Intrapatient functional clonality deconvoluted by coupling intracellular flow cytometry and next-generation sequencing in human leukemia
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E. Padron | Christopher Letson | A. List | R. Komrokji | P. Epling-Burnette | M. Ball | Qing Zhang | Y. Zhao | M. Balasis | A. Vedder | Christopher T Letson
[1] J. Shay,et al. Comparison of DNA Quantification Methods for Next Generation Sequencing , 2016, Scientific Reports.
[2] M. Stratton,et al. Mutation allele burden remains unchanged in chronic myelomonocytic leukaemia responding to hypomethylating agents , 2016, Nature Communications.
[3] Tomasz S Tkaczyk,et al. Src Inhibition Blocks c-Myc Translation and Glucose Metabolism to Prevent the Development of Breast Cancer. , 2015, Cancer research.
[4] Isabell C. Pechtl,et al. Abstract 4863: Streamlining NGS workflows using cancer samples by the application of the DNA Integrity Number (DIN) from the Genomic DNA ScreenTape assay , 2015 .
[5] K. Polyak,et al. Non-cell autonomous tumor-growth driving supports sub-clonal heterogeneity , 2014, Nature.
[6] Christopher A. Miller,et al. Functional heterogeneity of genetically defined subclones in acute myeloid leukemia. , 2014, Cancer cell.
[7] D. Birnbaum,et al. Prognostic score including gene mutations in chronic myelomonocytic leukemia. , 2013, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[8] O. Abdel-Wahab,et al. GM-CSF-dependent pSTAT5 sensitivity is a feature with therapeutic potential in chronic myelomonocytic leukemia. , 2013, Blood.
[9] E. Solary,et al. An evolutionary perspective on chronic myelomonocytic leukemia , 2013, Leukemia.
[10] E. Solary,et al. Clonal architecture of chronic myelomonocytic leukemias. , 2013, Blood.
[11] Andrew M. K. Brown,et al. Variable Clonal Repopulation Dynamics Influence Chemotherapy Response in Colorectal Cancer , 2013, Science.
[12] Joshua F. McMichael,et al. The Origin and Evolution of Mutations in Acute Myeloid Leukemia , 2012, Cell.
[13] A. Jankowska,et al. Mutations in the spliceosome machinery, a novel and ubiquitous pathway in leukemogenesis. , 2012, Blood.
[14] Ken Chen,et al. Clonal architecture of secondary acute myeloid leukemia. , 2012, The New England journal of medicine.
[15] Joshua F. McMichael,et al. Clonal evolution in relapsed acute myeloid leukemia revealed by whole genome sequencing , 2011, Nature.
[16] M. Stratton,et al. Somatic SF3B1 mutation in myelodysplasia with ring sideroblasts. , 2011, The New England journal of medicine.
[17] S. Sugano,et al. Frequent pathway mutations of splicing machinery in myelodysplasia , 2011, Nature.
[18] Michael D. Wilson,et al. ChIP-seq: using high-throughput sequencing to discover protein-DNA interactions. , 2009, Methods.
[19] Jonathan M Irish,et al. Single-cell profiling identifies aberrant STAT5 activation in myeloid malignancies with specific clinical and biologic correlates. , 2008, Cancer cell.
[20] Angel F. Lopez,et al. Chronic myelomonocytic leukemia requires granulocyte-macrophage colony-stimulating factor for growth in vitro and in vivo. , 2002, Experimental hematology.
[21] S. Carter,et al. Clonal evolution in hematological malignancies and therapeutic implications , 2014, Leukemia.