A collagen glucosyltransferase drives lung adenocarcinoma progression in mice
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Mitchell D. Miller | G. Phillips | J. Tainer | C. Creighton | M. Yamauchi | D. Gibbons | J. Kurie | K. Dalby | Yulong Chen | G. Fields | Chi-Lin Tsai | Xin Liu | Hou-Fu Guo | M. Terajima | P. Banerjee | Neus Bota-Rabassedas | Michal Tokmina-Roszyk | Xiaoyan Wang | Xiaochao Tan | Juhoon Lee | Roma Stawikowska | Jiang Yu | B. Leticia Rodriguez | Juhoon Lee | N. Bota-Rabassedas
[1] Luca Gasperini,et al. The stiffness of living tissues and its implications for tissue engineering , 2020, Nature Reviews Materials.
[2] Huanming Yang,et al. An Integrated Gene Expression Landscape Profiling Approach to Identify Lung Tumor Endothelial Cell Heterogeneity and Angiogenic Candidates. , 2020, Cancer cell.
[3] Philippe M. Campeau,et al. Bruck syndrome 2 variant lacking congenital contractures and involving a novel compound heterozygous PLOD2 mutation. , 2020, Bone.
[4] M. Yamauchi,et al. Role of Glycosyltransferase 25 domain 1 in Type I Collagen Glycosylation and Molecular Phenotypes. , 2019, Biochemistry.
[5] J. Uitto,et al. Mutations in PLOD3, encoding lysyl hydroxylase 3, cause a complex connective tissue disorder including recessive dystrophic epidermolysis bullosa-like blistering phenotype with abnormal anchoring fibrils and type VII collagen deficiency. , 2019, Matrix biology : journal of the International Society for Matrix Biology.
[6] F. Forneris,et al. SiMPLOD, a Structure‐Integrated Database of Collagen Lysyl Hydroxylase (LH/PLOD) Enzyme Variants , 2019, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[7] V. Olieric,et al. Molecular architecture of the multifunctional collagen lysyl hydroxylase and glycosyltransferase LH3 , 2018, Nature Communications.
[8] Torsten Schwede,et al. SWISS-MODEL: homology modelling of protein structures and complexes , 2018, Nucleic Acids Res..
[9] Mitchell D. Miller,et al. Pro-metastatic collagen lysyl hydroxylase dimer assemblies stabilized by Fe2+-binding , 2018, Nature Communications.
[10] Hong Jiang,et al. Tumor-associated fibrosis as a regulator of tumor immunity and response to immunotherapy , 2017, Cancer Immunology, Immunotherapy.
[11] G. Phillips,et al. A scalable lysyl hydroxylase 2 expression system and luciferase-based enzymatic activity assay. , 2017, Archives of biochemistry and biophysics.
[12] M. Girolami,et al. Regulation of post-Golgi LH3 trafficking is essential for collagen homeostasis , 2016, Nature Communications.
[13] H. Hamazaki,et al. Catalytic site of human protein-glucosylgalactosylhydroxylysine glucosidase: Three crucial carboxyl residues were determined by cloning and site-directed mutagenesis. , 2016, Biochemical and biophysical research communications.
[14] Lixia Diao,et al. Metastasis is regulated via microRNA-200/ZEB1 axis control of tumor cell PD-L1 expression and intratumoral immunosuppression , 2014, Nature Communications.
[15] G. Christofori,et al. The RNA-binding protein Rbfox2: an essential regulator of EMT-driven alternative splicing and a mediator of cellular invasion , 2014, Oncogene.
[16] J. Tazi,et al. MBNL1 and RBFOX2 cooperate to establish a splicing programme involved in pluripotent stem cell differentiation , 2013, Nature Communications.
[17] M. Yamauchi,et al. Lysine post-translational modifications of collagen. , 2012, Essays in biochemistry.
[18] Nicholas C. Flytzanis,et al. An EMT–Driven Alternative Splicing Program Occurs in Human Breast Cancer and Modulates Cellular Phenotype , 2011, PLoS genetics.
[19] Mikala Egeblad,et al. Matrix Crosslinking Forces Tumor Progression by Enhancing Integrin Signaling , 2009, Cell.
[20] Alexander Pertsemlidis,et al. Contextual extracellular cues promote tumor cell EMT and metastasis by regulating miR-200 family expression. , 2009, Genes & development.
[21] M. Garcia-Blanco,et al. TIA nuclear proteins regulate the alternate splicing of lysyl hydroxylase 2. , 2009, The Journal of investigative dermatology.
[22] H. Cox,et al. A connective tissue disorder caused by mutations of the lysyl hydroxylase 3 gene. , 2008, American journal of human genetics.
[23] L. Meyaard. The inhibitory collagen receptor LAIR‐1 (CD305) , 2008, Journal of leukocyte biology.
[24] Chunguang Wang,et al. Expanding the lysyl hydroxylase toolbox: New insights into the localization and activities of lysyl hydroxylase 3 (LH3) , 2007, Journal of cellular physiology.
[25] M. Granato,et al. The Myotomal diwanka (lh3) Glycosyltransferase and Type XVIII Collagen Are Critical for Motor Growth Cone Migration , 2006, Neuron.
[26] D. Reid,et al. Association of PLOD1 polymorphisms with bone mineral density in a population-based study of women from the UK , 2006, Osteoporosis International.
[27] R. Bank,et al. Phenotypic and molecular characterization of Bruck syndrome (osteogenesis imperfecta with contractures of the large joints) caused by a recessive mutation in PLOD2 , 2004, American journal of medical genetics. Part A.
[28] E. Middelkoop,et al. Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon. , 2004, Matrix biology : journal of the International Society for Matrix Biology.
[29] L. Ala‐Kokko,et al. A homozygous stop codon in the lysyl hydroxylase gene in two siblings with Ehlers–Danlos syndrome type VI , 1992, Nature Genetics.
[30] E. Beachey,et al. Collagen-Induced Platelet Aggregation: Involvement of an Active Glycopeptide Fragment (α1-CB5) , 1973, Science.
[31] M. Yamauchi,et al. Lysine Hydroxylation and Cross-Linking of Collagen. , 2019, Methods in molecular biology.