Genetic polymorphisms in glycolytic pathway are associated with the prognosis of patients with early stage non-small cell lung cancer

[1]  Yan Wang,et al.  Enolase-1 is a therapeutic target in endometrial carcinoma , 2015, Oncotarget.

[2]  L. Zentilin,et al.  Targeting of surface alpha-enolase inhibits the invasiveness of pancreatic cancer cells , 2015, Oncotarget.

[3]  S. Miyamoto,et al.  Hexokinase II integrates energy metabolism and cellular protection: Akting on mitochondria and TORCing to autophagy , 2015, Cell Death and Differentiation.

[4]  S. Miyamoto,et al.  Hexokinase II integrates energy metabolism and cellular protection: Akting on mitochondria and TORCing to autophagy , 2014, Cell Death and Differentiation.

[5]  W. Oyen,et al.  Glucose Metabolism in NSCLC Is Histology-Specific and Diverges the Prognostic Potential of 18FDG-PET for Adenocarcinoma and Squamous Cell Carcinoma , 2014, Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer.

[6]  A. Bode,et al.  Glycolytic genes in cancer cells are more than glucose metabolic regulators , 2014, Journal of Molecular Medicine.

[7]  H. Christofk,et al.  Dichloroacetate should be considered with platinum-based chemotherapy in hypoxic tumors rather than as a single agent in advanced non-small cell lung cancer , 2014, Journal of Cancer Research and Clinical Oncology.

[8]  J. Geschwind,et al.  Tumor glycolysis as a target for cancer therapy: progress and prospects , 2013, Molecular Cancer.

[9]  J. Park,et al.  Polymorphisms in DNA repair and apoptosis-related genes and clinical outcomes of patients with non-small cell lung cancer treated with first-line paclitaxel-cisplatin chemotherapy. , 2013, Lung Cancer.

[10]  M. S. Mcmurtry,et al.  Mitochondrial activation by inhibition of PDKII suppresses HIF1a signaling and angiogenesis in cancer , 2013, Oncogene.

[11]  E. Michelakis,et al.  Pyruvate dehydrogenase kinase as a novel therapeutic target in oncology , 2013, Front. Oncol..

[12]  A. Jemal,et al.  Cancer statistics, 2013 , 2013, CA: a cancer journal for clinicians.

[13]  E. Gazzano,et al.  The pentose phosphate pathway: an antioxidant defense and a crossroad in tumor cell fate. , 2012, Free radical biology & medicine.

[14]  Raymond K. Auerbach,et al.  An Integrated Encyclopedia of DNA Elements in the Human Genome , 2012, Nature.

[15]  L. de Geus-Oei,et al.  Differences in metabolism between adeno- and squamous cell non-small cell lung carcinomas: spatial distribution and prognostic value of GLUT1 and MCT4. , 2012, Lung cancer.

[16]  C. Harris,et al.  p53 negatively regulates transcription of the pyruvate dehydrogenase kinase Pdk2. , 2012, Cancer research.

[17]  Eytan Ruppin,et al.  Selection for Translation Efficiency on Synonymous Polymorphisms in Recent Human Evolution , 2011, Genome biology and evolution.

[18]  D. Hanahan,et al.  Hallmarks of Cancer: The Next Generation , 2011, Cell.

[19]  R. Gross,et al.  Reversible High Affinity Inhibition of Phosphofructokinase-1 by Acyl-CoA , 2011, The Journal of Biological Chemistry.

[20]  T. Wong,et al.  ENO1, a potential prognostic head and neck cancer marker, promotes transformation partly via chemokine CCL20 induction. , 2010, European journal of cancer.

[21]  J. Mackey,et al.  Metabolic Modulation of Glioblastoma with Dichloroacetate , 2010, Science Translational Medicine.

[22]  L. Tanoue,et al.  The new lung cancer staging system. , 2009, Chest.

[23]  A. Yalçin,et al.  Regulation of glucose metabolism by 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatases in cancer. , 2009, Experimental and molecular pathology.

[24]  M. Rigoulet,et al.  Mitochondrial Oxidative Phosphorylation Is Regulated by Fructose 1,6-Bisphosphate , 2008, Journal of Biological Chemistry.

[25]  D. Sabatini,et al.  Cancer Cell Metabolism: Warburg and Beyond , 2008, Cell.

[26]  M.-H. Lee,et al.  Roles of p53, Myc and HIF-1 in Regulating Glycolysis — the Seventh Hallmark of Cancer , 2008, Cellular and Molecular Life Sciences.

[27]  Ru Wei,et al.  The M2 splice isoform of pyruvate kinase is important for cancer metabolism and tumour growth , 2008, Nature.

[28]  T. Wolff,et al.  The kelch protein NS1-BP interacts with alpha-enolase/MBP-1 and is involved in c-Myc gene transcriptional control. , 2007, Biochimica et biophysica acta.

[29]  M. Spitz,et al.  Genetic Variants in Cell Cycle Control Pathway Confer Susceptibility to Lung Cancer , 2007, Clinical Cancer Research.

[30]  Emma Saavedra,et al.  Energy metabolism in tumor cells , 2007, The FEBS journal.

[31]  T. Roche,et al.  Pyruvate dehydrogenase kinase regulatory mechanisms and inhibition in treating diabetes, heart ischemia, and cancer , 2007, Cellular and Molecular Life Sciences.

[32]  G. Semenza,et al.  HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia. , 2006, Cell metabolism.

[33]  F. Zaibak,et al.  Introduction of in vitro transcribed ENO1 mRNA into neuroblastoma cells induces cell death , 2005, BMC Cancer.

[34]  H. Schumny,et al.  Next Generation , 2020 .

[35]  W. Hong,et al.  Enolase-alpha is frequently down-regulated in non-small cell lung cancer and predicts aggressive biological behavior. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.

[36]  T. Kasten,et al.  Analysis of the phosphofructokinase subunits and isoenzymes in human tissues. , 1988, The Biochemical journal.

[37]  Melissa C. Greven,et al.  An integrated encyclopedia of DNA elements in the human genome , 2014 .

[38]  J. Plotkin,et al.  Synonymous but not the same: the causes and consequences of codon bias , 2011, Nature Reviews Genetics.

[39]  R. Deberardinis,et al.  The biology of cancer: metabolic reprogramming fuels cell growth and proliferation. , 2008, Cell metabolism.

[40]  Sébastien Bonnet,et al.  A mitochondria-K+ channel axis is suppressed in cancer and its normalization promotes apoptosis and inhibits cancer growth. , 2007, Cancer cell.

[41]  C. Johnson.  Progress and Prospects , 1991 .

[42]  N. Dubrawsky.  Cancer statistics , 1989, CA: a cancer journal for clinicians.