Developmental regulation of O2 sensing in neonatal adrenal chromaffin cells from wild-type and NADPH-oxidase-deficient mice
暂无分享,去创建一个
[1] S. Lahiri,et al. Mice lacking in gp91 phox subunit of NAD(P)H oxidase showed glomus cell [Ca2+]i and respiratory responses to hypoxia , 2000, Brain Research.
[2] P. Várnai,et al. Identification of renox, an NAD(P)H oxidase in kidney. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[3] M. Dinauer,et al. NADPH oxidase is an O2 sensor in airway chemoreceptors: evidence from K+ current modulation in wild-type and oxidase-deficient mice. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[4] J. López-Barneo,et al. Secretory responses of intact glomus cells in thin slices of rat carotid body to hypoxia and tetraethylammonium. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[5] S. J. Kim,et al. Inhibition of apamin-sensitive K+ current by hypoxia in adult rat adrenal chromaffin cells , 2000, Pflügers Archiv.
[6] G. Semenza. Perspectives on Oxygen Sensing , 1999, Cell.
[7] S. Archer,et al. O2 sensing is preserved in mice lacking the gp91 phox subunit of NADPH oxidase. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[8] Yu Wang,et al. Selective modulation of membrane currents by hypoxia in intact airway chemoreceptors from neonatal rabbit , 1999, The Journal of physiology.
[9] Roger J. Thompson,et al. Anoxia differentially modulates multiple K+ currents and depolarizes neonatal rat adrenal chromaffin cells , 1998, The Journal of physiology.
[10] M. Dinauer,et al. Gp91(phox) is the heme binding subunit of the superoxide-generating NADPH oxidase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[11] G. Rychkov,et al. Oxygen‐sensing mechanisms are present in the chromaffin cells of the sheep adrenal medulla before birth , 1998, The Journal of physiology.
[12] M. Duchen,et al. Hypoxia‐induced catecholamine secretion in isolated newborn rat adrenal chromaffin cells is mimicked by inhibition of mitochondrial respiration , 1997, The Journal of physiology.
[13] C. Nurse,et al. Dopaminergic Properties of Cultured Rat Carotid Body Chemoreceptors Grown in Normoxic and Hypoxic Environments , 1997, Journal of neurochemistry.
[14] N. Mochizuki-oda,et al. Hypoxia‐Induced Catecholamine Release and Intracellular Ca2+ Increase via Suppression of K+ Channels in Cultured Rat Adrenal Chromaffin Cells , 1997, Journal of neurochemistry.
[15] Roger J. Thompson,et al. Developmental loss of hypoxic chemosensitivity in rat adrenomedullary chromaffin cells. , 1997, The Journal of physiology.
[16] M. Quinn,et al. Assembly of the Human Neutrophil NADPH Oxidase Involves Binding of p67phox and Flavocytochrome b to a Common Functional Domain in p47phox* , 1996, The Journal of Biological Chemistry.
[17] J. Pancrazio,et al. A major role for calcium-dependent potassium current in action potential repolarization in adrenal chromaffin cells , 1994, Brain Research.
[18] H. Yeger,et al. Oxygen sensing in airway chemoreceptors , 1993, Nature.
[19] H. Acker,et al. Involvement of an NAD(P)H oxidase as a pO2 sensor protein in the rat carotid body. , 1990, The Biochemical journal.
[20] C. Cheung. Fetal adrenal medulla catecholamine response to hypoxia-direct and neural components. , 1990, The American journal of physiology.
[21] T. Slotkin,et al. Adrenomedullary catecholamine release in the fetus and newborn: secretory mechanisms and their role in stress and survival. , 1988, Journal of developmental physiology.
[22] T. Slotkin,et al. Non-neurogenic adrenal catecholamine release in the neonatal rat: exocytosis or diffusion? , 1986, Brain research.
[23] H. Lagercrantz,et al. The "stress" of being born. , 1986, Scientific American.
[24] W. Kummer,et al. Subcellular localization and function of B-type cytochromes in carotid body and other paraganglionic cells. , 2002 .
[25] C. González,et al. NADPH oxidase inhibition does not interfere with low PO 2 transduction in rat and rabbit CB chemoreceptor cells , 1999 .
[26] A. Jackson. Oxygen sensing, plasticity and catecholaminergic functions in cultured chromaffin cells of rat carotid body and adrenal medulla: Modulation by chronic hypoxia and acetylcholine receptors , 1997 .
[27] David A. Williams,et al. Mouse model of X–linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production , 1995, Nature Genetics.
[28] T. Slotkin,et al. Adrenomedullary function in the neonatal rat: responses to acute hypoxia. , 1985, The Journal of physiology.