Acquisition of multidrug resistance by L1210 leukemia cells decreases their tumorigenicity and enhances their susceptibility to the host immune response
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[1] H. Verschueren,et al. Tumorigenicity of mouse T lymphoma cells is controlled by the level of major histocompatibility complex class I H-2Kk antigens , 2005, Clinical & Experimental Metastasis.
[2] J. González-Ros,et al. Susceptibility of multidrug resistance tumor cells to apoptosis induction by histone deacetylase inhibitors , 2003, International journal of cancer.
[3] Xiao-xiang Zheng,et al. A flow cytometry-based assay for quantitative analysis of cellular proliferation and cytotoxicity in vitro. , 2002, Journal of immunological methods.
[4] M. Gottesman. Mechanisms of cancer drug resistance. , 2002, Annual review of medicine.
[5] Scott W. Lowe,et al. Apoptosis A Link between Cancer Genetics and Chemotherapy , 2002, Cell.
[6] J. Jaffrezou,et al. Signaling pathways activated by daunorubicin. , 2001, Blood.
[7] M. Davey,et al. The drug resistance proteins, multidrug resistance-associated protein and P-glycoprotein, do not confer resistance to Fas-induced cell death. , 2001, Cytometry.
[8] M. Davey,et al. Drug resistance does not correlate with resistance to Fas-mediated apoptosis. , 2001, Leukemia research.
[9] D. Olive,et al. Human acute myeloid leukemia CD34+/CD38- progenitor cells have decreased sensitivity to chemotherapy and Fas-induced apoptosis, reduced immunogenicity, and impaired dendritic cell transformation capacities. , 2000, Cancer research.
[10] T. Blankenstein,et al. CD4+ T cell--mediated tumor rejection involves inhibition of angiogenesis that is dependent on IFN gamma receptor expression by nonhematopoietic cells. , 2000, Immunity.
[11] F. Lacombe,et al. Flow cytometric evaluation of fas expression in relation to response and resistance to anthracyclines in leukemic cells. , 2000, Cytometry.
[12] G. Gherardi,et al. Interferon-gamma up-regulates expression and activity of P-glycoprotein in human peripheral blood monocyte-derived macrophages. , 1999, Laboratory investigation; a journal of technical methods and pathology.
[13] I. Pastan,et al. Biochemical, cellular, and pharmacological aspects of the multidrug transporter. , 1999, Annual review of pharmacology and toxicology.
[14] W. Dalton,et al. Cytokine-based tumor cell vaccine is equally effective against parental and isogenic multidrug-resistant myeloma cells: the role of cytotoxic T lymphocytes. , 1999, Blood.
[15] M. Nagarkatti,et al. Role of spontaneous and interleukin-2-induced natural killer cell activity in the cytotoxicity and rejection of Fas+ and Fas- tumor cells. , 1998, Blood.
[16] R. Johnstone,et al. The drug efflux protein, P-glycoprotein, additionally protects drug-resistant tumor cells from multiple forms of caspase-dependent apoptosis. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[17] Guoyou Chen,et al. Involvement of MHC class I molecule and ICAM-1 in the enhancement of adhesion and cytotoxic susceptibility to immune effector cells of tumor cells transfected with the interleukin (IL)-2, IL-4 or IL-6 gene , 1997, Journal of Cancer Research and Clinical Oncology.
[18] Y. Komada,et al. Cytotoxic T‐lymphocytes recognizing P‐glycoprotein in murine multidrug‐resistant leukemias , 1997, European journal of haematology.
[19] W. Dalton,et al. Selection for drug resistance results in resistance to Fas-mediated apoptosis. , 1997, Blood.
[20] J. Neefjes,et al. Overexpression of the ABC transporter TAP in multidrug-resistant human cancer cell lines. , 1996, British Journal of Cancer.
[21] B. Seliger,et al. Suppression of MHC class I antigens in oncogenic transformants: association with decreased recognition by cytotoxic T lymphocytes. , 1996, Experimental hematology.
[22] G. Raghu,et al. The multidrug resistance phenotype confers immunological resistance , 1996, The Journal of experimental medicine.
[23] W T Bellamy,et al. P-glycoproteins and multidrug resistance. , 1996, Annual review of pharmacology and toxicology.
[24] I. Fichtner,et al. Characterization of four drug-resistant P388 sublines: resistance/sensitivity in vivo, resistance-and proliferation-markers, immunogenicity. , 1994, Anticancer research.
[25] S. Nagata,et al. Lethal effect of the anti-Fas antibody in mice , 1993, Nature.
[26] J. González-Ros,et al. Possible coexistence of two independent mechanisms contributing to anthracycline resistance in leukaemia P388 cells. , 1993, European journal of cancer.
[27] J. González-Ros,et al. Rapid kinetics of the interaction between daunomycin and drug‐sensitive or drug‐resistant P388 leukemia cells , 1992, FEBS letters.
[28] I. Roninson. The role of the MDR1 (P-glycoprotein) gene in multidrug resistance in vitro and in vivo. , 1992, Biochemical pharmacology.
[29] T. Grogan,et al. Altered expression of P‐glycoprotein and cellular adhesion molecules on human multi‐drug‐resistant tumor cells does not affect their susceptibility to NK‐ and LAK‐mediated cytotoxicity , 1991, International journal of cancer.
[30] V. Ling,et al. Resistance of multidrug‐resistant lines to natural killer‐like cell‐mediated cytotoxicity , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[31] H. Fuji,et al. Differential antigenic expression of the DBA/2 lymphoma L1210 and its sublines: cross-reactivity with C3H mammary tumors as defined by syngeneic monoclonal antibodies. , 1983, Cancer research.
[32] M. Murakami,et al. Differential tumor immunogenicity of DBA/2 mouse lymphoma L1210 and its sublines. III. Control of host resistance to drug-resistant L1210 sublines by H-2-linked and non-H-2-linked genes. , 1983, Journal of the National Cancer Institute.