Longer and Shorter Forms of Sendai Virus C Proteins Play Different Roles in Modulating the Cellular Antiviral Response
暂无分享,去创建一个
D. Garcin | D. Kolakofsky | J. Curran | Joseph Curran | Daniel Kolakofsky | Dominique Garcin | Masae Itoh | M. Itoh
[1] S. Goodbourn,et al. Sendai Virus and Simian Virus 5 Block Activation of Interferon-Responsive Genes: Importance for Virus Pathogenesis , 1999, Journal of Virology.
[2] D. Garcin,et al. A highly recombinogenic system for the recovery of infectious Sendai paramyxovirus from cDNA: generation of a novel copy‐back nondefective interfering virus. , 1995, The EMBO journal.
[3] D. Kolakofsky,et al. The Sendai virus nonstructural C proteins specifically inhibit viral mRNA synthesis. , 1992, Virology.
[4] R. Lamb,et al. The synthesis of sendai virus polypeptides in infected cells. , 1977, Virology.
[5] B. Jacobs,et al. Site-directed mutagenic analysis of reovirus sigma 3 protein binding to dsRNA. , 1994, Virology.
[6] D. Kolakofsky,et al. TRANSLATIONAL GYMNASTICS ON THE SENDAI VIRUS P/C MRNA , 1998 .
[7] C. Orvell,et al. The effects of monoclonal antibodies on biologic activities of structural proteins of Sendai virus. , 1982, Journal of immunology.
[8] R. Compans,et al. The short Sendai virus leader region controls induction of programmed cell death. , 1998, Virology.
[9] Y. Nagai,et al. Sendai virus C proteins are categorically nonessential gene products but silencing their expression severely impairs viral replication and pathogenesis , 1998, Genes to cells : devoted to molecular & cellular mechanisms.
[10] Aseem Kumar,et al. Defective TNF-α-Induced Apoptosis in STAT1-Null Cells Due to Low Constitutive Levels of Caspases , 1997 .
[11] L. Roux,et al. The rule of six, a basic feature for efficient replication of Sendai virus defective interfering RNA , 1993, Journal of virology.
[12] K. Takeuchi,et al. Knockout of the Sendai virus C gene eliminates the viral ability to prevent the interferon-alpha/beta-mediated responses. , 1999, FEBS letters.
[13] D. Valenzuela,et al. Single amino acid changes that render human IFN‐alpha 2 biologically active on mouse cells. , 1987, The EMBO journal.
[14] G. Stark,et al. How cells respond to interferons. , 1998, Annual review of biochemistry.
[15] D. Kolakofsky,et al. Sendai Virus Y Proteins Are Initiated by a Ribosomal Shunt , 1998, Molecular and Cellular Biology.
[16] S. Kim,et al. The neutralization of type I IFN biologic actions by anti-IFNAR-2 monoclonal antibodies is not entirely due to inhibition of Jak-Stat tyrosine phosphorylation. , 2000, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[17] S. Goodbourn,et al. The beta-interferon promoter responds to priming through multiple independent regulatory elements. , 1994, The Journal of biological chemistry.
[18] S. Goodbourn,et al. STAT1 Is Inactivated by a Caspase* , 1998, The Journal of Biological Chemistry.
[19] Y. Nagai,et al. The paramyxovirus, Sendai virus, V protein encodes a luxury function required for viral pathogenesis , 1997, The EMBO journal.
[20] H. Hotta,et al. Increased Induction of Apoptosis by a Sendai Virus Mutant Is Associated with Attenuation of Mouse Pathogenicity , 1998, Journal of Virology.
[21] Y. Nagai,et al. Versatility of the Accessory C Proteins of Sendai Virus: Contribution to Virus Assembly as an Additional Role , 2000, Journal of Virology.
[22] D. Kolakofsky,et al. An N-terminal domain of the Sendai paramyxovirus P protein acts as a chaperone for the NP protein during the nascent chain assembly step of genome replication , 1995, Journal of virology.
[23] D. Garcin,et al. Normal cellular replication of Sendai virus without the trans-frame, nonstructural V protein. , 1997, Virology.
[24] H. Hotta,et al. Isolation of an avirulent mutant of Sendai virus with two amino acid mutations from a highly virulent field strain through adaptation to LLC-MK2 cells. , 1997, The Journal of general virology.
[25] D. Kolakofsky,et al. Inhibition of Sendai virus genome replication due to promoter-increased selectivity: a possible role for the accessory C proteins , 1997, Journal of virology.
[26] B. Jacobs,et al. Site-Directed Mutagenic Analysis of Reovirus σ3 Protein Binding to dsRNA , 1994 .
[27] D. Kolakofsky,et al. The Various Sendai Virus C Proteins Are Not Functionally Equivalent and Exert both Positive and Negative Effects on Viral RNA Accumulation during the Course of Infection , 1998, Journal of Virology.
[28] D. Garcin,et al. Sendai Virus C Proteins Counteract the Interferon-Mediated Induction of an Antiviral State , 1999, Journal of Virology.
[29] K. Takeuchi,et al. Sendai Virus Blocks Alpha Interferon Signaling to Signal Transducers and Activators of Transcription , 2000, Journal of Virology.
[30] G. Stark,et al. How Stat1 mediates constitutive gene expression: a complex of unphosphorylated Stat1 and IRF1 supports transcription of the LMP2 gene , 2000, The EMBO journal.
[31] A. Aguzzi,et al. Deficient signaling in mice devoid of double‐stranded RNA‐dependent protein kinase. , 1995, The EMBO journal.
[32] D. Garcin,et al. Partial characterization of a Sendai virus replication promoter and the rule of six. , 1996, Virology.
[33] D. Garcin,et al. A point mutation in the Sendai virus accessory C proteins attenuates virulence for mice, but not virus growth in cell culture. , 1997, Virology.
[34] M. Malim,et al. Evidence for a newly discovered cellular anti-HIV-1 phenotype , 1998, Nature Medicine.
[35] C. Schindler. STATs as activators of apoptosis. , 1998, Trends in cell biology.
[36] S. Goodbourn,et al. The V Protein of Simian Virus 5 Inhibits Interferon Signalling by Targeting STAT1 for Proteasome-Mediated Degradation , 1999, Journal of Virology.
[37] D. Garcin,et al. Sendai Virus C Proteins Must Interact Directly with Cellular Components To Interfere with Interferon Action , 2000, Journal of Virology.
[38] K. Takeuchi,et al. Knockout of the Sendai virus C gene eliminates the viral ability to prevent the interferon‐α/β‐mediated responses , 1999 .
[39] Sheila M. Thomas,et al. Two mRNAs that differ by two nontemplated nucleotides encode the amino coterminal proteins P and V of the paramyxovirus SV5 , 1988, Cell.
[40] D. Kolakofsky,et al. An acidic activation-like domain of the Sendai virus P protein is required for RNA synthesis and encapsidation. , 1994, Virology.
[41] D. Kolakofsky,et al. A stuttering model for paramyxovirus P mRNA editing. , 1990, The EMBO journal.
[42] D. Garcin,et al. The Sendai paramyxovirus accessory C proteins inhibit viral genome amplification in a promoter-specific fashion , 1996, Journal of virology.
[43] J. Curran. Reexamination of the Sendai virus P protein domains required for RNA synthesis: a possible supplemental role for the P protein. , 1996, Virology.
[44] R. Flavell,et al. Activation of the STAT signaling pathway can cause expression of caspase 1 and apoptosis , 1997, Molecular and cellular biology.
[45] D. Trono. When accessories turn out to be essential , 1998, Nature Medicine.