Oct4 plays a role in 2, 3, 7, 8 - tetrachlorobenzo-p-dioxin (TCDD) inducing cleft palate and inhibiting mesenchymal proliferation.
暂无分享,去创建一个
Xinxin Liu | L. Cui | Zengli Yu | Ning Li | Z. Wan | Xiaozhuan Liu | Yao Chen | Z. He | Zhan Gao | Y. Tao | Mengmeng Ji | Zhidong He | Xin-xin Liu | Zengli Yu
[1] Yan Zhang,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin Promotes Proliferation of Astrocyte Cells via the Akt/STAT3/Cyclin D1 Pathway. , 2019, Biomedical and environmental sciences : BES.
[2] A. Hirata,et al. Cleft palate formation after palatal fusion occurs due to the rupture of epithelial basement membranes. , 2018, Journal of cranio-maxillo-facial surgery : official publication of the European Association for Cranio-Maxillo-Facial Surgery.
[3] P. Chou,et al. Exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin increases the activation of aryl hydrocarbon receptor and is associated with the aggressiveness of osteosarcoma MG-63 osteoblast-like cells , 2018, Oncology letters.
[4] Yue Liu,et al. OCT4B regulates p53 and p16 pathway genes to prevent apoptosis of breast cancer cells. , 2018, Oncology letters.
[5] Wei Zhang,et al. Alterations in DNA methyltransferases and methyl-CpG binding domain proteins during cleft palate formation as induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in mice. , 2018, Molecular medicine reports.
[6] K. Tsim,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin induces alterations in myogenic differentiation of C2C12 cells. , 2018, Environmental pollution.
[7] Ding-wen Zhang,et al. Preliminary research on DNA methylation changes during murine palatogenesis induced by TCDD. , 2017, Journal of cranio-maxillo-facial surgery : official publication of the European Association for Cranio-Maxillo-Facial Surgery.
[8] Lanjuan Li,et al. Tryptophan derivatives regulate the transcription of Oct4 in stem-like cancer cells , 2015, Nature Communications.
[9] Byeong-chun Lee,et al. Oct4 overexpression facilitates proliferation of porcine fibroblasts and development of cloned embryos , 2014, Zygote.
[10] H. Schöler,et al. Role of Oct4 in the early embryo development , 2014, Cell Regeneration.
[11] Yixuan Fan,et al. Oct4 and Sox2 overexpression improves the proliferation and differentiation of bone mesenchymal stem cells in Xiaomeishan porcine. , 2013, Genetics and molecular research : GMR.
[12] T. Babak,et al. Oct4 Is Required ∼E7.5 for Proliferation in the Primitive Streak , 2013, PLoS genetics.
[13] M. Medvedovic,et al. Disruption of Aryl Hydrocarbon Receptor Homeostatic Levels during Embryonic Stem Cell Differentiation Alters Expression of Homeobox Transcription Factors that Control Cardiomyogenesis , 2013, Environmental health perspectives.
[14] N. Takahashi,et al. Teratogenic Factors Affect Transcription Factor Expression , 2013, Bioscience, biotechnology, and biochemistry.
[15] R. Redett,et al. Cleft Lip and Palate , 2013, Eplasty.
[16] Lei Chen,et al. OCT4 Positively Regulates Survivin Expression to Promote Cancer Cell Proliferation and Leads to Poor Prognosis in Esophageal Squamous Cell Carcinoma , 2012, PloS one.
[17] Xiaomeng He,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin induces a proteomic pattern that defines cleft palate formation in mice. , 2012, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[18] T. Beaty,et al. BMP4 Was Associated with NSCL/P in an Asian Population , 2012, PloS one.
[19] Natalia B. Ivanova,et al. Distinct lineage specification roles for NANOG, OCT4, and SOX2 in human embryonic stem cells. , 2012, Cell stem cell.
[20] R. Jiang,et al. Palatogenesis: morphogenetic and molecular mechanisms of secondary palate development , 2012, Development.
[21] H. Schöler,et al. Concise Review: Oct4 and More: The Reprogramming Expressway , 2012, Stem cells.
[22] J. Santos,et al. Risk variants in BMP4 promoters for nonsyndromic cleft lip/palate in a Chilean population , 2011, BMC Medical Genetics.
[23] J. Trosko,et al. Metformin Represses Self-Renewal of the Human Breast Carcinoma Stem Cells via Inhibition of Estrogen Receptor-Mediated OCT4 Expression , 2011, PloS one.
[24] Holger Schwender,et al. Evidence for gene‐environment interaction in a genome wide study of nonsyndromic cleft palate , 2011, Genetic epidemiology.
[25] A. Yen,et al. Activation of the aryl hydrocarbon receptor AhR Promotes retinoic acid-induced differentiation of myeloblastic leukemia cells by restricting expression of the stem cell transcription factor Oct4. , 2011, Cancer research.
[26] M. Pisano,et al. Palate morphogenesis: current understanding and future directions. , 2010, Birth defects research. Part C, Embryo today : reviews.
[27] Jungho Kim,et al. Oct-4 controls cell-cycle progression of embryonic stem cells , 2009, The Biochemical journal.
[28] J. Little,et al. Cleft lip and palate , 2009, The Lancet.
[29] Lin Qiu,et al. Transforming growth factor-β3 expression up-regulates on cleft palates induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in mice , 2009, Toxicology and industrial health.
[30] A. Jugessur,et al. Mutations in BMP4 are associated with subepithelial, microform, and overt cleft lip. , 2009, American journal of human genetics.
[31] A. Nawshad. Palatal seam disintegration: To die or not to die? that is no longer the question , 2008, Developmental dynamics : an official publication of the American Association of Anatomists.
[32] K. Mishima,et al. Morphological and immunohistochemical studies on cleft palates induced by 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin in mice , 2008, Congenital anomalies.
[33] C. Tohyama,et al. The 2005 World Health Organization reevaluation of human and Mammalian toxic equivalency factors for dioxins and dioxin-like compounds. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.
[34] K. Mishima,et al. Cleft lip and palate in mice treated with 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin: a morphological in vivo study , 2006, Congenital anomalies.
[35] Emily A. Stevens,et al. The Teratogenic Sensitivity to 2,3,7,8-Tetrachlorodibenzo-p-dioxin Is Modified by a Locus on Mouse Chromosome 3 , 2006, Molecular Pharmacology.
[36] M. Marazita,et al. Current concepts in the embryology and genetics of cleft lip and cleft palate. , 2004, Clinics in plastic surgery.
[37] D. Waxman,et al. P450 gene induction by structurally diverse xenochemicals: central role of nuclear receptors CAR, PXR, and PPAR. , 1999, Archives of biochemistry and biophysics.
[38] R. Baggs,et al. Aryl hydrocarbon receptor activation in genital tubercle, palate, and other embryonic tissues in 2,3,7, 8-tetrachlorodibenzo-p-dioxin-responsive lacZ mice. , 1998, Toxicology and applied pharmacology.
[39] Y. Bergman,et al. Retinoic acid represses Oct-3/4 gene expression through several retinoic acid-responsive elements located in the promoter-enhancer region , 1994, Molecular and cellular biology.
[40] L S Birnbaum,et al. A critical review of the developmental toxicity and teratogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin: recent advances toward understanding the mechanism. , 1990, Teratology.
[41] K. Yamashita,et al. Pathogenesis of cleft palate in mouse embryos exposed to 2,3,7, 8-tetrachlorodibenzo-p-dioxin (TCDD). , 2000, Teratogenesis, carcinogenesis, and mutagenesis.