Effect of Hydrogen Sulfide on Kidney Injury in Rat Model of Crush Syndrome.

[1]  Vijay Viswanathan,et al.  Increased levels of circulating (TNF-α) is associated with (-308G/A) promoter polymorphism of TNF-α gene in Diabetic Nephropathy. , 2018, International journal of biological macromolecules.

[2]  Yanzhang Li,et al.  Hydrogen sulfide ameliorates chronic renal failure in rats by inhibiting apoptosis and inflammation through ROS/MAPK and NF-κB signaling pathways , 2017, Scientific Reports.

[3]  T. Simić,et al.  Novel Biomarkers of Heart Failure. , 2017, Advances in clinical chemistry.

[4]  Y. Inoue,et al.  Early Therapeutic Intervention for Crush Syndrome: Characterization of Intramuscular Administration of Dexamethasone by Pharmacokinetic and Biochemical Parameters in Rats. , 2016, Biological and Pharmaceutical Bulletin.

[5]  Hui Zhou,et al.  Minocycline Attenuates Kidney Injury in a Rat Model of Streptozotocin-Induced Diabetic Nephropathy. , 2016, Biological & pharmaceutical bulletin.

[6]  Shingo Nakamura,et al.  Improved angiogenesis and healing in crush syndrome by fibroblast growth factor-2-containing low-molecular-weight heparin (Fragmin)/protamine nanoparticles. , 2015, Journal of Surgical Research.

[7]  Yanzhang Li,et al.  Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury , 2015, Oxidative medicine and cellular longevity.

[8]  A. Şener,et al.  Hydrogen sulphide and the kidney: important roles in renal physiology and pathogenesis and treatment of kidney injury and disease. , 2015, Nitric oxide : biology and chemistry.

[9]  I. Velasco,et al.  Plasma cytokine expression after lower-limb compression in rats☆ , 2014, Revista brasileira de ortopedia.

[10]  Irineu Tadeu Velasco,et al.  Expressão de citoquinas plasmáticas após compressão de membros inferiores de ratos , 2015 .

[11]  H. Kimura.  The physiological role of hydrogen sulfide and beyond. , 2014, Nitric oxide : biology and chemistry.

[12]  Xiaoping Zhou,et al.  Hydrogen Sulfide Alleviates Diabetic Nephropathy in a Streptozotocin-induced Diabetic Rat Model , 2014, The Journal of Biological Chemistry.

[13]  A. Otunctemur,et al.  Protective effect of hydrogen sulfide on gentamicin-induced renal injury , 2014, Renal failure.

[14]  T. Maehara,et al.  Improved survival rate by temperature control at compression sites in rat model of crush syndrome. , 2014, The Journal of surgical research.

[15]  R. Nishikata,et al.  Oxidative stress may be involved in distant organ failure in tourniquet shock model mice. , 2014, Legal medicine.

[16]  B. A. Silva,et al.  Leukocyte infiltration in lung, muscle, and liver after limb compression in rats. , 2013, Pathophysiology : the official journal of the International Society for Pathophysiology.

[17]  Jie Zhao,et al.  Exogenous hydrogen sulfide protects against global cerebral ischemia/reperfusion injury via its anti-oxidative, anti-inflammatory and anti-apoptotic effects in rats , 2013, Brain Research.

[18]  P. Vallés,et al.  Apoptosis modulated by oxidative stress and inflammation during obstructive nephropathy. , 2012, Inflammation & allergy drug targets.

[19]  J. Wallace,et al.  Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury. , 2012, Antioxidants & redox signaling.

[20]  Y. Morimoto,et al.  Nitrite reduces ischemia/reperfusion-induced muscle damage and improves survival rates in rat crush injury model , 2012, The journal of trauma and acute care surgery.

[21]  J. Kellum,et al.  Application of the RIFLE criteria in patients with crush-related acute kidney injury after mass disasters. , 2011, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[22]  W. Huo,et al.  Kidney injury molecule-1 (KIM-1): a novel kidney-specific injury molecule playing potential double-edged functions in kidney injury. , 2010, Transplantation reviews.

[23]  Y. Goto,et al.  Hydrogen sulfide increases glutathione production and suppresses oxidative stress in mitochondria. , 2010, Antioxidants & redox signaling.

[24]  C. Edelstein,et al.  Mediators of Inflammation in Acute Kidney Injury , 2010, Mediators of inflammation.

[25]  D. Zorov,et al.  Myoglobin causes oxidative stress, increase of NO production and dysfunction of kidney's mitochondria. , 2009, Biochimica et biophysica acta.

[26]  Pin-Lan Li,et al.  Production and Actions of Hydrogen Sulfide, a Novel Gaseous Bioactive Substance, in the Kidneys , 2009, Journal of Pharmacology and Experimental Therapeutics.

[27]  O. Erel,et al.  A new automated colorimetric method for measuring total oxidant status. , 2005, Clinical biochemistry.

[28]  B. Geng,et al.  Endogenous hydrogen sulfide regulation of myocardial injury induced by isoproterenol. , 2004, Biochemical and biophysical research communications.

[29]  O. Erel,et al.  A novel automated method to measure total antioxidant response against potent free radical reactions. , 2004, Clinical biochemistry.

[30]  B. Aggarwal,et al.  TNF-Induced Signaling in Apoptosis , 1999, Journal of Clinical Immunology.

[31]  E. Bottinger,et al.  TGF-β signaling in renal disease , 2002 .

[32]  R. Vanholder,et al.  Acute renal failure related to the crush syndrome: towards an era of seismo-nephrology? , 2000, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[33]  T. Shimazu,et al.  Analysis of 372 patients with Crush syndrome caused by the Hanshin-Awaji earthquake. , 1997, The Journal of trauma.

[34]  O. Better,et al.  History of the crush syndrome: from the earthquakes of Messina, Sicily 1909 to Spitak, Armenia 1988. , 1997, American journal of nephrology.

[35]  O. Better.  The crush syndrome revisited (1940-1990). , 1990, Nephron.

[36]  P. Jablonski,et al.  AN EXPERIMENTAL MODEL FOR ASSESSMENT OF RENAL RECOVERY FROM WARM ISCHEMIA , 1983, Transplantation.

[37]  E. Bywaters,et al.  Crush Injuries with Impairment of Renal Function , 1941, British medical journal.