived, Posttransitional, Follicular B Cell Populations1 , 2007, The Journal of Immunology.
[13] W. Wurst,et al. Conditional brain-specific knockdown of MAPK using Cre/loxP regulated RNA interference , 2007, Nucleic acids research.
[14] W. Pear,et al. Notch activity synergizes with B-cell-receptor and CD40 signaling to enhance B-cell activation. , 2007, Blood.
[15] Wei Li,et al. Identification of CD36 as a new surface marker of marginal zone B cells by transcriptomic analysis. , 2007, Molecular immunology.
[16] M. Reth,et al. Testing gene function early in the B cell lineage in mb1-cre mice , 2006, Proceedings of the National Academy of Sciences.
[17] David Allman,et al. Regulation of peripheral B cell maturation. , 2006, Cellular immunology.
[18] D. Allman,et al. Characterization of marginal zone B cell precursors , 2005, The Journal of experimental medicine.
[19] T. Phan,et al. Altered Migration, Recruitment, and Somatic Hypermutation in the Early Response of Marginal Zone B Cells to T Cell-Dependent Antigen1 , 2005, The Journal of Immunology.
[20] N. Tamaoki,et al. Delta-like 1 is necessary for the generation of marginal zone B cells but not T cells in vivo , 2004, Nature Immunology.
[21] C. S. Swindle,et al. Activated Notch2 Potentiates CD8 Lineage Maturation and Promotes the Selective Development of B1 B Cells , 2003, Molecular and Cellular Biology.
[22] S. Aizawa,et al. Notch2 is preferentially expressed in mature B cells and indispensable for marginal zone B lineage development. , 2003, Immunity.
[23] J. Kearney,et al. Marginal-zone B cells , 2002, Nature Reviews Immunology.
[24] T. Honjo,et al. Notch–RBP-J signaling is involved in cell fate determination of marginal zone B cells , 2002, Nature Immunology.
[25] S. Shinton,et al. Resolution of Three Nonproliferative Immature Splenic B Cell Subsets Reveals Multiple Selection Points During Peripheral B Cell Maturation1 , 2001, The Journal of Immunology.
[26] L. Strobl,et al. EBNA2 and Notch signalling in Epstein-Barr virus mediated immortalization of B lymphocytes. , 2001, Seminars in cancer biology.
[27] R. Hardy,et al. B cell development pathways. , 2001, Annual review of immunology.
[28] J. Kearney,et al. B1 cells: similarities and differences with other B cell subsets. , 2001, Current opinion in immunology.
[29] L. Strobl,et al. Activated Notch1 Can Transiently Substitute for EBNA2 in the Maintenance of Proliferation of LMP1-Expressing Immortalized B Cells , 2001, Journal of Virology.
[30] Y. Kanda,et al. Binding of Delta1, Jagged1, and Jagged2 to Notch2 Rapidly Induces Cleavage, Nuclear Translocation, and Hyperphosphorylation of Notch2 , 2000, Molecular and Cellular Biology.
[31] J. Aster,et al. Notch1 expression in early lymphopoiesis influences B versus T lineage determination. , 1999, Immunity.
[32] P. Sideras,et al. B Cell Development in the Spleen Takes Place in Discrete Steps and Is Determined by the Quality of B Cell Receptor–Derived Signals , 1999, The Journal of experimental medicine.
[33] J. Kearney,et al. IgMhighCD21high lymphocytes enriched in the splenic marginal zone generate effector cells more rapidly than the bulk of follicular B cells. , 1999, Journal of immunology.
[34] P. Cohen,et al. Phosphorylation of the Transcription Factor Forkhead Family Member FKHR by Protein Kinase B* , 1999, The Journal of Biological Chemistry.
[35] Geert J. P. L. Kops,et al. Direct control of the Forkhead transcription factor AFX by protein kinase B , 1999, Nature.
[36] M. Greenberg,et al. Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor , 1999, Cell.
[37] K. Rajewsky,et al. B lymphocyte-specific, Cre-mediated mutagenesis in mice. , 1997, Nucleic acids research.
[38] Klaus Rajewsky,et al. Impairment of T-cell-dependent B-cell responses and B-l cell development in CD19-deficient mice , 1995, Nature.
[39] B. Koller,et al. Abnormal B lymphocyte development, activation, and differentiation in mice that lack or overexpress the CD19 signal transduction molecule. , 1995, Immunity.
[40] D. Tuveson,et al. The CD19 complex of B lymphocytes. Activation of phospholipase C by a protein tyrosine kinase-dependent pathway that can be enhanced by the membrane IgM complex. , 1991, Journal of immunology.
[41] B. Dörken,et al. CD19 monoclonal antibody HD37 inhibits anti-immunoglobulin-induced B cell activation and proliferation. , 1987, Journal of immunology.