Positive and negative regulation of NF-kappaB by COX-2: roles of different prostaglandins.
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[1] M. Kay Washington,et al. Prostaglandin E2 Increases Growth and Motility of Colorectal Carcinoma Cells* , 2001, The Journal of Biological Chemistry.
[2] J. Masferrer,et al. COX‐2 is expressed in human pulmonary, colonic, and mammary tumors , 2000, Cancer.
[3] C. Y. Wang,et al. NF-kappaB-induced loss of MyoD messenger RNA: possible role in muscle decay and cachexia. , 2000, Science.
[4] C. Glass,et al. 15-deoxy-delta 12,14-prostaglandin J2 inhibits multiple steps in the NF-kappa B signaling pathway. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[5] M. Mayo,et al. Akt Suppresses Apoptosis by Stimulating the Transactivation Potential of the RelA/p65 Subunit of NF-κB , 2000, Molecular and Cellular Biology.
[6] G. Natoli,et al. Anti-inflammatory cyclopentenone prostaglandins are direct inhibitors of IκB kinase , 2000, Nature.
[7] R. DuBois,et al. The role of cyclooxygenases in inflammation, cancer, and development , 1999, Oncogene.
[8] G. Haegeman,et al. The Nuclear Factor-κB Engages CBP/p300 and Histone Acetyltransferase Activity for Transcriptional Activation of the Interleukin-6 Gene Promoter* , 1999, The Journal of Biological Chemistry.
[9] S. Narumiya,et al. Role of the prostaglandin E receptor subtype EP1 in colon carcinogenesis. , 1999, Cancer research.
[10] C. Glass,et al. Transcriptional Activation by NF-κB Requires Multiple Coactivators , 1999, Molecular and Cellular Biology.
[11] D. Gilroy,et al. Inducible cyclooxygenase may have anti-inflammatory properties , 1999, Nature Medicine.
[12] J. Cusack,et al. Control of inducible chemoresistance: Enhanced anti-tumor therapy through increased apoptosis by inhibition of NF-κB , 1999, Nature Medicine.
[13] B. Williams,et al. Involvement of Double-stranded RNA-activated Protein Kinase in the Synergistic Activation of Nuclear Factor-κB by Tumor Necrosis Factor-α and γ-Interferon in Preneuronal Cells* , 1999, The Journal of Biological Chemistry.
[14] S. Narumiya,et al. Prostanoid receptors: structures, properties, and functions. , 1999, Physiological reviews.
[15] M. Neurath,et al. Role of NF-κB in immune and inflammatory responses in the gut , 1998 .
[16] Han,et al. Specific NF‐κB blockade selectively inhibits tumour necrosis factor‐α‐induced COX‐2 but not constitutive COX‐1 gene expression in HT‐29 cells , 1998, Immunology.
[17] A. Baldwin,et al. NF-kappaB activation provides the potential link between inflammation and hyperplasia in the arthritic joint. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[18] A. Baldwin,et al. Activation of Nuclear Factor-κB-dependent Transcription by Tumor Necrosis Factor-α Is Mediated through Phosphorylation of RelA/p65 on Serine 529* , 1998, The Journal of Biological Chemistry.
[19] M. Olivier,et al. Prostaglandin E2 Up-regulates HIV-1 Long Terminal Repeat-driven Gene Activity in T Cells via NF-κB-dependent and -Independent Signaling Pathways* , 1998, The Journal of Biological Chemistry.
[20] C. Y. Wang,et al. NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation. , 1998, Science.
[21] P. Lipsky,et al. Cyclooxygenase in biology and disease , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[22] H. Kamitani,et al. Expression of 15-Lipoxygenase by Human Colorectal Carcinoma Caco-2 Cells during Apoptosis and Cell Differentiation* , 1998, The Journal of Biological Chemistry.
[23] J. W. Woods,et al. Subcellular Localization of Prostaglandin Endoperoxide H Synthases-1 and -2 by Immunoelectron Microscopy* , 1998, The Journal of Biological Chemistry.
[24] S. Ghosh,et al. Phosphorylation of NF-kappa B p65 by PKA stimulates transcriptional activity by promoting a novel bivalent interaction with the coactivator CBP/p300. , 1998, Molecular cell.
[25] J. Morrow,et al. Modulation of apoptosis and Bcl-2 expression by prostaglandin E2 in human colon cancer cells. , 1998, Cancer research.
[26] G. Krishnaswamy,et al. Prostaglandin H synthase expression is variable in human colorectal adenocarcinoma cell lines. , 1997, Experimental cell research.
[27] C. Der,et al. Oncogenic Ha-Ras-induced Signaling Activates NF-κB Transcriptional Activity, Which Is Required for Cellular Transformation* , 1997, The Journal of Biological Chemistry.
[28] S. Narumiya,et al. Ligand binding specificities of the eight types and subtypes of the mouse prostanoid receptors expressed in Chinese hamster ovary cells , 1997, British journal of pharmacology.
[29] O. Nielsen,et al. Expression of cyclooxygenase-2 mRNA in active inflammatory bowel disease. , 1997, The American journal of gastroenterology.
[30] H. Erdjument-Bromage,et al. The Transcriptional Activity of NF-κB Is Regulated by the IκB-Associated PKAc Subunit through a Cyclic AMP–Independent Mechanism , 1997, Cell.
[31] M. Karin,et al. Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases. , 1997, The New England journal of medicine.
[32] R. DuBois,et al. Cyclooxygenase-2 expression in human colon cancer cells increases metastatic potential. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[33] M. Mayo,et al. Involvement of Egr-1/RelA Synergy in Distinguishing T Cell Activation from Tumor Necrosis Factor-α–induced NF-κB1 Transcription , 1997, The Journal of experimental medicine.
[34] M. Runge,et al. Hypoxia Induces Cyclooxygenase-2 via the NF-κB p65 Transcription Factor in Human Vascular Endothelial Cells* , 1997, The Journal of Biological Chemistry.
[35] Bruno C. Hancock,et al. Suppression of Intestinal Polyposis in Apc Δ716 Knockout Mice by Inhibition of Cyclooxygenase 2 (COX-2) , 1996, Cell.
[36] C. McCall,et al. Protein-tyrosine Kinase Activation Is Required for Lipopolysaccharide Induction of Interleukin 1β and NFκB Activation, but Not NFκB Nuclear Translocation* , 1996, The Journal of Biological Chemistry.
[37] M. Baggiolini. Activation and recruitment of neutrophil leukocytes , 1995, Clinical and experimental immunology.
[38] M. Schmitz,et al. Transactivation domain 2 (TA2) of p65 NF-kappa B. Similarity to TA1 and phorbol ester-stimulated activity and phosphorylation in intact cells. , 1995, The Journal of biological chemistry.
[39] J. Otto,et al. Different Intracellular Locations for Prostaglandin Endoperoxide H Synthase-1 and −2 (*) , 1995, The Journal of Biological Chemistry.
[40] M. Baggiolini,et al. Interleukin-8 and the chemokine family. , 1995, International journal of immunopharmacology.
[41] R. Coffey,et al. Up-regulation of cyclooxygenase 2 gene expression in human colorectal adenomas and adenocarcinomas. , 1994, Gastroenterology.
[42] P. Baeuerle,et al. The p65 subunit is responsible for the strong transcription activating potential of NF‐kappa B. , 1991, The EMBO journal.
[43] K. Matsushima,et al. Neutrophil chemotactic factor produced by lipopolysaccharide (LPS)-stimulated human blood mononuclear leukocytes: partial characterization and separation from interleukin 1 (IL 1). , 1987, Journal of immunology.
[44] R. Garavito,et al. Cyclooxygenases: structural, cellular, and molecular biology. , 2000, Annual review of biochemistry.
[45] J. Vane,et al. Cyclooxygenases 1 and 2. , 1998, Annual review of pharmacology and toxicology.
[46] M J May,et al. NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses. , 1998, Annual review of immunology.
[47] P. Baeuerle,et al. NF-kappa B activation in human knee-joint synovial tissue during the early stage of joint inflammation. , 1997, Biochemical Society transactions.
[48] A. Baldwin,et al. THE NF-κB AND IκB PROTEINS: New Discoveries and Insights , 1996 .
[49] K. Wu,et al. Role of endothelium in thrombosis and hemostasis. , 1996, Annual review of medicine.
[50] A. Ford-hutchinson,et al. The role of arachidonic acid oxygenation products in pain and inflammation. , 1984, Annual review of immunology.