Reactive oxygen species (ROS) and reactive nitrogen species (RNS) generation by silica in inflammation and fibrosis.
暂无分享,去创建一个
[1] P. Baeuerle,et al. Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF‐kappa B transcription factor and HIV‐1. , 1991, The EMBO journal.
[2] W. Rom,et al. Crucial role of interleukin-1beta and nitric oxide synthase in silica-induced inflammation and apoptosis in mice. , 2002, American journal of respiratory and critical care medicine.
[3] Vicki Stone,et al. Surface modification of quartz inhibits toxicity, particle uptake, and oxidative DNA damage in human lung epithelial cells. , 2002, Chemical research in toxicology.
[4] V. Souvannavong,et al. Silica induces apoptosis in macrophages and the release of interleukin‐1 α and interleukin‐1β , 1993, Journal of leukocyte biology.
[5] R. Linden,et al. Apoptosis underlies immunopathogenic mechanisms in acute silicosis. , 2002, American journal of respiratory cell and molecular biology.
[6] Xianglin Shi,et al. Reactivity of Free Radicals on Hydroxylated Quartz Surface and Its Implications for Pathogenicity of Silicas: Experimental and Quantum Mechanical Study , 2001 .
[7] D. Costa,et al. Surface oxygen radicals originating via redox reactions during the mechanical activation of crystalline SiO2 in hydrogen peroxide , 1990 .
[8] Z. Dong,et al. Freshly Fractured Crystalline Silica Induces Activator Protein-1 Activation through ERKs and p38 MAPK* , 1999, The Journal of Biological Chemistry.
[9] J. Bruch,et al. Studies of quartz cytotoxicity on peritoneal macrophages of guinea pigs pretreated with polyvinylpyridine N-oxide. , 1975, Environmental research.
[10] Xianglin Shi,et al. Generation of free radicals from freshly fractured silica dust. Potential role in acute silica-induced lung injury. , 1988, The American review of respiratory disease.
[11] Luchuan Liang,et al. Oxidative Stress Activates Metal-responsive Transcription Factor-1 Binding Activity , 1996, The Journal of Biological Chemistry.
[12] R. Tiozzo,et al. Pure-silica zeolites (Porosils) as model solids for the evaluation of the physicochemical features determining silica toxicity to macrophages. , 2000, Chemical research in toxicology.
[13] J. Carter,et al. Mitochondrial-derived oxidants and quartz activation of chemokine gene expression. , 2001, Advances in experimental medicine and biology.
[14] R. Hamilton,et al. Cell surface regulation of silica-induced apoptosis by the SR-A scavenger receptor in a murine lung macrophage cell line (MH-S). , 2001, Toxicology and applied pharmacology.
[15] N. Blough,et al. Reaction of superoxide with nitric oxide to form peroxonitrite in alkaline aqueous solution , 1985 .
[16] Y. M. Park,et al. Silica-induced apoptosis in vitro and in vivo. , 1999, Toxicology letters.
[17] P. Borm,et al. The quartz hazard: a variable entity. , 1998, The Annals of occupational hygiene.
[18] D. Costa,et al. A novel type of active site at the surface of crystalline SiO2 (a-quartz) and its possible impact on pathogenicity , 1991 .
[19] P. Cerutti,et al. The roles of hydrogen peroxide and superoxide as messengers in the activation of transcription factor NF-kappa B. , 1995, Chemistry & biology.
[20] H. Castro-Faria-Neto,et al. Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages , 2000, Nature.
[21] J. Bravman,et al. Silica-induced activation of c-Jun-NH2-terminal amino kinases, protracted expression of the activator protein-1 proto-oncogene, fra-1, and S-phase alterations are mediated via oxidative stress. , 2001, Cancer research.
[22] Vincent Castranova,et al. Silica and Silica-Induced Lung Diseases , 1995 .
[23] B. Fubini. Surface chemistry and quartz hazard , 1998 .
[24] L. Kobzik,et al. Environmental particulate-mediated cytokine production in lung epithelial cells (A549): role of preexisting inflammation and oxidant stress. , 1998, Journal of toxicology and environmental health. Part A.
[25] R. Tiozzo,et al. Relationship between surface properties and cellular responses to crystalline silica: studies with heat-treated cristobalite. , 1999, Chemical research in toxicology.
[26] B. Fubini,et al. Free radical generation in the toxicity of inhaled mineral particles: the role of iron speciation at the surface of asbestos and silica , 2001, Redox report : communications in free radical research.
[27] V. Castranova,et al. Freshly fractured quartz inhalation leads to enhanced lung injury and inflammation. Potential role of free radicals. , 1995, American journal of respiratory and critical care medicine.
[28] E. Papirer. Adsorption on Silica Surfaces , 2000 .
[29] D. Landsittel,et al. Enhanced nitric oxide and reactive oxygen species production and damage after inhalation of silica. , 2002, American journal of physiology. Lung cellular and molecular physiology.
[30] R. Linden,et al. FAS Ligand Triggers Pulmonary Silicosis , 2001, The Journal of experimental medicine.
[31] B. Fubini,et al. Cytotoxic and transforming effects of silica particles with different surface properties in Syrian hamster embryo (SHE) cells. , 2000, Toxicology in vitro : an international journal published in association with BIBRA.
[32] F. Chen,et al. Essential role of NF-kappa B activation in silica-induced inflammatory mediator production in macrophages. , 1995, Biochemical and biophysical research communications.
[33] B. Mossman,et al. Activation of NF-kappaB-dependent gene expression by silica in lungs of luciferase reporter mice. , 2002, American journal of physiology. Lung cellular and molecular physiology.
[34] R. Iyer,et al. Silica-induced apoptosis mediated via scavenger receptor in human alveolar macrophages. , 1996, Toxicology and applied pharmacology.
[35] V. Castranova,et al. Silica-Induced Pulmonary Inflammation in Rats: Activation of NF-κB and Its Suppression by Dexamethasone , 1998 .
[36] X. Shi,et al. Enhanced generation of free radicals from phagocytes induced by mineral dusts. , 1992, American journal of respiratory cell and molecular biology.
[37] Xianglin Shi,et al. Generation of Reactive Oxygen Species by Quartz Particles and Its Implication for Cellular Damage , 1995 .
[38] A. Ghio,et al. Role of surface complexed iron in oxidant generation and lung inflammation induced by silicates. , 1992, The American journal of physiology.
[39] Z. Dong,et al. The role of hydroxyl radical as a messenger in the activation of nuclear transcription factor NF-κB , 1999, Molecular and Cellular Biochemistry.
[40] H. Pahl,et al. Regulation of the transcription factors NF-kappa B and AP-1 by redox changes. , 1994, Chemico-biological interactions.
[41] B. Fubini,et al. Modulation of silica pathogenicity by surface processes , 1999 .
[42] W. MacNee,et al. Aluminium lactate treatment of DQ12 quartz inhibits its ability to cause inflammation, chemokine expression, and nuclear factor-kappaB activation. , 2001, Toxicology and applied pharmacology.
[43] Roel P F Schins,et al. DNA damage in lung epithelial cells isolated from rats exposed to quartz: role of surface reactivity and neutrophilic inflammation. , 2002, Carcinogenesis.
[44] F. Chen,et al. Dependence and reversal of nitric oxide production on NF-kappa B in silica and lipopolysaccharide-induced macrophages. , 1995, Biochemical and biophysical research communications.
[45] Zhuo Zhang,et al. Critical role of GSH in silica-induced oxidative stress, cytotoxicity, and genotoxicity in alveolar macrophages. , 1999, American journal of physiology. Lung cellular and molecular physiology.
[46] K. Schulze-Osthoff,et al. Role of Reactive Oxygen Intermediates in Activation-induced CD95 (APO-1/Fas) Ligand Expression* , 1998, The Journal of Biological Chemistry.
[47] J. Finkelstein,et al. Silica-induced chemokine expression in alveolar type II cells is mediated by TNF-α-induced oxidant stress. , 1999, American journal of physiology. Lung cellular and molecular physiology.
[48] P. Borm,et al. Expression of antioxidant enzymes in rat lungs after inhalation of asbestos or silica. , 1992, The Journal of biological chemistry.
[49] D. Taatjes,et al. Increased manganese superoxide dismutase protein in type II epithelial cells of rat lungs after inhalation of crocidolite asbestos or cristobalite silica. , 1992, The American journal of pathology.
[50] Xianglin Shi,et al. Protective Effects of Silanol Group Binding Agents on Quartz Toxicity to Rat Lung Alveolar Cells , 1995 .
[51] A. Legrand,et al. The surface properties of silicas , 1998 .
[52] B. Mossman,et al. Use of transgenic luciferase reporter mice to determine activation of transcription factors and gene expression by fibrogenic particles. , 2001, Chest.
[53] C. Ong,et al. Reactive oxygen species and caspase activation mediate silica-induced apoptosis in alveolar macrophages. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[54] A Teass,et al. Augmentation of pulmonary reactions to quartz inhalation by trace amounts of iron-containing particles. , 1997, Environmental health perspectives.
[55] B. Fubini,et al. The Role of Mechanochemistry in the Pulmonary Toxicity Caused by Particulate Minerals , 2000 .
[56] P. Carré,et al. Reactive oxygen intermediates as regulators of TNF-alpha production in rat lung inflammation induced by silica. , 1996, Journal of immunology.
[57] J. Leigh,et al. Silica-induced apoptosis in alveolar and granulomatous cells in vivo. , 1997 .
[58] B. Fubini,et al. Possible role of ascorbic acid in the oxidative damage induced by inhaled crystalline silica particles. , 2000, Chemical Research in Toxicology.
[59] M Volante,et al. Chemical functionalities at the silica surface determining its reactivity when inhaled. Formation and reactivity of surface radicals. , 1990, Toxicology and industrial health.
[60] V. Castranova,et al. Intratracheal instillation of silica up-regulates inducible nitric oxide synthase gene expression and increases nitric oxide production in alveolar macrophages and neutrophils. , 1994, American journal of respiratory cell and molecular biology.
[61] Å. Swensson,et al. Influence of mode of disintegration on the fibrogenetic power of quartz particles. , 1971, La Medicina del lavoro.
[62] B. Fubini,et al. The surface chemistry of crushed quartz dust in relation to its pathogenicity , 1987 .
[63] C. Dawson,et al. Hydroxyl radical production and lung injury in the rat following silica or titanium dioxide instillation in vivo. , 1995, American journal of respiratory cell and molecular biology.
[64] J. Finkelstein,et al. Antioxidant treatment attenuates cytokine and chemokine levels in murine macrophages following silica exposure. , 1999, Toxicology and applied pharmacology.
[65] K. Ha,et al. Silica-induced generation of reactive oxygen species in Rat2 fibroblast: role in activation of mitogen-activated protein kinase. , 1999, Biochemical and biophysical research communications.
[66] B. Lehnert,et al. Silica-induced generation of extracellular factor(s) increases reactive oxygen species in human bronchial epithelial cells. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[67] K. Donaldson,et al. Detection of surface free radical activity of respirable industrial fibres using supercoiled phi X174 RF1 plasmid DNA. , 1995, Carcinogenesis.
[68] D. Frazer,et al. Inhibition of Stimulant‐Induced Activation of Phagocytic Cells With Tetrandrine , 1991, Journal of leukocyte biology.