Red blood cell distribution width is associated with increased interactions of blood cells with vascular wall
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M. Wojtkowski | Z. Gaciong | A. Szkulmowska | L. Prahl Wittberg | D. Religa | P. Religa | F. Lundell | N. Berg | S. Ananthaseshan | M. Sacharczuk | P. Poznański | P. Andziak | K. Bojakowski | T. Guzik | Dominik S Skiba | J. McKenzie | H. Menkens | Dominik Skiba | Niclas Berg | Jordan McKenzie
[1] Toshiko Tanaka,et al. Proteins in the pathway from high red blood cell width distribution to all-cause mortality , 2022, EBioMedicine.
[2] Yuanmin Li,et al. ΔRDW: A Novel Indicator with Predictive Value for the Diagnosis and Treatment of Multiple Diseases , 2021, International journal of general medicine.
[3] J. Michel,et al. Red Blood Cells and Hemoglobin in Human Atherosclerosis and Related Arterial Diseases , 2020, International journal of molecular sciences.
[4] M. Westover,et al. Association of Red Blood Cell Distribution Width With Mortality Risk in Hospitalized Adults With SARS-CoV-2 Infection , 2020, JAMA network open.
[5] M. Schwartz,et al. Endothelial-to-Mesenchymal Transition, Vascular Inflammation, and Atherosclerosis , 2020, Frontiers in Cardiovascular Medicine.
[6] Maciej Wojtkowski,et al. In vivo brain imaging with multimodal optical coherence microscopy in a mouse model of thromboembolic photochemical stroke , 2020, Neurophotonics.
[7] A. Chaudhury,et al. Single-cell modeling of routine clinical blood tests reveals transient dynamics of human response to blood loss , 2019, eLife.
[8] S. Doblas,et al. Red blood cells collision with the wall in human coronary arteries promotes oxidative stress in early stage atheroma , 2019, Archives of Cardiovascular Diseases Supplements.
[9] Sabia Z. Abidi,et al. Simultaneous polymerization and adhesion under hypoxia in sickle cell disease , 2018, Proceedings of the National Academy of Sciences.
[10] W. Tan,et al. In Vitro Model of Physiological and Pathological Blood Flow with Application to Investigations of Vascular Cell Remodeling. , 2015, Journal of visualized experiments : JoVE.
[11] P. Religa,et al. Human cytomegalovirus inhibits erythropoietin production. , 2014, Journal of the American Society of Nephrology : JASN.
[12] T. Walshe,et al. The Role of Shear-Induced Transforming Growth Factor-&bgr; Signaling in the Endothelium , 2013, Arteriosclerosis, thrombosis, and vascular biology.
[13] Maciej Wojtkowski,et al. Assessment of the flow velocity of blood cells in a microfluidic device using joint spectral and time domain optical coherence tomography. , 2013, Optics express.
[14] C. Murry,et al. Monocyte Chemoattractant Protein 1–Mediated Migration of Mesenchymal Stem Cells Is a Source of Intimal Hyperplasia , 2013, Arteriosclerosis, thrombosis, and vascular biology.
[15] A. Chauhan,et al. Red blood cells mediate the onset of thrombosis in the ferric chloride murine model. , 2013, Blood.
[16] A. H. Isfahani,et al. Forces on a wall-bound leukocyte in a small vessel due to red cells in the blood stream. , 2012, Biophysical journal.
[17] G. Panasenko,et al. Finite platelet size could be responsible for platelet margination effect. , 2011, Biophysical journal.
[18] P. Siesjö,et al. Detection of human cytomegalovirus in medulloblastomas reveals a potential therapeutic target. , 2011, The Journal of clinical investigation.
[19] S. Jackson,et al. Erythrocyte Hemolysis and Hemoglobin Oxidation Promote Ferric Chloride-induced Vascular Injury* , 2009, Journal of Biological Chemistry.
[20] A. Hansen,et al. Anti-VEGF agents confer survival advantages to tumor-bearing mice by improving cancer-associated systemic syndrome , 2008, Proceedings of the National Academy of Sciences.
[21] Lance L. Munn,et al. Lattice-Boltzmann simulation of blood flow in digitized vessel networks , 2008, Comput. Math. Appl..
[22] Michael M. Dupin,et al. Blood Cell Interactions and Segregation in Flow , 2008, Annals of Biomedical Engineering.
[23] J. Hathcock. Flow effects on coagulation and thrombosis. , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[24] J. Thyberg,et al. Allogenic immune response promotes the accumulation of host-derived smooth muscle cells in transplant arteriosclerosis. , 2005, Cardiovascular research.
[25] E. Schiffrin. Role of endothelin-1 in hypertension and vascular disease. , 2001, American journal of hypertension.
[26] M J Pearson,et al. Influence of erythrocyte aggregation on leukocyte margination in postcapillary venules of rat mesentery. , 2000, American journal of physiology. Heart and circulatory physiology.
[27] L. Snyder,et al. Oxidation and erythrocyte senescence. , 2000, Current opinion in hematology.
[28] R K Jain,et al. Selectin- and integrin-mediated T-lymphocyte rolling and arrest on TNF-alpha-activated endothelium: augmentation by erythrocytes. , 1995, Biophysical journal.
[29] D. Faller,et al. Sickle erythrocytes, after sickling, regulate the expression of the endothelin-1 gene and protein in human endothelial cells in culture. , 1995, The Journal of clinical investigation.
[30] R. Heethaar,et al. Near-wall excess of platelets induced by lateral migration of erythrocytes in flowing blood. , 1993, The American journal of physiology.
[31] D. Slaaf,et al. Concentration profile of blood platelets differs in arterioles and venules. , 1992, The American journal of physiology.
[32] H. Weiss,et al. The effect of shear rate on platelet interaction with subendothelium exposed to citrated human blood. , 1980, Microvascular research.
[33] J. Nelson,et al. Measurement of fluid-flow-velocity profile in turbid media by the use of optical Doppler tomography. , 1997, Applied optics.
[34] S Chien,et al. The interaction of leukocytes and erythrocytes in capillary and postcapillary vessels. , 1980, Microvascular research.