Specificity of TLS-CHOP rearrangement for classic myxoid/round cell liposarcoma: absence in predominantly myxoid well-differentiated liposarcomas.

[1]  M. Ladanyi,et al.  Monoclonality of multifocal myxoid liposarcoma: confirmation by analysis of TLS-CHOP or EWS-CHOP rearrangements. , 2000, Clinical cancer research : an official journal of the American Association for Cancer Research.

[2]  M. Sasaki,et al.  Characteristics of genomic breakpoints in TLS-CHOP translocations in liposarcomas suggest the involvement of Translin and topoisomerase II in the process of translocation , 1999, Oncogene.

[3]  G. Adelmant,et al.  Human Translocation Liposarcoma-CCAAT/Enhancer Binding Protein (C/EBP) Homologous Protein (TLS-CHOP) Oncoprotein Prevents Adipocyte Differentiation by Directly Interfering with C/EBPβ Function* , 1998, The Journal of Biological Chemistry.

[4]  M. Ushijima,et al.  Detection of TLS/FUS‐CHOP Fusion Transcripts in Myxoid and Round Cell Liposarcomas by Nested Reverse Transcription‐Polymerase Chain Reaction Using Archival Paraffin‐embedded Tissues , 1998, Diagnostic molecular pathology : the American journal of surgical pathology, part B.

[5]  T. Ishida,et al.  Oncogenic transformation and inhibition of adipocytic conversion of preadipocytes by TLS/FUS-CHOP type II chimeric protein. , 1997, The American journal of pathology.

[6]  F. Mitelman,et al.  Additional evidence of a variant translocation t(12;22) with EWS/CHOP fusion in myxoid liposarcoma: clinicopathological features , 1997, The Journal of pathology.

[7]  C. Fletcher,et al.  Dedifferentiated myxoid liposarcoma: a clinicopathological study suggesting a closer relationship between myxoid and well‐differentiated liposarcoma , 1997, Histopathology.

[8]  D. Ron TLS-CHOP and the role of RNA-binding proteins in oncogenic transformation. , 1997, Current topics in microbiology and immunology.

[9]  F. Mitelman,et al.  Genomic PCR detects tumor cells in peripheral blood from patients with myxoid liposarcoma , 1996, Genes, chromosomes & cancer.

[10]  F. Mitelman,et al.  Combined morphologic and karyotypic study of 59 atypical lipomatous tumors. Evaluation of their relationship and differential diagnosis with other adipose tissue tumors (a report of the CHAMP Study Group). , 1996, The American journal of surgical pathology.

[11]  M. Höglund,et al.  Expression patterns of the human sarcoma-associated genes FUS and EWS and the genomic structure of FUS. , 1996, Genomics.

[12]  F. Mitelman,et al.  Combined morphologic and karyotypic study of 28 myxoid liposarcomas. Implications for a revised morphologic typing, (a report from the CHAMP Group). , 1996, The American journal of surgical pathology.

[13]  F. Sim,et al.  The clinicopathologic spectrum of myxoid and round cell liposarcoma: A study of 95 cases , 1996, Cancer.

[14]  M. Höglund,et al.  Fusion of the EWS and CHOP genes in myxoid liposarcoma. , 1996, Oncogene.

[15]  F. Mitelman,et al.  Correlation between clinicopathological features and karyotype in lipomatous tumors. A report of 178 cases from the Chromosomes and Morphology (CHAMP) Collaborative Study Group. , 1996, The American journal of pathology.

[16]  S. Mori,et al.  Chimeric TLS/FUS-CHOP gene expression and the heterogeneity of its junction in human myxoid and round cell liposarcoma. , 1995, The American journal of pathology.

[17]  F. Mitelman,et al.  Two distinct FUS breakpoint clusters in myxoid liposarcoma and acute myeloid leukemia with the translocations t(12;16) and t(16;21). , 1995, Oncogene.

[18]  C. Fletcher,et al.  Translocation t(12;16)(q13;p11) in myxoid liposarcoma and round cell liposarcoma: molecular and cytogenetic analysis. , 1995, Cancer research.

[19]  M. Höglund,et al.  Characterization of the CHOP breakpoints and fusion transcripts in myxoid liposarcomas with the 12;16 translocation. , 1994, Cancer research.

[20]  F. Mitelman,et al.  No rearrangements of the CHOP gene in malignant fibrous histiocytoma. , 1994, Cancer genetics and cytogenetics.

[21]  S. Butterwith Molecular events in adipocyte development. , 1994, Pharmacology & therapeutics.

[22]  M. Fernö,et al.  Liposarcoma: A population‐based epidemiologic and prognostic study of features of 43 patients, including tumor DNA content , 1993, International journal of cancer.

[23]  M. Ladanyi,et al.  EWS Rearrangement in E wing's Sarcoma and Peripheral Neuroectodermal Tumor Molecular Detection and Correlation with Cytogenetic Analysis and MIC2 Expression , 1993, Diagnostic molecular pathology : the American journal of surgical pathology, part B.

[24]  P. Cin,et al.  Cytogenetic and fluorescence in situ hybridization investigation of ring chromosomes characterizing a specific pathologic subgroup of adipose tissue tumors. , 1993, Cancer genetics and cytogenetics.

[25]  N. Mandahl,et al.  Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma , 1993, Nature.

[26]  R. Larson,et al.  Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma , 1993, Nature Genetics.

[27]  F. Mitelman,et al.  Rearrangement of the transcription factor gene CHOP in myxoid liposarcomas with t(12;16)(q13;p11) , 1992, Genes, chromosomes & cancer.

[28]  S. Leong,et al.  Cytogenetic findings in liposarcoma correlate with histopathologic subtypes , 1992, Cancer.

[29]  D. Ron,et al.  CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription. , 1992, Genes & development.