Blood and lymph node dissemination of clonal genome-intact HIV-1 DNA sequences during suppressive antiretroviral therapy.
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Xu G. Yu | G. Pantaleo | M. Cavassini | M. Lichterfeld | J. Corpataux | M. Perreau | J. Z. zur Wiesch | Julian Schulze zur Wiesch | Jane E. Blackmer | Guinevere Q. Lee | Riddhima Banga | H. Kuo | Ce Gao | Guinevere Q. Lee | Xu G. Yu | G. Lee
[1] S. Hughes,et al. HIV-1 in lymph nodes is maintained by cellular proliferation during antiretroviral therapy. , 2019, The Journal of clinical investigation.
[2] E. Boritz,et al. Memory CD4 + T-Cells Expressing HLA-DR Contribute to HIV Persistence During Prolonged Antiretroviral Therapy , 2019, Front. Microbiol..
[3] Z. Grossman,et al. No evidence of ongoing HIV replication or compartmentalization in tissues during combination antiretroviral therapy: Implications for HIV eradication , 2019, Science Advances.
[4] G. Pantaleo,et al. Lymph node migratory dendritic cells modulate HIV-1 transcription through PD-1 engagement , 2019, PLoS pathogens.
[5] B. Walker,et al. HIV-1 DNA sequence diversity and evolution during acute subtype C infection , 2019, Nature Communications.
[6] S. Migueles,et al. Longitudinal HIV sequencing reveals reservoir expression leading to decay which is obscured by clonal expansion , 2019, Nature Communications.
[7] W. Paxton,et al. Blood CXCR3+ CD4 T Cells Are Enriched in Inducible Replication Competent HIV in Aviremic Antiretroviral Therapy-Treated Individuals , 2018, Front. Immunol..
[8] J. Lifson,et al. Defining total-body AIDS-virus burden with implications for curative strategies , 2017, Nature Medicine.
[9] E. Rosenberg,et al. Clonal expansion of genome-intact HIV-1 in functionally polarized Th1 CD4+ T cells , 2017, The Journal of clinical investigation.
[10] J. Mullins,et al. Clonal Expansion of Human Immunodeficiency Virus-Infected Cells and Human Immunodeficiency Virus Persistence During Antiretroviral Therapy. , 2017, The Journal of infectious diseases.
[11] G. Pantaleo,et al. PD-1+ and follicular helper T cells are responsible for persistent HIV-1 transcription in treated aviremic individuals , 2016, Nature Medicine.
[12] L. Picker,et al. A B cell follicle sanctuary permits persistent productive SIV infection in elite controllers , 2014, Nature Medicine.
[13] Sarah B. Laskey,et al. Replication-Competent Noninduced Proviruses in the Latent Reservoir Increase Barrier to HIV-1 Cure , 2013, Cell.
[14] G. Pantaleo,et al. Follicular helper T cells serve as the major CD4 T cell compartment for HIV-1 infection, replication, and production , 2013, The Journal of experimental medicine.
[15] Geneviève Boucher,et al. HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation , 2009, Nature Medicine.