La biocompatibilité des solutions de dialyse péritonéale
暂无分享,去创建一个
O. Moranne | J. Rougier | P. Ronco | É. Boulanger | M. Wautier | J. Wautier | D. Pagniez
[1] P. Gane,et al. The triggering of human peritoneal mesothelial cell apoptosis and oncosis by glucose and glycoxydation products. , 2004, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[2] K. Craig,et al. The Euro-Balance Trial: the effect of a new biocompatible peritoneal dialysis fluid (balance) on the peritoneal membrane. , 2004, Kidney international.
[3] N. Lameire,et al. The Effects of Peritoneal Dialysis Solutions on Peritoneal Host Defense , 2004, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[4] Y. Ohta,et al. Glucose degradation products (GDP) retard remesothelialization independently of D-glucose concentration. , 2003, Kidney international.
[5] H. Morita,et al. Short-Term Biological Effects of a New and Less Acidic Fluid for Peritoneal Dialysis , 2003, Blood Purification.
[6] R. Tilton,et al. Inhibition of the interaction of AGE-RAGE prevents hyperglycemia-induced fibrosis of the peritoneal membrane. , 2003, Journal of the American Society of Nephrology : JASN.
[7] K. Książek,et al. Mesothelial Toxicity of Peritoneal Dialysis Fluids is Related Primarily to Glucose Degradation Products, Not to Glucose Per Se , 2003, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[8] T. Chan,et al. Different effects of amino acid-based and glucose-based dialysate from peritoneal dialysis patients on mesothelial cell ultrastructure and function. , 2003, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[9] S. Meijer,et al. Single Exposure of Mesothelial Cells to Glucose Degradation Products (GDPs) Yields Early Advanced Glycation End-Products (AGEs) and a Proinflammatory Response , 2003, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[10] T. Niwa,et al. Immunohistochemical detection of an AGE, a ligand for macrophage receptor, in peritoneum of CAPD patients. , 2003, Kidney international. Supplement.
[11] N. Lameire,et al. Effects of conventional and new peritoneal dialysis fluids on leukocyte recruitment in the rat peritoneal membrane. , 2003, Journal of the American Society of Nephrology : JASN.
[12] M. Kasuga,et al. Methylglyoxal induces apoptosis through activation of p38 mitogen-activated protein kinase in rat mesangial cells. , 2003, Kidney international.
[13] C. Wanner,et al. Glucose degradation products in PD fluids: do they disappear from the peritoneal cavity and enter the systemic circulation? , 2003, Kidney international.
[14] S. Mujais,et al. Pharmacokinetics of icodextrin in peritoneal dialysis patients. , 2002, Kidney international. Supplement.
[15] C. Holmes,et al. Biocompatibility of icodextrin. , 2002, Kidney international. Supplement.
[16] A. Wieslander,et al. 3,4-Dideoxyglucosone-3-ene (3,4-DGE): a cytotoxic glucose degradation product in fluids for peritoneal dialysis. , 2002, Kidney international.
[17] V. Monnier,et al. Alterations in renal mitochondrial respiration in response to the reactive oxoaldehyde methylglyoxal. , 2002, American journal of physiology. Renal physiology.
[18] R. Krediet,et al. Clinical advantages of new peritoneal dialysis solutions. , 2002, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[19] J. van de Voorde,et al. Hemodynamic effects of peritoneal dialysis solutions on the rat peritoneal membrane: role of acidity, buffer choice, glucose concentration, and glucose degradation products. , 2002, Journal of the American Society of Nephrology : JASN.
[20] J. Wautier,et al. [Advanced glycosylation end products (AGE): new toxins?]. , 2002, Nephrologie.
[21] J. Wautier,et al. AGEs bind to mesothelial cells via RAGE and stimulate VCAM-1 expression. , 2002, Kidney international.
[22] M. Brownlee. Biochemistry and molecular cell biology of diabetic complications , 2001, Nature.
[23] P. Södersten,et al. Acute effects of peritoneal dialysis solutions on appetite in non-uremic rats. , 2001, Kidney international.
[24] S. Davies,et al. Peritoneal glucose exposure and changes in membrane solute transport with time on peritoneal dialysis. , 2001, Journal of the American Society of Nephrology : JASN.
[25] R. Krediet,et al. Bicarbonate/lactate-based peritoneal dialysis solution increases cancer antigen 125 and decreases hyaluronic acid levels. , 2001, Kidney international.
[26] J. Bernheim,et al. The Effect of Advanced Glycation End-Products and Aminoguanidine on Tnfα Production by Rat Peritoneal Macrophages , 2001, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[27] A. Christensson,et al. Long-term clinical effects of a peritoneal dialysis fluid with less glucose degradation products. , 2001, Kidney international.
[28] T. Miyata,et al. Effect of dwell time on carbonyl stress using icodextrin and amino acid peritoneal dialysis fluids. , 2000, Kidney international.
[29] P. Stenvinkel,et al. Biocompatibility of New Peritoneal Dialysis Solutions: What Can We Hope to Achieve? , 2000, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[30] C. Holmes,et al. Strategies to Reduce Glucose Exposure in Peritoneal Dialysis Patients , 2000, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[31] U. Frei,et al. Effect of glucose degradation products on human peritoneal mesothelial cell function. , 2000, Journal of the American Society of Nephrology : JASN.
[32] A. Tranaeus. A long-term study of a bicarbonate/lactate-based peritoneal dialysis solution--clinical benefits. The Bicarbonate/Lactate Study Group. , 2000, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[33] T. Miyata,et al. Glucose degradation product methylglyoxal enhances the production of vascular endothelial growth factor in peritoneal cells: role in the functional and morphological alterations of peritoneal membranes in peritoneal dialysis , 1999, FEBS letters.
[34] J. Wautier,et al. HEMOSTASIS, THROMBOSIS, AND VASCULAR BIOLOGY Cultured Endothelial Cells From Human Arteriovenous Malformations Have Defective Growth Regulation , 2015 .
[35] K. Nitta,et al. Accumulation of advanced glycation end products in the peritoneal vasculature of continuous ambulatory peritoneal dialysis patients with low ultra-filtration. , 1999, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[36] M. Jadoul,et al. Accumulation of carbonyls accelerates the formation of pentosidine, an advanced glycation end product: carbonyl stress in uremia. , 1998, Journal of the American Society of Nephrology : JASN.
[37] J. Cohen. Dialysis fluids and monocytes: suicide or murder? , 1998, Kidney international.
[38] C. Holmes,et al. Reduced Glucose Degradation Products in Bicarbonate/Lactate-Buffered Peritoneal Dialysis Solutions Produced in Two-Chambered Bags , 1997, Peritoneal dialysis international : journal of the International Society for Peritoneal Dialysis.
[39] C van Ypersele de Strihou,et al. Clearance of pentosidine, an advanced glycation end product, by different modalities of renal replacement therapy. , 1997, Kidney international.
[40] P. Ronco,et al. Hyperosmolality suppresses but TGF beta 1 increases MMP9 in human peritoneal mesothelial cells. , 1997, Kidney international.
[41] T. Shigematsu,et al. Immunohistochemical detection of advanced glycosylation end-products in the peritoneum and its possible pathophysiological role in CAPD. , 1997, Kidney international.
[42] C. Holmes,et al. In vitro effects of bicarbonate and bicarbonate-lactate buffered peritoneal dialysis solutions on mesothelial and neutrophil function. , 1996, Journal of the American Society of Nephrology : JASN.
[43] T. Goodship,et al. Acid-base regulation in peritoneal dialysis. , 1994, Kidney international. Supplement.
[44] G. Sacchi,et al. Peritoneal vascular changes in continuous ambulatory peritoneal dialysis (CAPD): an in vivo model for the study of diabetic microangiopathy. , 1989 .