Activation of Invariant NK T Cells in Periodontitis Lesions
暂无分享,去创建一个
Justus M. Kebschull | P. Papapanou | P. Hoffmann | S. Perner | P. Brossart | I. Schmidt-Wolf | B. Krämer | M. Kebschull | S. Jepsen | Michael D. Nowak | Manuela Haupt | M. Haupt
[1] J. Strominger,et al. CD1d-Restricted NKT Cells Express a Chemokine Receptor Profile Indicative of Th1-Type Inflammatory Homing Cells 1 , 2003, The Journal of Immunology.
[2] R. Steinman,et al. Prolonged IFN-γ–producing NKT response induced with α-galactosylceramide–loaded DCs , 2002, Nature Immunology.
[3] M. Rubin,et al. Aberrant Cytoplasmic Expression of p63 and Prostate Cancer Mortality , 2009, Cancer Epidemiology Biomarkers & Prevention.
[4] E. Nemoto,et al. Porphyromonas gingivalis fimbriae induce unique dendritic cell subsets via Toll-like receptor 2. , 2009, Journal of periodontal research.
[5] S. Sansano,et al. Peroxisome-derived lipids are self antigens that stimulate invariant natural killer T cells in the thymus , 2012, Nature Immunology.
[6] A. Cummins,et al. Deficiency of 6B11+ Invariant NK T-Cells in Celiac Disease , 2008, Digestive Diseases and Sciences.
[7] P. Papapanou,et al. Epidemiologic patterns of chronic and aggressive periodontitis. , 2010, Periodontology 2000.
[8] L. Vitale,et al. Immune suppression induced by Actinobacillus actinomycetemcomitans: effects on immunoglobulin production by human B cells , 1990, Infection and immunity.
[9] D. Giordano,et al. Effects of oral commensal and pathogenic bacteria on human dendritic cells. , 2009, Oral microbiology and immunology.
[10] P. Askenase,et al. TLR-Dependent IL-4 Production by Invariant Vα14+Jα18+ NKT Cells to Initiate Contact Sensitivity In Vivo1 , 2005, The Journal of Immunology.
[11] B. Beutler,et al. Exogenous and endogenous glycolipid antigens activate NKT cells during microbial infections , 2005, Nature.
[12] S. Akira,et al. Pattern Recognition Receptors and Inflammation , 2010, Cell.
[13] Mitchell Kronenberg,et al. Toward an understanding of NKT cell biology: progress and paradoxes. , 2005, Annual review of immunology.
[14] R. Darveau,et al. Periodontitis: a polymicrobial disruption of host homeostasis , 2010, Nature Reviews Microbiology.
[15] I. Saiki,et al. Differential Regulation of Th1 and Th2 Functions of NKT Cells by CD28 and CD40 Costimulatory Pathways1 , 2001, The Journal of Immunology.
[16] B. Dye,et al. Overview and quality assurance for the oral health component of the National Health and Nutrition Examination Survey (NHANES), 2005-08. , 2011, Journal of public health dentistry.
[17] D. Ho,et al. Recognition of bacterial glycosphingolipids by natural killer T cells , 2005, Nature.
[18] R. Brutkiewicz,et al. Cell wall glycosphingolipids of Sphingomonas paucimobilis are CD1d‐specific ligands for NKT cells , 2005, European journal of immunology.
[19] B. Dye,et al. Overview and quality assurance for the oral health component of the National Health and Nutrition Examination Survey (NHANES), 2003-04. , 2008, Journal of public health dentistry.
[20] M. Sanda,et al. Defective NKT Cell Activation by CD1d+ TRAMP Prostate Tumor Cells Is Corrected by Interleukin-12 with alpha-Galactosylceramide , 2010, PloS one.
[21] D. Graves,et al. Cytokine Profiling of Macrophages Exposed to Porphyromonas gingivalis, Its Lipopolysaccharide, or Its FimA Protein , 2005, Infection and Immunity.
[22] M. Sanz,et al. Variability in the response of human dendritic cells stimulated with Porphyromonas gingivalis or Aggregatibacter actinomycetemcomitans. , 2008, Journal of periodontal research.
[23] A. Gobl,et al. Interferon‐α but not ‐β Genes Require De Novo Protein Synthesis for Efficient Expression in Human Monocytes , 1992 .
[24] L. Bry,et al. Mechanism of CD1d-restricted natural killer T cell activation during microbial infection , 2003, Nature Immunology.
[25] K. Okuda,et al. Purification and characterization of a thiol-protease from Bacteroides gingivalis strain 381. , 1987, Oral microbiology and immunology.
[26] Sam Forster,et al. INTERFEROME: the database of interferon regulated genes , 2008, Nucleic Acids Res..
[27] P. Papapanou,et al. “Gum Bug, Leave My Heart Alone!”—Epidemiologic and Mechanistic Evidence Linking Periodontal Infections and Atherosclerosis , 2010, Journal of dental research.
[28] G. Armitage,et al. Comparison of the clinical features of chronic and aggressive periodontitis. , 2010, Periodontology 2000.
[29] S. Kaufmann,et al. Lysosomal alpha-galactosidase controls the generation of self lipid antigens for natural killer T cells. , 2010, Immunity.
[30] P. Askenase,et al. TLR-dependent IL-4 production by invariant Valpha14+Jalpha18+ NKT cells to initiate contact sensitivity in vivo. , 2005, Journal of immunology.
[31] R. Darveau. The oral microbial consortium's interaction with the periodontal innate defense system. , 2009, DNA and cell biology.
[32] G. Garlet. Destructive and Protective Roles of Cytokines in Periodontitis: A Re-appraisal from Host Defense and Tissue Destruction Viewpoints , 2010, Journal of dental research.
[33] G. Besra,et al. Invariant natural killer T cells recognize lipid self-antigen induced by microbial danger signals , 2011, Nature Immunology.
[34] D. Umetsu,et al. Vaccine-Induced Antibody Isotypes Are Skewed by Impaired CD4 T Cell and Invariant NKT Cell Effector Responses in MyD88-Deficient Mice1 , 2009, The Journal of Immunology.
[35] G. Seymour,et al. Immunological differences and similarities between chronic periodontitis and aggressive periodontitis. , 2010, Periodontology 2000.
[36] G. Hajishengallis,et al. Pathogen induction of CXCR4/TLR2 cross-talk impairs host defense function , 2008, Proceedings of the National Academy of Sciences.
[37] P. Eke,et al. Overview and quality assurance for the oral health component of the National Health and Nutrition Examination Survey (NHANES), 2009-2010: Oral health overview: NHANES 2009-2010 , 2011 .
[38] H. Fujiwara,et al. A novel function of Valpha14+CD4+NKT cells: stimulation of IL-12 production by antigen-presenting cells in the innate immune system. , 1999, Journal of immunology.
[39] C. Prussin,et al. TCR V alpha 24 and V beta 11 coexpression defines a human NK1 T cell analog containing a unique Th0 subpopulation. , 1997, Journal of immunology.
[40] F. Baron,et al. Aggregatibacter actinomycetemcomitans as indicator for aggressive periodontitis by two analysing strategies. , 2007, Journal of clinical periodontology.
[41] H. Fujiwara,et al. A Novel Function of Vα14+CD4+NKT Cells: Stimulation of IL-12 Production by Antigen-Presenting Cells in the Innate Immune System , 1999, The Journal of Immunology.
[42] G. Armitage,et al. Development of a classification system for periodontal diseases and conditions. , 1999, Annals of periodontology.
[43] R. Steinman,et al. Prolonged IFN-gamma-producing NKT response induced with alpha-galactosylceramide-loaded DCs. , 2002, Nature immunology.
[44] T. Giese,et al. CpG ODN enhance antigen‐specific NKT cell activation via plasmacytoid dendritic cells , 2005, European journal of immunology.
[45] A. Ohta,et al. The A2aR adenosine receptor controls cytokine production in iNKT cells , 2010, European journal of immunology.
[46] A. Gobl,et al. Interferon-alpha but not -beta genes require de novo protein synthesis for efficient expression in human monocytes. , 1992, Scandinavian journal of immunology.
[47] Q. Wang,et al. Distribution of Porphyromonas gingivalis fimA genotypes in chronic apical periodontitis associated with symptoms. , 2010, Journal of endodontics.
[48] I. Wilson,et al. Natural killer T cells recognize diacylglycerol antigens from pathogenic bacteria , 2006, Nature Immunology.
[49] R. Steinman,et al. Activation of Natural Killer T Cells by -Galactosylceramide Rapidly Induces the Full Maturation of Dendritic Cells In Vivo and Thereby Acts as an Adjuvant for Combined CD4 and CD8 T Cell Immunity to a Coadministered Protein , 2003 .
[50] G. Seymour,et al. Histopathological features of chronic and aggressive periodontitis. , 2010, Periodontology 2000.
[51] R. Page,et al. The host response to the microbial challenge in periodontitis: assembling the players. , 1997, Periodontology 2000.
[52] Anneliese O. Speak,et al. Activation of invariant NKT cells by toll-like receptor 9-stimulated dendritic cells requires type I interferon and charged glycosphingolipids. , 2007, Immunity.
[53] S. Socransky,et al. Microbial etiological agents of destructive periodontal diseases. , 1994, Periodontology 2000.