Caspase-9 Activation Results in Downstream Caspase-8 Activation and Bid Cleavage in 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Induced Parkinson's Disease
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M. Beal | V. Viswanath | J. Andersen | Lichuan Yang | Sylvia F. Chen | Lichuan Yang | M. Flint Beal | F. Yantiri | R. Boonplueang | Julie K. Andersen | Sylvia Chen | Fang Feng Stevenson | Veena Viswanath | Yongqin Wu | Rapee Boonplueang | Ferda Yantiri | F. Stevenson | Yongqin Wu | Julie K. Andersen
[1] Y. Lazebnik,et al. Caspases: enemies within. , 1998, Science.
[2] E. Hirsch,et al. Caspase-8 Is an Effector in Apoptotic Death of Dopaminergic Neurons in Parkinson's Disease, But Pathway Inhibition Results in Neuronal Necrosis , 2001, The Journal of Neuroscience.
[3] J. Blenis,et al. Caspase-8 Is Required for Cell Death Induced by Expanded Polyglutamine Repeats , 1999, Neuron.
[4] W. Tatton,et al. Etiology and pathogenesis of Parkinson's disease. , 1999, Annual review of neuroscience.
[5] J. Velier,et al. Caspase-8 and Caspase-3 Are Expressed by Different Populations of Cortical Neurons Undergoing Delayed Cell Death after Focal Stroke in the Rat , 1999, The Journal of Neuroscience.
[6] G. Núñez,et al. Caspases: the proteases of the apoptotic pathway , 1998, Oncogene.
[7] R. Flavell,et al. Caspase knockouts: matters of life and death , 1999, Cell Death and Differentiation.
[8] J. Penney,et al. 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyride Neurotoxicity Is Attenuated in Mice Overexpressing Bcl-2 , 1998, The Journal of Neuroscience.
[9] S. Hersch,et al. Huntington aggregates may not predict neuronal death in Huntington's disease. , 1999, Annals of neurology.
[10] G. Salvesen,et al. Caspases: Intracellular Signaling by Proteolysis , 1997, Cell.
[11] M. Zimmermann,et al. Increase in bax expression in substantia nigra following 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) treatment of mice , 1996, Neuroscience Letters.
[12] N. Hattori,et al. Mitochondrial dysfunction in parkinson's disease , 1998, Annals of neurology.
[13] M. Hayden,et al. Caspases and neurodegeneration: on the cutting edge of new therapeutic approaches , 2000, Clinical genetics.
[14] E. Melamed,et al. Transgenic mice expressing human Bcl-2 in their neurons are resistant to 6-hydroxydopamine and 1-methyl-4-phenyl-1,2,3,6- tetrahydropyridine neurotoxicity. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[15] S. Kumar,et al. Mechanisms mediating caspase activation in cell death , 1999, Cell Death and Differentiation.
[16] S. Kish,et al. In situ detection of apoptotic nuclei in the substantia nigra compacta of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-treated mice using terminal deoxynucleotidyl transferase labelling and acridine orange staining , 1997, Neuroscience.
[17] J. Beckmann,et al. Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally. , 1998, Immunity.
[18] A. Fisher,et al. Crystal structure of baculovirus P35: role of a novel reactive site loop in apoptotic caspase inhibition , 1999, The EMBO journal.
[19] S. Hersch,et al. Huntingtin aggregates may not predict neuronal death in Huntington's disease , 1999 .
[20] D. Perl,et al. A fluorescent double‐labeling method to detect and confirm apoptotic nuclei in parkinson's disease , 1998, Annals of neurology.
[21] J C Reed,et al. Ion Channel Activity of the BH3 Only Bcl-2 Family Member, BID* , 1999, The Journal of Biological Chemistry.
[22] J. Rothman,et al. Baculovirus p35 prevents developmentally programmed cell death and rescues a ced‐9 mutant in the nematode Caenorhabditis elegans. , 1994, The EMBO journal.
[23] H. Steller,et al. Blocking apoptosis prevents blindness in Drosophila retinal degeneration mutants , 1998, Nature.
[24] J. Mark Treherne,et al. The role of apoptosis in neurodegenerative diseases. , 1997, Progress in drug research. Fortschritte der Arzneimittelforschung. Progres des recherches pharmaceutiques.
[25] R. Elble. Animal Models of Action Tremor , 2008, Movement disorders : official journal of the Movement Disorder Society.
[26] G. Salvesen,et al. Caspases - controlling intracellular signals by protease zymogen activation. , 2000, Biochimica et biophysica acta.
[27] Emad S. Alnemri,et al. Ordering the Cytochrome c–initiated Caspase Cascade: Hierarchical Activation of Caspases-2, -3, -6, -7, -8, and -10 in a Caspase-9–dependent Manner , 1999, The Journal of cell biology.
[28] V. Viswanath,et al. Transgenic mice neuronally expressing baculoviral p35 are resistant to diverse types of induced apoptosis, including seizure-associated neurodegeneration. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[29] Junying Yuan,et al. Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.
[30] Xiaodong Wang,et al. Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors , 1998, Cell.
[31] Y. Agid,et al. Apoptosis and autophagy in nigral neurons of patients with Parkinson's disease. , 1997, Histology and histopathology.
[32] S. Przedborski,et al. Mechanisms of MPTP toxicity. , 1998, Movement disorders : official journal of the Movement Disorder Society.
[33] Sharad Kumar,et al. Targeted disruption of caspase genes in mice: What they tell us about the functions of individual caspases in apoptosis , 1999, Immunology and cell biology.
[34] T. Kuwana,et al. Apoptosis Induction by Caspase-8 Is Amplified through the Mitochondrial Release of Cytochrome c * , 1998, The Journal of Biological Chemistry.
[35] A Hartley,et al. Complex I Inhibitors Induce Dose‐Dependent Apoptosis in PC12 Cells: Relevance to Parkinson's Disease , 1994, Journal of neurochemistry.
[36] S. Paul,et al. Peptide inhibitors of caspase-3-like proteases attenuate MPP+ -induced toxicity of cultured fetal rat mesencephalic dopamine neurons , 1998, Neuroscience.
[37] J. Mankovich,et al. Inhibition of ICE family proteases by baculovirus antiapoptotic protein p35. , 1995, Science.
[38] Keisuke Kuida,et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice , 1996, Nature.
[39] Keisuke Kuida,et al. Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9 , 1998, Cell.
[40] S. Srinivasula,et al. Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade , 1997, Cell.
[41] M. Ruberg,et al. Neuropharmacologic aspects of apoptosis: significance for neurodegenerative diseases. , 1999, Clinical neuropharmacology.
[42] E. Hirsch,et al. Caspase-3: A vulnerability factor and final effector in apoptotic death of dopaminergic neurons in Parkinson's disease. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[43] S. Paul,et al. Peptide inhibitors of caspase-3-like proteases attenuate 1-methyl-4-phenylpyridinum-induced toxicity of cultured fetal rat mesencephalic dopamine neurons. , 1998, Neuroscience.
[44] Y. Oh,et al. Cleavage of Bax is mediated by caspase‐dependent or ‐independent calpain activation in dopaminergic neuronal cells: protective role of Bcl‐2 , 2001, Journal of neurochemistry.
[45] B. Miller,et al. Frontotemporal dementia: Neuropil spheroids and presynaptic terminal degeneration , 1998, Annals of neurology.
[46] K. Jellinger,et al. Activation of caspase-3 in single neurons and autophagic granules of granulovacuolar degeneration in Alzheimer's disease. Evidence for apoptotic cell death. , 1999, The American journal of pathology.
[47] Y. Mizuno,et al. Histochemical detection of apoptosis in Parkinson's disease , 1996, Journal of the Neurological Sciences.
[48] S. Korsmeyer,et al. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death , 2001, Science.
[49] D. Green,et al. Suicidal Tendencies: Apoptotic Cell Death by Caspase Family Proteinases* , 1999, The Journal of Biological Chemistry.
[50] B. Hyman,et al. Do defecs in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases? , 1993, Trends in Neurosciences.
[51] E. Hirsch,et al. CD95 (APO‐1/Fas) and Parkinson's disease , 1998, Annals of neurology.
[52] S. Tabrizi,et al. Mitochondria in the etiology and pathogenesis of parkinson's disease , 1998, Annals of neurology.
[53] A J Hughes,et al. Clinicopathological aspects of Parkinson's disease. , 1997, European neurology.
[54] M. Beal,et al. Homocysteic acid lesions in rat striatum spare somatostatin—;Neuropeptide Y (NADPH-diaphorase) neurons , 1990, Neuroscience Letters.
[55] I. Ziv,et al. Modulation of control mechanisms of dopamine-induced apoptosis--a future approach to the treatment of Parkinson's disease? , 1997, Journal of neural transmission. Supplementum.
[56] K. Jellinger. Cell death mechanisms in Parkinson's disease , 2000, Journal of Neural Transmission.
[57] G M Rubin,et al. Expression of baculovirus P35 prevents cell death in Drosophila. , 1994, Development.
[58] P. Demoly,et al. [Transgenic mice]. , 1992, Annales de dermatologie et de venereologie.
[59] C. Martínez-A,et al. Blocked negative selection of developing T cells in mice expressing the baculovirus p35 caspase inhibitor , 1999, The EMBO journal.
[60] David G. Kirsch,et al. Caspase-3-dependent Cleavage of Bcl-2 Promotes Release of Cytochrome c * , 1999, The Journal of Biological Chemistry.
[61] V. Kidd,et al. Caspase-8 Activation and Bid Cleavage Contribute to MCF7 Cellular Execution in a Caspase-3-dependent Manner during Staurosporine-mediated Apoptosis* , 2000, The Journal of Biological Chemistry.
[62] J. Miyazaki,et al. Targeted expression of baculovirus p35 caspase inhibitor in oligodendrocytes protects mice against autoimmune‐mediated demyelination , 2000, The EMBO journal.
[63] E. Alnemri,et al. Interaction of the baculovirus anti-apoptotic protein p35 with caspases. Specificity, kinetics, and characterization of the caspase/p35 complex. , 1998, Biochemistry.
[64] J. Andersen. Does neuronal loss in Parkinson's disease involve programmed cell death? , 2001, BioEssays : news and reviews in molecular, cellular and developmental biology.
[65] J. Wands,et al. P53- and CD95-associated apoptosis in neurodegenerative diseases. , 1998, Laboratory investigation; a journal of technical methods and pathology.
[66] H. Gundersen,et al. Unbiased stereological estimation of the total number of neurons in the subdivisions of the rat hippocampus using the optical fractionator , 1991, The Anatomical record.
[67] L. Greene,et al. Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor. , 1976, Proceedings of the National Academy of Sciences of the United States of America.